Emotion processing deficits in the different dimensions of psychometric schizotypy.
Not exact matches
This is demonstrated in the studies reviewed in the special issue, which use computational models to examine brain
processes, such as learning,
emotion, dopamine signaling and information
processing, and how
processes interact in
deficits underlying psychiatric disease.
Because attention shifting
deficits have also been reported for autism, Riby and Hancock propose that the relationship between the prefrontal cortex (involved in inhibiting behavior) and the amygdala (involved in
emotion processing) may be crucial in both of these neuro - developmental disorders.
Individuals with brain injury experience
deficits in emotional
processing and social cognition, most notably the inability to recognize
emotions expressed by facial features.
People with
deficits in emotional
processing have difficulty recognizing and interpreting the facial expressions of six universal
emotions: happiness, sadness, fear, anger, surprise and disgust.
For example researchers have argued that the social and emotional
deficits seen in AB may be mediated by impaired connectivity between the
emotion processing and regulation network [146,147].
Moreover, the emotional
processing deficits associated with CU traits, may predispose parents of children elevated on these traits to significant challenges throughout their task of
emotion socialization.
It has been suggested that the observed
deficits in decision making may directly result from aberrant
emotion processing as for example observed after frontal brain damage [91].
The neurobiological mechanism underlying
emotion regulation
deficits (simply put: a more active emotional response system and less effective regulation of the emotional response) is both (1) the normative developmental imbalance during adolescence (reviewed above), and (2) a parallel
process that underlies and further exacerbates risk for addiction and comorbid psychopathology more generally.
The neurobiological
processes underlying
emotion regulation
deficits can manifest across development in the form of internalizing, externalizing, and / or SUDs, depending on the manner in which the response and regulation system becomes dysfunctional [54].
Deficits in
emotion regulation emerge during adolescence in part due to the dual systems model of imbalanced neurological development between the response and regulation systems, a risk
process which is then exacerbated in contexts where the same response and regulation system malfunctions in various ways that can lead to multiple forms of psychopathology.
For some youth, the broad externalizing and internalizing difficulties outlined above may be explained by underlying
deficits in
emotion regulation (i.e., the set of
processes that control
emotions; Gross and Thompson 2007; Mazefsky et al. 2013; Rieffe et al. 2011; Weiss 2014).