Sentences with phrase «in normal mouse groups»

In normal mouse groups, the dominant mouse trims the whiskers and facial hair of subordinate mice, but the mutant mice, which were housed together, all had full sets of whiskers.

Not exact matches

Three groups of middle - aged mice (about a year old) were studied: one group ate a normal diet, in which fewer than 30 percent of calories came from fat, while two others were fed high - calorie diets in which 60 percent of the calories came from fat.

By examining the brains of these mice, the researchers observed a substantial decrease in inhibitory CA2 neurons, as compared to a control group of normal, healthy mice — a change remarkably similar to that previously observed in postmortem examinations of people with schizophrenia.

The researchers induced two groups of mice — germ - free (GF) mice, which are raised in a sterile environment, and specific - pathogen - free mice raised under normal laboratory conditions — to develop forms of asthma or ulcerative colitis.

The study compared intestinal wound healing in two groups of mice: 1) typical mice (wild type) found in nature and 2) mice genetically deficient in the healing factor IL - 10, specifically in macrophages, which impairs their ability to have normal wound repair.

In the new study, Ji's group put SHANK3 - deficient mice through a battery of sensory tests, finding that the animals had lower sensitivity than normal mice to heat and heat - related pain — akin to the soreness a person feels after a sunburn.

In an article entitled «Prion protein modulates monoaminergic systems and depressive - like behavior in mice» and published in the Journal of Biological Chemistry, the group shows that mice lacking normal prions show a depressive - like behavior similar to depression symptoms found in patients with Alzheimer's and prion diseases, namely Creutzfeldt - Jakob Disease (CJD), variant Creutzfeldt - Jakob Disease (vCJD), Gerstmann - Sträussler - Scheinker syndrome, Fatal Familial Insomnia and kurIn an article entitled «Prion protein modulates monoaminergic systems and depressive - like behavior in mice» and published in the Journal of Biological Chemistry, the group shows that mice lacking normal prions show a depressive - like behavior similar to depression symptoms found in patients with Alzheimer's and prion diseases, namely Creutzfeldt - Jakob Disease (CJD), variant Creutzfeldt - Jakob Disease (vCJD), Gerstmann - Sträussler - Scheinker syndrome, Fatal Familial Insomnia and kurin mice» and published in the Journal of Biological Chemistry, the group shows that mice lacking normal prions show a depressive - like behavior similar to depression symptoms found in patients with Alzheimer's and prion diseases, namely Creutzfeldt - Jakob Disease (CJD), variant Creutzfeldt - Jakob Disease (vCJD), Gerstmann - Sträussler - Scheinker syndrome, Fatal Familial Insomnia and kurin the Journal of Biological Chemistry, the group shows that mice lacking normal prions show a depressive - like behavior similar to depression symptoms found in patients with Alzheimer's and prion diseases, namely Creutzfeldt - Jakob Disease (CJD), variant Creutzfeldt - Jakob Disease (vCJD), Gerstmann - Sträussler - Scheinker syndrome, Fatal Familial Insomnia and kurin patients with Alzheimer's and prion diseases, namely Creutzfeldt - Jakob Disease (CJD), variant Creutzfeldt - Jakob Disease (vCJD), Gerstmann - Sträussler - Scheinker syndrome, Fatal Familial Insomnia and kuru.

With this information in hand, the researchers then performed another experiment with two more groups of normal mice that were septic.

Obese mice gained less weight after exercise or WBV than obese mice in the sedentary group, although they remained heavier than normal mice.

In March three groups reported separately in Science that they had repeated Faustman's protocols and reproduced her most important result, stopping the disease process in about half their mice and getting the animals to recover normal functioIn March three groups reported separately in Science that they had repeated Faustman's protocols and reproduced her most important result, stopping the disease process in about half their mice and getting the animals to recover normal functioin Science that they had repeated Faustman's protocols and reproduced her most important result, stopping the disease process in about half their mice and getting the animals to recover normal functioin about half their mice and getting the animals to recover normal function.

For comparison, bacteria grown in a laboratory on Earth in normal gravity infected another group of mice.

The investigators reached this conclusion by comparing the integrity and development of the blood - brain barrier between two groups of mice: the first group was raised in an environment where they were exposed to normal bacteria, and the second (called germ - free mice) was kept in a sterile environment without any bacteria.

But in mixed groups of mutated and genetically normal male mice, there was no social hierarchy.

Monitoring groups of mice, one group receiving normal levels of protein in the diet and the other group nourished with low levels of protein for five weeks, researchers were able to uncover the damaging effect a reduction of protein can cause to the structure of this vital organ.

The cloned mice started dying off at the age of 311 days — about half the age the normal mice began their decline, the group reports in the 11 February online version of Nature Genetics.

Results showed the first two groups didn't develop PCOS, unlike the mice in the control group, whereas the test subjects with ovary ARs still contracted the disease, albeit at a lower rate than the normal mice.

But when the group checked for arrival of ILC2 cells at peripheral organs — a journey made mainly by mature ILC2s — they observed far fewer ILC2 cells in lung and intestine of mutant compared to normal mice, meaning that precursors likely require intact VHL for maturation.

To confirm that macrophages throw an entirely different molecular switch to turn on Nr4a1, the group exposed mice missing the monocyte E2 switch to a noxious toxin found in bacterial membranes, as a way of seeing whether macrophages can still mount normal inflammatory responses.

Instead, exercise seemed to reverse mitochondrial damage, causing the mitochondria in the exercise group to appear healthy like those of the normal mice.