Hence, suppression of peroxynitrate formation should decrease lipid peroxidation thereby decreasing damage created by secondary injury within the context of
CNS injury.
Discover the factors that inhibit axon regrowth following
CNS injury and the potential therapeutics being developed to overome them, in this special report from Future Neurology.
As an Associate Professor at OSU, my research has been concentrated in the areas of aging, neuroimmunology, and neurotrauma.Overall, my research aim is to determine the degree to which the bi-directional communication between the brain and the immune system is affected by age, stress, and traumatic
CNS injury and to delineate the mechanism by which inflammatory cytokine pathways cause long - lasting complications (e.g., cognitive decline and depression).
The current focus of Dr. Trakhtenberg's research is on solving the fundamental problem of the failure of CNS projection neurons to regenerate injured axons and developing translational approaches for treating optic neuropathies (eg, glaucoma) and other types of
CNS injuries.
The focus of my laboratory is on exploring novel approaches for solving the fundamental problem of the failure of CNS projection neurons to regenerate injured axons and developing translational approaches for treating optic neuropathies (e.g., glaucoma) and other types of
CNS injuries.
While previous studies had demonstrated a synergistic effect of BMP and LIF on the astrocytic differentiation of human neural stem cells [15], it remains unclear whether BMP and LIF induce distinct types of astrocytes and if so, what the functional properties of these astrocytes may be with respect to repairing
CNS injuries.
Not exact matches
Starting with transplants of human oligodendrocytes in the late 1980s [40], and more recently with populations of human oligodendrocyte progenitor cells isolated from the developing or adult
CNS, or from human embryonic stem cells, it has been possible to generate extensive myelination upon transplantation into spinal cord
injury or into congenital mouse models of hypomyelination [41]--[48].
A failure of normal astrocyte generation by
CNS precursor cells has been discovered to be a consequence of the mutations that cause Vanishing White Matter leukodystrophy [7], and dysfunction of astrocytes has also been suggested to be of importance in models of amyotrophic lateral sclerosis [8], forebrain ischemic
injury [9], epileptic seizures [10], Huntington's disease [11], tuberous sclerosis [12] and Rett syndrome [13].
However, performing 5 - 10 minutes of dynamic stretching before the actual workout will prepare you for a better performance and reduce your risk of
injury by activating your
CNS, improving your blood circulation, priming your muscles for maximum force production and improving your flexibility.
There is emerging evidence for acne, PCOS, exercise performance, wound healing, longevity, cancer,
CNS Oxygen Toxicity, autism, Angelman's Syndrome, ALS, MS, Neuropathies, Alzheimer's disease, brain
injury, neurodegeneration, anxiety, depression, bipolar and schizoaffective disorder.
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It's one of the most important things you can do for long - term success, because every so often your body needs a week of FULL recovery to come back from any minor nagging
injuries and to let your
CNS take a break.
It's not recommended to go over 8 for deadlifts due to the demands on the central nervous system (
CNS) and the possibility of
injury from doing high - rep deadlifts.
These
injuries disrupt the signals that are carried back and forth between the body and the brain, which comprises the central nervous system (
CNS).
In the other prospective investigation of longer - term family outcomes, our research team (Wade et al., 2002) followed children with severe TBI, moderate TBI, and orthopedic
injuries not involving the central nervous system (
CNS) at a follow - up conducted an average of 4 years after
injury.