Joslin Diabetes Center, «
Understand cell stress pathways in modulating glucagon release,» led by Dr. Rohit Kulkarni
Not exact matches
However, the prime minister
stressed the public at large must
understand and accept new scientific developments such as the MMR vaccination, stem
cell research and GM foods.
His discoveries are vital for
understanding how
cells respond to
stress and infection.
«By
understanding how
stress accelerates invasion in aggressive breast tumor
cells, this work will inform future studies into whether beta - blockers could be a useful adjuvant therapy in the treatment of some aggressive breast cancers.»
But until he
understands how the
cells are
stressed in the first place, he won't know much: «We frankly don't have a clue as to how much microwave radiation is needed to cause irreversible damage to cellular proteins.
On the one hand, the researchers now
understand which mechanisms the
stressed T
cells need to protect themselves against NK
cells.
Several functions vary among the members of this group of bacteria, such as metabolism of nitrogen, respiration,
cell wall and capsule,
stress response, secondary metabolite biosynthesis, regulation and
cell signaling; this variation in gene functions could help us
understand the ability of colonization and adaptability of these rhizosphere - colonizing bacteria.
Altieri and his team are currently focused on three programs to
understand the biology of survivin with respect to
cell cycle progression, in particular, mitosis,
cell survival and the cellular
stress response and how exploiting these pathways may provide new cancer therapeutics.
Understanding how yeast cope with osmotic
stress is useful to
understand how human
cells respond to medical treatments, but the principles are also applicable to many other fields, including agriculture.
By studying these processes, we hope to gain a better
understanding of how
cells respond to various
stresses to ensure the normal development of tissues and uncover novel pathways that could be exploited to prevent disease progression.
The geometric homeostasis of mature epithelia observed at short enough time - scale thus raises the problem to
understand how
cells, if considered as noisy individual actuators, do adapt across individual intercellular contacts to locally balance their time - average contractile
stress.
Through a complicated and little
understood signaling machinery — a domino effect of molecular reactions in a
cell that ultimately produces a certain signal — Natural Killer
cells then destroy such»
stressed»
cells.
So, if I
understand correctly, what you're saying is that when your body feels as though it's constantly
stressed out, whether from exercise or some other stressor, what can happen is that it switches on pathways to develop insulin resistance so that, rather than putting food stuff into, say, muscle storage or liver storage, you might actually create new fat
cells or put glucose, you know, that has been converted into triglycerides, et cetera, into fat
cells so that your body has storage to rely upon in times of need even though you're not necessarily in a time of need.
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