Sentences with phrase «protect telomeres»

Exercise is an important lifestyle choice with anti-aging effects that also helps maintain a healthy weight, possibly by producing a compound that acts to protect the telomeres.

that also helps maintain a healthy weight, possibly by producing a compound that acts to protect the telomeres.

How exactly does exercise protect our telomeres?

Recent research has shown that Astragalus may protect the telomeres from degradation.

Meditation also helps to protect our telomeres, the protective caps at the end of our chromosomes.

U. Biswas, M. Stevense, and R. Jessberger SMC1α Substitutes for Many Meiotic Functions of SMC1β but Can not Protect Telomeres from Damage.

Rai R, Chen Y, Lei M, Chang S. TRF2 - RAP1 is required to protect telomeres from engaging in homologous recombination - mediated deletions and fusions.

Intriguingly, the study also found that drugs called statins, which are better known for their cholesterol - lowering properties, appeared to alleviate the effects of telomere damage — and may even have protected telomeres against degradation.

TRF2 (Telomerase Repeat binding Factor - 2), an important shelterin component, not only protects the telomere [5, 6] but also exhibits extra-telomeric functions [7, 8].

Astragalus root is an adaptogenic herb that helps reduce stress and aging by protecting the telomeres of cells.

Why decaf coffee does not protect telomere length the researchers don't know.

That doesn't help us with the original question, What was it about the Ornish intervention that so powerfully protected telomeres after just three months?

Not exact matches

Doing so keeps the telomeres found on the ends of your DNA strands long and able to protect chromosomes from deterioration.

Telomeres are repetitive stretches of DNA that cap natural chromosome ends to protect them from being damaged or fused together during DNA replication.

Voice: The 2009 Nobel Prize in Physiology or Medicine goes to Harvard's Jack Szostak, Johns Hopkins's Carol Greider and Elizabeth Blackburn at U.C. San Francisco for their work on how chromosomes are protected by telomeres and the enzyme telomerase.

Individuals with one altered gene had longer telomeres, the caps on the ends of chromosomes that wear away as we get older, and appeared to be protected against diabetes, the researchers report.

The key to cancer cell immortality are the cell's telomeres, repetitive stretches of DNA at the ends of chromosomes that may protect the chromosomes when they divide.

Telomeres are pieces of DNA that protect the ends of chromosomes.

Telomeres, the caps of DNA which protect the ends of chromosomes, shorten every time cells divide.

Individuals carrying the variant had shorter telomeres, stretches of DNA at the ends of chromosomes that protect them from daily wear — and also aging

Telomeres are bits of DNA that protect the ends of chromosomes from unraveling or degrading.

Klingelhutz and his team immortalized immature precursor fat cells by adding in two genes from HPV (the virus that causes cervical cancer) along with a gene for part of an enzyme that controls the length of cells» telomeres — the pieces of DNA that protect chromosome tips from deterioration.

Telomeres are the caps that protect the ends of chromosomes and they shorten every time a cell divides.

Telomeres, compound structures at the end of each chromosome that protects the end of the chromosome from deterioration, are the genetic key to longevity.

The protein produced by this gene protects the chromosome ends of the DNA from damage, and controls telomere maintenance by the telomerase enzyme.

Many forms of aplastic anemia share an important link with the aging process: the shortening of telomeres, structures that protect the ends of chromosomes.

Biological age, Samani says, is related to the length of telomeres — stretches of DNA at the ends of chromosomes, which protect these precious packages of genes from daily wear and tear.

A molecular biologist born in Hobart, Australia, Blackburn is best known for her 2009 Nobel Prize — winning discovery of telomeres, caps on the ends of chromosomes that protect genetic information from damage and are thought to play an important role in aging and cancer.

In healthy cells, telomeres protect the chromosome by tucking away any overhanging ends of DNA strands to form a lasso - like structure known as a T - loop.

They found that the inactivation of POT1 caused by these mutations leads to longer and potentially unprotected telomeres, regions at the end of our chromosomes that protect chromosomes from damage.

Inflammation also erodes telomeres, the «caps» at the ends of chromosomes that protect genes from degradation, which can lead to early cell death, premature aging and even cancer.

The finding relates to telomeres, the caps that protect the tips of chromosomes when cells divide.

James Christiansen, professor of biology at Drake University in DesMoines, is studying how telomeres, the simple, non-genetic DNAsequences that sheathe the ends of chromosomes, function in reptiles.Each time a healthy human cell divides, it loses a little bit of thetelomere, until the strands are too short to protect the chromosomes.At that point the DNA in a cell begins to break down, which triggerssenescence and death.

«Telomeres function a bit like the plastic caps at the ends of shoelaces and protect the coding DNA from loss during cell division.

Telomeres are repetitive nucleotide sequences found at the end of chromosomes which protect them from deteriorating during the process of replication.

Scientists at King's College London have found that people who have previously suffered from acne are likely to have longer telomeres (the protective repeated nucleotides found at the end of chromosomes) in their white blood cells, meaning their cells could be better protected against aging.

«This suggests that statins were protecting against the worst cases of telomere degradation.

The length of the telomere «caps» of DNA that protect the tips of chromosomes may predict cancer risk and be a potential target for future therapeutics, University of Pittsburgh Cancer Institute (UPCI) scientists will report today at the AACR Annual Meeting in Washington, D.C.

Telomeres are repetitive sequences of DNA that protect the ends of chromosomes from deteriorating.

This was a key finding, as telomeres serve important functions in cell division and protecting mammalian chromosomes.

Telomeres are DNA regions at the ends of our chromosomes that protect the genetic data of cells, preventing mutations and alterations in the DNA that could potentially cause disease.

The study demonstrates that RingoA is active at telomeres — structures that protect the ends of chromosomes and where Cdk2 is also found.

The myth has a kernel of truth, because the ends of chromosomes are protected by specialized stretches of DNA called telomeres.

Two independent groups of scientists have now linked some of these cases to mutations in genes encoding telomerase, an enzyme that protects the fragile ends of chromosomes, known as telomeres.

The study, publishing online January 18 in the American Journal of Epidemiology, found elderly women with less than 40 minutes of moderate - to - vigorous physical activity per day and who remain sedentary for more than 10 hours per day have shorter telomeres — tiny caps found on the ends of DNA strands, like the plastic tips of shoelaces, that protect chromosomes from deterioration and progressively shorten with age.

Telomeres (in white) cap the ends of human chromosomes, protecting the genetic information from damage.

Telomeres are chromosome - protecting caps located on the ends of DNA strands.

Their findings on telomeres, the stretches of DNA at the ends of chromosomes that protect our genetic code and make it possible for cells to divide, suggest a potential target for preventive measures against cancer, aging and other diseases.

Much like the plastic caps on our shoelaces that keep them from fraying, telomeres protect our chromosomes and preserve our genetic information.

Telomeres are repetitive DNA sequences that protect the end of the chromosome from being recognized as sites of DNA damage.