Not exact matches
A significantly higher amount of stem cells had survived and integrated into the wound tissue in mice that had received celecoxib, and there were fewer inflammatory white blood cells and lower
levels of
cytokines in their wounds, including one
cytokine called
interleukin - 17A.
iNKT cells are also capable of producing high
levels of
cytokines such as interferon - γ and
interleukin - 4.
These cells secrete high
levels of
interleukin - 22 (IL - 22), a key
cytokine for maintaining barrier tissues during inflammation.
Although vaccination induces an inflammatory response during pregnancy, the magnitude and the duration of response is much lower and shorter, respectively, for influenza vaccination than viral infection.27 Like infection, influenza vaccination during pregnancy has been reported to induce a transient increase in the
levels of a number of proinflammatory
cytokines, including
interleukin 6, tumor necrosis factor α, and C - reactive protein.27 - 30 Studies on mice found an association between high
interleukin - 6
levels during pregnancy and abnormal behavior and brain structure.19 However, in epidemiological studies, associations between maternal
cytokine levels and ASD have been mixed.
Meditation lowers inflammation by signalling the brain to lower inflammatory
cytokines, specifically
interleukin - 6, which is a biomarker that can indicate unhealthy
levels of inflammation in the body.
Furthermore, it is usually associated with elevated
levels of pro-inflammatory
cytokines and acute phase proteins, such as interferons (IFNs),
interleukin (Il)-1, Il - 6, tumor necrosis factor - α (TNF - α), and C - reactive protein (CRP).
For example, white light exposure reduces
levels of the pro-inflammatory
cytokines TNF - α, IL - 6, interferon - gamma, and
interleukin - 12 and increases
levels of anti-inflammatory
cytokines interleukin - 10 and TGF - beta.
White adipose tissue messenger RNA
levels and serum concentrations of inflammatory
cytokines (tumor necrosis factor - α,
interleukin - 6, and monocyte chemoattractant protein - 1) were reduced by C3G, as did macrophage infiltration in adipose tissue.
The numerous flavonoids found in astragalus appear to enhance cell - mediated immunity by increasing the number of T - helper cell type 2
cytokines, increasing
levels of tumor necrosis factor and
interleukin - 6, which stimulates the activity of macrophages, responsible for killing potentially harmful cells.
People who took 1000 mg of curcumin for 6 weeks showed lower blood
levels of inflammatory
cytokines interleukin - 1β and TNF - α, as well as lower salivary cortisol.
For example, KBs were recently reported to act as neuroprotective agents by raising ATP
levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the
cytokines tumour necrosis factor - α and
interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.