Sentences with phrase «as young cells»

As young cells mature and prepare to produce antibodies, however, these genes move to the interior of the nuclei.

Not exact matches

Hobson was upset — she remembered her early experience as being all about paying her dues — but she realized something: «It all goes back to the cell phone,» she says, meaning that since cell phones have been around, young people had access to instant answers (be it from their mom or Google).

Though they should never be used as an excuse to leave a baby unattended at significant distances, cell phones do allow parents the opportunity to listen in on young children so that they can render immediate assistance in the event something occurs.

Campaign staff also relied on cell phones to reach segments of the population less likely to be on a computer regularly, such as young people, minorities and the poor.

Tomorrow, September 30, join Preserve Our Legacy Inc. as they host a fundraising event for young people afflicted with sickle cell anemia.

No more cell phones for young drivers: As part of a package of passenger car - related statutory amendments, Cuomo also wants to ban even hands - free mobile phone and electronic device use for junior permit and junior license holders (those under 18).

Researchers show for the first time that healthy older men and women can generate just as many new brain cells as younger people.

Healthy people in their 70s have just as many young nerve cells, or neurons, in a memory - related part of the brain as do teenagers and young adults, researchers report in the April 5 Cell Stem Cell.

After receiving an injection of neural stem cells from young mice, however, they performed as well as healthy mice did.

Eye diseases — such as age - related macular degeneration, as well as a genetic condition called Stargardt's macular dystrophy that afflicts young people — are considered excellent candidates for stem cell therapy because the eye is an immune - privileged site, meaning transplanted cells are not as likely to be rejected as foreign compared with transplants elsewhere.

The Eppendorf & Science Prize in Neurobiology recognizes outstanding international neurobiological research based on current methods and advances in the field of molecular and cell biology by a young early - career scientist, as described in a 1,000 - word essay based on research performed within the last three years.

In his study, some of the cells his team initially flagged as young neurons turned out to be mature cells upon further investigation.

Stem cell biologist Amy Wagers and cardiologist Richard Lee, both of the Harvard Stem Cell Institute, wondered if any circulating factors, in young blood, such as hormones, might affect aging heacell biologist Amy Wagers and cardiologist Richard Lee, both of the Harvard Stem Cell Institute, wondered if any circulating factors, in young blood, such as hormones, might affect aging heaCell Institute, wondered if any circulating factors, in young blood, such as hormones, might affect aging hearts.

And even cells that were young, such as white blood cells that may be just a few days or weeks old, still carried the distinct genetic imprint of their 50 - year - old donor.

Furthermore, examination of aged mice showed dramatically reduced levels of Cbf - beta in bone marrow cells, as compared to younger mice.

Our study suggests otherwise, though, as we found that a certain type of B cell is quite abundant in the ventricles, meninges, and choroid plexus in the brains of young mice.

Journals depend on their authors, but equally, researchers in the life sciencesespecially young investigatorsneed to publish in «brand name» journals, such as Cell, Nature and Science, to advance their careers.

As demonstrated by Leonard Hayflick a half - century ago, human cells have a limited replicative lifespan, with older cells reaching this limit sooner than younger cells.

She has recently been awarded the NIH Director's New Innovator Award and been honored as a World Economic Forum Young Scientist and as one of Cell's 40 under 40.

Scientists were even more stunned in July 2002 when researchers led by stem cell biologist Catherine Verfaillie at the University of Minnesota reported that bone marrow — derived cells they had injected into young embryos contributed to all three embryonic layers, just as embryonic stem cells would do.

When the induced pluripotent stem cell method was used, as expected, the patterns in the neurons were indistinguishable between young and old derived samples.

Cardiovascular disease in these young patients develops as vulnerable cells lining the interior of major arteries (vessels that carry blood away from the heart) accumulate the toxic protein and die.

The therapy has led to remission rates as high as 90 % in children and young adults with B - cell acute lymphoblastic leukemia (ALL); however, the side - effects can be extreme and difficult to manage.

Although we did observe positive effects on some aging traits, such as memory impairments and reduced red blood cell counts, our studies showed that similar drug effects are also seen in young mice, indicating that rapamycin did not influence these measures by slowing aging, but rather via other, aging - independent, mechanisms.»

So they do not make as quickly more differentiated cells as the young ones.»

As the cells get older, they acquire clumps of proteins and extra pieces of DNA, but when Angelika Amon at the Massachusetts Institute of Technology and colleagues tracked spores from old and young yeast cells they found that such abnormalities disappeared, meaning all spores had the same lifespan.

The special cells also would eliminate additional surgeries for younger adults and children with congenital heart defects, who need new pacemakers as they grow.

«But as soon as I saw that they [Lander and Cell] were not giving the young people, the people who actually did the work, and Jennifer and Emmanuelle, adequate credit, I just said, «No, I have to correct what I know to be false.

The young mice showed signs of brain deterioration as well, including inflammation and decreased birthrates of new nerve cells.

Previous in vitro studies conducted by researchers in other countries showed that this molecule was able to reduce the multiplication and increase the mortality of cells from melanoma, the most aggressive type of skin cancer, as well as breast cancer and neuroblastoma, a tumor that typically affects patients aged 15 or younger.

By STEPHEN YOUNG Few phenomena in biology are quite as dramatic as the cell cycle, the ordered sequence of events by which cells grow and divide.

«This breakthrough holds substantial promise as the base technology for the application of the next - generation solar cells, as well as various IoT devices and displays,» says Professor Jin Young Kim.

But now that we know what kinds of changes occur as these cells age, we can ask which of these changes reverse themselves when an old cell goes back to becoming a young cell» — as appeared to be the case when tissues of older mice were exposed to blood from younger mice.

Instead, they found, in quiescent satellite cells taken from the younger mice, copious instances in which histones in the vicinity of genes ordinarily reserved for other tissues were marked with both «stop» and «go» signals, just as genes associated with development to mature - muscle status were.

As soon as we got it out of the package, she became a diligent young investigator, studying the leg of a fly, dog cardiac muscle and onion epidermal cellAs soon as we got it out of the package, she became a diligent young investigator, studying the leg of a fly, dog cardiac muscle and onion epidermal cellas we got it out of the package, she became a diligent young investigator, studying the leg of a fly, dog cardiac muscle and onion epidermal cells.

The team found they could replicate the healing abilities of the engineered mice by giving nongenetically altered ones drugs that help activate certain metabolic processes — the same pathway Lin28a stimulates — revving up and energizing cells as if they were much younger.

«This important study not only adds more genetic risk factors to the list of those known for testicular germ cell tumours — the most common cancer in young men — but also adds detail to the emerging picture of testicular cancer as a strongly heritable disease.

Using human fat - derived stem cells from young (aged 24 - 36 years), middle - aged (aged 48 - 55 years) and elderly (aged 60 - 81) participants, the team used ECIS to collect complex measurements during the growth and differentiation stages the hASC exhibited as they converted to bone cells.

It is awarded annually to one young scientist for the most outstanding neurobiological research based on methods of molecular and cell biology conducted by him / her during the past three years, as described in a 1,000 - word entrance essay.

The team also tested the method on other proteins, such as those found in young muscle cells.

«In a young, healthy individual, hypoxia — low oxygen levels — triggers the body to make factors that help coordinate the growth of new blood vessels but this process doesn't work as well as we age,» says Gregg Semenza, M.D., Ph.D., professor of pediatrics and genetic medicine and director of the vascular biology program at the Johns Hopkins Institute for Cell Engineering.

Health improvement (allowing to post - pone / escape the diseases and thus live, healthier / disease - free longer, but not above human MLSP of around 122 years; thus these therapies do not affect epigenetic aging whatsoever, they are degenerative aging problems not regular healthy aging problem (except OncoSENS - only when you Already Have Cancer - which cancer increases epigenetic aging, but cancer removal thus does not change anything / makes no difference about what happens in the other cells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to Mcells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to Mcells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to MCells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to Mcells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to MLSP).

Sure enough, when the researchers examined the brains of PD patients, they found more cells exhibiting signs of senescence than in people without the disease — and especially astrocytes, as they had expected.7 This was true even after matching patients for age, meaning that PD subjects had even more senescent astrocytes in their SNcs than is typical for people their age (ranging in this case from 50 — 92 years at autopsy)-- and remember, aging already drives an increase in the burden of these cells as compared with young people, even in those who have yet to develop Parkinson's disease.7

«We found that older people have similar ability to make thousands of hippocampal new neurons from progenitor cells as younger people do,» lead author Maura Boldrini and associate professor of neurobiology at Columbia Universtiy says.

Researchers are working to improve stem cell transplants so that they don't require immune - depleting chemotherapy beforehand, and clinical trials evaluating novel immunotherapy approaches, such as CAR T cells, that might be able to help young patients while sparing them the debilitating side effects.

In a proof - of - principle study published in the journal Cell Stem Cell, the researchers report that defects in the regeneration of the myelin sheaths surrounding nerves, which are lost in diseases such as multiple sclerosis may be at least partially corrected following exposure of an old animal to the circulatory system of a young animal.

Cells with similar characteristics accumulate during normal aging as well as in younger persons infected with human immunodeficiency virus, suggesting that the process of replicative senescence is not an artifact of cell culture but is also occurring in vivo.

Using melanoma cells and both young and old normal skin cells as a model, the lab is trying to unravel what these changes may be, and how they affect tumor progression.

The challenge takes on even more urgency with recent developments, including a federal administration now more open to exploring the potential of stem cells, the recent FDA approval of a human trial involving embryonic stem cells, as well as the reported case of a young boy who developed a brain tumor four years after receiving a stem - cell treatment for a rare genetic disorder.

The researchers also detected surprising numbers of aged beta cells in people as young as 20.