Measures utilized include the Post Traumatic Stress Disorder Symptom Check List (PCLC), the Assessment Scale for Mindful Interventions (ASMI), and the Arousal Response during Trauma
Memory Reactivation (ART - MR).
Our findings provide new evidence for how spindles segment time into
memory reactivation events and reactivation blinks — when spindles are refractory.
We previously found that slow - wave phase at time of stimulation predicts this behavioral memory benefit, supporting the hypothesis that
memory reactivation is most likely during cortical upstates (Batterink, Creery & Paller, 2016).
The slow - oscillation upstates are theorized to provide a window of opportunity for memory consolidation, and
memory reactivation underlying consolidation may occur preferentially during these times.
This technique, targeted
memory reactivation (TMR), selectively enhances memory consolidation.
«Thus, direct induction of sleep spindles — for example, via transcranial electrical stimulation — perhaps combined with targeted
memory reactivation, may enable us to further improve memory performance while we sleep.»
Not exact matches
Staresina along with Scott Cairney at the University of York, UK, suspected that experimental
reactivation of
memories might lead to a surge of sleep spindles in a sleeping person's brain.
In their new paper, Shi and colleagues report that
reactivation of cocaine
memories in rats alters the expression of a protein that releases lactate from glia and enables nerve cells to take up lactate.
To deepen this segmentation and
reactivation mechanism of
memories, the researchers designed an experiment in order to recreate in a simplified way these «boundary events»; the participants had to observe a sequence of images of the same category — for example, human faces — that was interrupted by an element of a different category — for example, an object.
Both
memory T cells subtypes can be reactivated with current immunotherapy treatments, and
reactivation of both requires DC1 dendritic cells.
This is the first description of a human
memory T cell population that is susceptible to being lost through end - stage differentiation due to the combined effects of lifelong virus
reactivation in the presence of bystander differentiation - inducing factors.
The
reactivation process may also help certain
memories to become richer and stronger, as they become integrated together with other previously stored knowledge.