Sentences with phrase «cu behaviour»

Recently, Waller et al. [67] applied Belsky's model in relation to the development of CU behaviour.

In the first study [44], temperament (fearlessness) of the biological mother predicted CU behaviour of the adopted child at 27 months, via earlier fearlessness measured at 18 months; similarly, low affiliative behaviour of biologic mothers directly predicted child CU behaviours, although without any correlation with child affiliative behaviours tested at 18 months.

It was found that greater dysfunctional representations were significantly associated with higher CU behaviours but not conduct problems, and that dysfunctional family representations partially accounted for the link between sensitive parenting and later CU behaviours, indicating that the internalization of caregiving may be one of multiple developmental mechanisms contributing to the association between parenting and callous - unemotional conduct.

In this earlier age, «CU behaviours» is a more adequate definition rather than «CU traits», given the lesser evidences about their stability over time [67].

Although most of the studies about CU traits associated to CD are focused on children and adolescents, more recently the interest has been expanded to early childhood and toddler age, in order to detect early sings of CU behaviours.

Higher CU behaviours in children as early as 3 year - old are related to lower guilt and empathy, more proactive aggression [67], and, perspectively, to CU traits in late childhood [67, 82, 83].

In summary, all these data suggest that the trajectory of children at high risk for CU behaviours could be modified by high levels of positive parenting [67].

Furthermore, more positively reinforcing mothers buffered inherited risk for early CU behaviours.

Not exact matches

Callous Unemotional (CU) traits are a meaningful specifier in subtyping CD for more severe antisocial and aggressive behaviours in adult psychopathology; they represent the affective dimension of adult psychopathy, but they can be also detected in childhood and adolescence.

It was found that serotonin level was a significant predictor of high CU traits even when antisocial behaviour severity was included as a covariate.

In the study by Muratori et al. [24] involving children with disruptive behaviour disorder, a higher level of socio - economic status at baseline was related to lower level of CU traits.

Of all the variables entered, only antisocial behaviour severity was a significant predictor of CU group membership (β = 1.15, Wald criteria = 5.05, p = 0.025, Exp (B) = 3.15).

This study also demonstrated that within a sample of children with antisocial behaviour problems there is a relationship between CU traits and peripheral serotonin levels that is independent from antisocial behaviour severity.

A recent meta - analysis showed that low family socio - economic status is associated with higher levels of children's antisocial behaviours, and indicated that this relationship is stronger when CU traits are considered as outcome variables [72].

High levels of CU traits have, however, been shown to be a risk factor for the greater use of aggression and antisocial behaviours [5]--[7].

It was predicted that boys with antisocial behaviour problems and high levels of CU traits would have lower levels of serum serotonin than boys with antisocial behaviour problems and low levels of CU traits.

Participants were rated by clinical psychologists, blind to levels of CU traits, on levels of antisocial behaviour problems, ADHD, autism spectrum disorders and anxiety and depression.

It should be noted that heightened levels of aggression and antisocial behaviour do not demarcate high levels of CU traits.

They use observational measures to score guilt (in breaking a valued object situation), empathy (hurting the parent during play) and fearlessness in toddlers and preschoolers, and related these findings tolater parent - rated measures of CU traits and externalizing behaviours during scholar and pre-adolescent age.

Furthermore, the high heritability of CU traits and their association with more chronic and serious aggression and antisocial behaviour problems make them a strong candidate for the driving force behind the familial transmission of aggressive behaviour that Halperin et al. [38] argue is mediated, in part, by reduced central serotonin function.

This result supports the idea that previously reported mixed findings regarding the relationship between serotonin and antisocial and aggressive behaviour in children may be linked to variation in the levels of CU traits within and between samples.

Current evidence, however, is inconsistent and the precise role of CU traits versus antisocial and aggressive behaviour, and other comorbidities, is unclear.

High CU traits are also related to more severe and chronic antisocial behaviour [5], [6], greater use of proactive aggression [7], and specific patterns of neural dysfunction, specifically with regards to the amygdala [8]--[11].

Serotonin has, however, been implicated in behaviours often found to be positively associated with CU traits; that is, aggression, antisocial behaviour and impulsivity.

Viding et al. [4] found that genetic variability accounted for 71 % of the variance in antisocial behaviour problems in 9 year old children with high levels of CU traits but only 36 % in those with low levels of CU traits.

In addition, given the high heritability of antisocial behaviour problems in the presence of high levels of CU traits, it was hypothesized that CU traits would be significantly associated with functional polymorphisms of the serotonin - system.

Childhood antisocial behaviour problems characterised by high levels of CU traits have been found to be highly heritable [3], [4].

Salekin et al. [96] tested a positive psychological intervention, named Mental Models, to reduce the behaviour problems in youth with higher CU traits.

These finding suggest that antisocial behaviour problems characterised by high levels of CU traits may have unique aetiological mechanisms associated with specific cognitive and affective impairments that are heavily dependent on genetics.

Therefore, they could represent the first step in identifying the presence of CU traits, and the possible developmental pathways to antisocial behaviours, and in promoting early interventions.

Our results suggest that from pre-birth, children with CU traits who were also exposed to more PT, are potentially more liable in developing behaviours that are adaptive in harsh environments, thereby providing some support for «fetal programming».

CU traits have been shown to differentiate distinct subgroups of children and adolescents with serious EB (e.g., conduct problems, conduct disorder, aggression and antisocial behaviour)[9].

Also, findings are discussed in support of a general empathy deficit for distress cues which may underlie the problem behaviour of young males with CU traits.

Adolescents with conduct disorder (CD) and elevated callous - unemotional (CU) traits have been reported to present with a more severe and persistent pattern of antisocial behaviour than those with low levels of CU traits.