Hyperglycemia increases muscle protein breakdown and limits the protein synthesis — the process of creating new proteins.
Not exact matches
The research from Medicus substantiates that RBT's RiBran ™ and RiSolubles ® products reduce
hyperglycemia; improves insulin levels and insulin sensitivity; and
increases adiponectin levels.
Worrisome is that already in the state of prediabetes
hyperglycemia is associated with
increased risk of type 2 diabetes, cardiovascular disease, dementia and cancer.
Previous research has shown that
hyperglycemia (high blood sugar) after CABG and other cardiac surgery is associated with
increased morbidity and mortality; however, more recent studies have shown that liberal maintenance of blood glucose levels (< 180 mg / dL) after CABG surgery can be safer and more advantageous in both diabetic and non-diabetic patients.
In contrast, in mice with normal immune systems, emulsifiers induced low - grade or mild intestinal inflammation and metabolic syndrome, characterized by
increased levels of food consumption, obesity,
hyperglycemia and insulin resistance.
The
increased risk of
hyperglycemia associated with prenatal exposure to famine is also passed down to the next generation, according to a new study of hundreds of families affected by widespread starvation in mid-20th Century China.
Her research team found that cellular oxidative stress (arising because of reactive oxygen species)
increases in mice exposed to THS, damaging proteins, fats and DNA, and leading to
hyperglycemia (excess glucose in the blood stream) and insulinemia (excess insulin in the blood)-- a condition also called insulin resistance.
Thus,
increased MYCL does not appear to be an early marker in the pathogenesis of diabetes but a consequence of chronic
hyperglycemia (228).
Josin researchers made two major findings: They identified the mechanisms by which GLP - 1 can induce protective actions on the glomerular (renal) endothelial cells by inhibiting the signaling pathway of Ang II and its pro-inflammatory effect; and demonstrated a dual signaling mechanism by which
hyperglycemia, via PKCβ activation, can
increase Ang II action and inhibit GLP - 1's protective effects by reducing the expression of GLP - 1 receptors in the glomerular endothelial cells.
PROVIDENCE, R.I. [Brown University]-- The
increased risk of
hyperglycemia associated with prenatal exposure to famine is also passed down to the next generation, according to a new study of hundreds of families affected by widespread starvation in mid-20th Century China.
This
increase in insulin sensitivity lasts 24 - 48 hours after strength training so is a vital part in helping to maintain steady blood glucose levels and reduce your chances of suffering from
hyperglycemia and its complications.
Burdock also appears to
increase carbohydrate tolerance and prevents
hyperglycemia, another possible benefits for diabetics.
The thiazide diuretics raise cholesterol and triglyceride levels, make the blood more prone to clot, thus
increasing your risk of stroke, and make blood sugar unstable,
increasing your risk of
hyperglycemia and diabetes.
Both insulin and SUs
increase insulin to reduce
hyperglycemia.
The free fatty acids inhibit the oxidation of glucose for energy, creating insulin resistance, the condition that normally
increases with aging, and that can lead to
hyperglycemia and «diabetes.»
However, when diabetes (
hyperglycemia - high blood sugar), is treated with insulin or medication that
increases insulin production, glucose levels can easily return to the normal range.
Low - volume high - intensity interval training reduces
hyperglycemia and
increases muscle mitochondrial capacity in patients with type 2 diabetes.
Many high - carbohydrate, low - fat diets may be counterproductive to weight control because they markedly
increase postprandial
hyperglycemia and hyperinsulinemia.
Here's the part they chose that links fatty liver to fructose: «Excess intrahepatic triglycerides -LCB- fat in the liver -RCB- are formed in response to
hyperglycemia, with
increased potentiation from dietary fructose.»
They commented that high carbohydrate diets which cause
hyperglycemia have been demonstrated to worsen the efficacy of the endothelial system and are associated with an
increased CVD risk.
In a puppy with an underdeveloped or improperly developed pancreas, insulin production would be low or absent, resulting in
increased blood sugar, or
hyperglycemia.
An
increase in the release of cortisol and glucagon can cause insulin resistance and
hyperglycemia.
The persistence of clinical signs (
increased urination, appetite and thirst) as well as the development of chronic complications (such as ketoacidosis, fatty liver, pancreatitis, cataract, bacterial infections) are directly related to the severity and duration of
hyperglycemia.
Most cats with blood sugar readings too high (
hyperglycemia) or too low (hypoglycemia) actually have an
increased appetite.
Feeding diabetic cats a very low - carbohydrate, high - protein diet improved
hyperglycemia, reduced insulin dosage, and
increased the rate of diabetic remission (55).
If this system is disrupted for any reason, the glucose can not enter the cell and subsequently, the level of glucose
increases in the blood stream (
hyperglycemia) and the cells go «hungry».
This is because your cat's insulin needs can change for a variety of reasons (infection, stress, anorexia, etc.) and also because the curve generated at the vet clinic is influenced by stress
hyperglycemia which is a false
increase in blood glucose due to the cat's internal reaction to this stress.
Often if a cat's blood glucose is persistently elevated, the symptoms or signs associated with
hyperglycemia will return (
increased thirst, urination and appetite).
Laboratory examination of dogs having pancreatic cancer shows non specific results like anemia (low red blood cell count), neutrophilia (
increase of neutrophilic white blood cells in blood), bilirubinemia (Bilirubin in blood) and
hyperglycemia (
Increased glucose level in the blood).
Increased glucagon appears to contribute to development of severe
hyperglycemia by mobilizing hepatic stores of glucose and to development of ketoacidosis by
increasing the oxidation of fatty acids in the liver.