Plaque forms into calculus or tartar in as little as 1 to 2 days.
Not exact matches
I
formed it
into a very basic
plaque type shape.
Earlier animal studies have shown that A-beta can move
into the brain if it's injected
into the bloodstream, but scientists didn't know whether A-beta from the blood can be plentiful enough to
form plaques in the brain.
While previous investigations
into the protein's effects have used either mice in which gene expression was knocked out or transgenic animals that expressed human gene variants throughout their lifetimes, the MGH - MIND - led study used a different approach to investigate the effects of introducing the variant
forms of the protein
into brains in which
plaque formation had already begun.
A definitive diagnosis of Alzheimer's includes dementia and two distortions in the brain: amyloid
plaques, sticky accumulations of misfolded pieces of protein known as amyloid beta peptides; and neurofibrillary tangles,
formed when proteins called tau clump
into long filaments that twist around each other like ribbons.
Bacteria
form plaque that mineralizes
into calculus throughout a person's life.
To better explain this finding, the team turned to yet another protein, amyloid beta — which in its soluble
form, before building up and hardening
into plaques, has already been found to be toxic to the synapses.
Amyloid - β clumps
into whitish
plaques, tau
forms ribbons called tangles and α - synuclein creates fibrous deposits called inclusions.
«An iron
plaque forms on the surface of the roots that does not allow arsenic to go up
into the rice plant.»
The nature of those
plaques finally came
into focus in 1984, when George Glenner, a research scientist at the University of California, San Diego, identified the peptide called amyloid - beta and hypothesized that Alzheimer's was caused by «amyloidosis» of the brain, a process in which insoluble
forms of an amyloid protein accumulate.
Glenner's research eventually morphed
into the «amyloid cascade hypothesis,» which says that the formation of amyloid - beta
plaques leads to tangled
forms of another protein, tau, and ultimately to inflammation in the brain.
Improperly processed, APP is broken
into the toxic peptides that
form Alzheimer's
plaques.
The compound sticks to the free - floating
forms of the protein amyloid, which build up
into damaging
plaques in the brain.
To me it seem like
plaques form when the blood becomes «saturated» with cholesterol and it «precipitates» out in the walls of the arteries where it is eaten by immune cells which turn
into foam cells when they become engorged with cholesterol.
Aβ results from the normal cleavage of amyloid precursor protein (APP), but its accumulation and aggregation
into plaques represents the quintessential feature of AD.27 Aβ is found in orders of magnitude greater in AD brains than in healthy brains.28 This fact is noteworthy because lower concentrations of Aβ tend to stay soluble; higher concentrations
form plaques more readily.29
Glycation is a factor of glucose concentration exposure and time, with more AGEs
forming upon longer exposure to higher concentrations of glucose.33 It follows that in a body that is hyperinsulinemic, and a brain that is insulin - resistant, the peripheral hyperinsulinism will inhibit the clearance of soluble Aβ by IDE, thereby causing it to remain in the extracellular space for an extended amount of time, and the functional «hyperglycemia» in the brain will provide an elevated level of glucose — the perfect storm for glycation of Aβ and its aggregation
into insoluble
plaques.
The body perceives this
plaque as an injury and sends inflammatory cells
into the vessel walls, where they set off a cascade of events that can ultimately cause
plaque to rupture and a clot to
form over it.
Other sources say the
plaque could be
formed from excessive calcium intake that doesn't get
into bone - matter because other nutrients that help calcium get
into bone - matter are missing.
Brushing the teeth is the «gold standard» (same as for people), and every day would be great, but no less than every other day, in order to prevent the bio film (
plaque) from
forming into calculus and tartar (the hard yellow brown deposit on the teeth at the gingival margins).
Plaque begins to
form on the tooth within hours, and it must be disrupted or it will turn
into tartar.
When small bits of food are wedged between the teeth bacteria proliferate,
plaque forms and eventually it develops
into calculus (tartar).
Gum disease is the natural median result of food debris that is allowed to be inhabited and colonized by bacteria until they
form plaque and solidify
into tartar.
Gingivitis Bacteria and
plaque -
forming foods can build up on your pet's teeth and harden
into tartar, which causes inflamed gums, Gingivitis.
It takes 48 hours for
plaque to mineralize
into tartar and if you brush every day, tartar won't have the chance to
form.»
Bacteria stick to the smooth tooth surfaces
forming plaque, which thickens, mineralizes, hardens and roughens
into calculus, also known as tartar.
As this mixture builds up in her mouth, it begins to
form plaque, which eventually hardens
into tartar and discolors your kitten's teeth.
If food particles and bacteria are allowed to accumulate along the cat's gumline, it can
form plaque, which, when combined with saliva and minerals, will transform
into calculus.
Left untreated, this
plaque will
form into a hardened substance known as tartar, which can begin to
form underneath the gums of the pet, and cause damage to their gums as well as their teeth.
The crunchy kibble pieces
into which these ingredients are
formed help clean teeth and reduce
plaque.»