Sentences with phrase «sfa intakes»

MUFA has, however, been associated with higher HDL cholesterol concentrations, 15,30 ⇓ reflected in lower total: HDL cholesterol and LDL: HDL cholesterol ratios, as potentially important predictors of cardiovascular risk.21 Higher SFA intakes in exchange for carbohydrate in the DELTA (Dietary Effects on Lipoproteins and Thrombogenic Activity) study were associated with a lower Lp (a) level, 31 an effect associated in the present study with higher MUFA intakes.

From the study you have mentioned, although they found a lower risk of IHD with a higher intake of SFA, they do state that these results may have been due to «small SFA intake range (IQR: 13.2 — 16.6 % of energy) at a high mean intake level (15.0 % of energy)».

Conversely, decreased SFA intake improves measures of both CVD and T2D risk.»

Not exact matches

I still believe fat (especially saturated) is very important, but ingest it like Jimmy Moore and probably we end up having some problems (Eating A LOT of SFA rich foods raises your overall PUFA intake anyways).

From their conclusions, «total mortality was inversely associated with intakes of saturated fatty acids (SFA)».

Because of the lack of dietary data on individual fatty acids in the Iranian food composition table, we were unable to compare dietary intakes of SFAs, TFAs, monounsaturated fatty acids, and PUFAs across quintiles of PHVOs and non-HVOs.

In another randomized (7), crossover saturated fat food comparison three diets equal in calories were studied to identify heart disease risk in relation to SFAs or carb intake.

I wonder if the increased intake of fats (SFAs) on the keto / low carb diet are causing this, as I have changed nothing else perhaps by increasing total cholesterol?

Agreed about the Rabbits, however, taking a look into animal fats and saturated fatty acid intake I see no direct correlation between SFA's and CHD:

Hi John, that's a good question and there are different opinions when it comes to the «ideal» saturated fat (SFA) intake.

Depression was associated with higher GI quintiles; younger age; higher BMI; less physical activity; higher intakes of SFAs, MUFAs, PUFAs, and trans fat; and lower intakes of fruit, vegetables, dietary fiber, and Healthy Eating Index score.

Model 1 was energy partition adjusted; model 2 adjusted for variables in model 1 plus age, race - ethnicity, education, income, BMI, diabetes, hypertension, myocardial infarction, stroke, cardiovascular disease, cancer, Alzheimer disease, hormone replacement therapy, physical activity, alcohol, smoking, stressful life events, social support, and energy - adjusted intakes of SFAs, MUFAs, PUFAs, and trans fatty acids; model 3 adjusted for variables in model 2 plus energy - adjusted intakes of fruit, vegetables, legumes, nuts / seeds, and fiber and Healthy Eating Index score.