HDL cholesterol particles act as cholesterol vacuum cleaners, removing excess cholesterol from tissues all over your body.
Not exact matches
(5) Research has shown that in most cases eating up to 3 eggs per day can actually improve your good
cholesterol (
HDL) and your bad
cholesterol (LDL) will stay the same or change from being small dense LDL
particles to large LDL
particles.
In individuals with high
HDL -
cholesterol levels in the circulation, the lipid composition of the
HDL particle was more beneficial regarding heart disease risk.
Based on this Finnish study, and earlier ones, the terms «
HDL -
cholesterol» and «
HDL particle» should not be confused to indicate heart disease risk.
HDL particles remove
cholesterol from the blood vessel walls and transport it to the liver to be excreted from the body via bile.
In individuals, whose
HDL -
cholesterol levels were low, the quality of
HDL particles was also impaired; they contained smaller amounts of lipid molecules which are known to be antioxidant and thus protective to arteries.
It is likely that molecules other than
cholesterol — the lipid and protein molecules on the surface of the
HDL particle — are responsible for the protective effects of
HDL particles against heart disease.
Another area focuses on the
HDL particle (carrier of good
cholesterol in the blood).
Instead, the coconut oil's overall effect raised
HDL while lowering triglycerides and small LDL
cholesterol particles, which is definitely a good thing.
According to the Cooper Institute, a good way to determine what kind of LDL
particles you have is to find your ratio of triglycerides to
HDL cholesterol.
It also transports
cholesterol from extra-hepatic sites, including the arterial wall, to the liver for excretion via reverse
cholesterol transport.77 Results from IER (60 - 85 % ER / alternate days) trials have thus far been inconsistent, with some reporting decreases 40, and others increases 37, 39, but with the majority showing no effects on
HDL cholesterol levels.38, 42, 43, 45, 49, 51, 52Inconsistencies may have arisen due to the biphasic response of
HDL documented by studies of ER induced weight - loss, whereby levels typically decrease during active weight - loss then either return to baseline or (less commonly) rise following attainment of weight stability.78 Complementary increases in
HDL levels and
particle size have been shown when endurance exercise have been combined with IER 42, 46, 47 however further discussion goes beyond the scope of this review.
So, for those you listening in, just to kinda bring this full circle here briefly, high cortisol, in the case of high cortisol, one of the things that Dr. Bryan recommended was to look at things like your
cholesterol particles and your
HDL, to look at things like your testosterone, to look at things like the ACTH that we talked about, and some of these other variables that can affect ACTH, and then of course, something like an oxidative stress panel.
In multiple human studies, the low - fat diet has actually made some important risk factors worse, raising triglycerides, lowering
HDL (the good)
cholesterol and making the LDL
particles smaller (10, 11, 12, 13).
This way you can raise your
HDL cholesterol («good»
cholesterol), lower your triglycerides and reduce levels of small
particle LDL
cholesterol («bad»
cholesterol).
In a 2010 study published in the American Journal of Clinical Nutrition, the researchers found that when you replace saturated fat with a higher carbohydrate intake, you increase insulin resistance and obesity, and increase your chances of getting heart disease by increasing triglycerides and small LDL
particles, and reducing beneficial
HDL cholesterol.
We are just starting to understand how the body carries
cholesterol to and from cells in the form of LDL, VDL, and
HDL particles.
If you eat properly — which is really the only known good way to regulate LDL
particle size — then it does the right thing; it takes the
cholesterol to your tissues, the
HDL takes it back to your liver, and nothing gets stuck causing damage.
The DASH diet, significantly reduced LDL
cholesterol, large LDL
particle size (reduced LDL
particle size raises risk) and
HDL cholesterol (more
HDL cholesterol lowers risk).
Lipoprotein lipid profile disturbances associated with an elevated TG level include low High Density Lipoprotein (
HDL)
cholesterol and
HDL particle (
HDL - P), and a predominance of small dense Low Density Lipoprotein (LDL) and LDL
particle (LDL - P).
People who do this typically have by a year, two years, or so, sometimes longer:
HDL cholesterol drops, triglycerides start to climb back up (even if they'd dropped substantially at the start), small LDL
particles on lipoprotein analysis — increase.
If you eat properly, which is really the only known good way to regulate LDL
particle size, then it does the right thing; it takes the
cholesterol to your tissues, the
HDL takes it back to your liver, and nothing gets stuck causing damage.
One good type of testing includes testing the
particle size of both LDL and
HDL cholesterol.
For a large proportion of the population, however, the effect of higher - carbohydrate diets, particularly those enriched in refined carbohydrates, coupled with the rising incidence of overweight and obesity, creates a metabolic state that can favor a worsening of the atherogenic dyslipidemia that is characterized by elevated triglycerides, reduced
HDL cholesterol, and increased concentrations of small, dense LDL
particles (6, 7).
Regarding long - term NK, a study on obese patients showed the positive effects of the ketogenic diet after 24 weeks, including weight loss, increased
HDL cholesterol and decreased LDL
particles, and decreased triglycerides and blood glucose.
Studies of atherogenic lipoprotein concentrations and properties have raised questions about the benefit of lowering saturated fat intakes by increasing carbohydrate intake, which can induce atherogenic dyslipidemia, and the benefit of increasing monounsaturated fat intakes, which does not lead to improvements in the properties of LDL
particles that are associated with atherosclerosis in animal models, although substitution with monounsaturated fat rather than carbohydrate has been shown to reduce the ratio of total and LDL
cholesterol to
HDL cholesterol.