Sentences with phrase «university cystic fibrosis»

The 65Roses team is a group of individuals who run and raise money to financially assist cystic fibrosis patients cared for at the Augusta University Cystic Fibrosis Center.

Not exact matches

As a postdoc at the University of Iowa Carver College of Medicine in Iowa City, Rogers used the pig model to better understand the genetic underpinnings of cystic fibrosis — research that earned him a first - author publication in Science in the 26 September 2008 issue (p. 1837).

A study published by scientists at University of Massachusetts Medical School and the University of Alabama at Birmingham provides insight into the mechanism of action of the drug ataluren, which is showing promise in treating Duchenne muscular dystrophy and cystic fibrosis.

Hughes ran into conflict with the university and his former sponsor, the National Institutes of Health (NIH), over his efforts to diagnose genetic diseases — such as Tay - Sachs disease and cystic fibrosis — in test - tube embryos scheduled for implantation (Science, 24 January, p. 472).

Researchers from Queen's University Belfast have discovered why antibiotics for treating people with cystic fibrosis are becoming less effective and how fat soluble vitamins might offer a viable solution.

Meanwhile, a collaboration between David Schaffer of the University of California at Berkeley and pulmonologist Joseph Zabner of the University of Iowa has scientists excited about the possibility of curing cystic fibrosis using another variant of the virus to deliver healthy genes to lung tissue.

Vice-Chancellor of Queen's University Belfast, Professor Patrick Johnston said: «Although this is good news in one sense, these figures pose a major challenge to healthcare providers in that adult cystic fibrosis services will need to be developed quickly and effectively to meet demand.

The number of people living with cystic fibrosis into adulthood in the UK is expected to increase dramatically — by as much as 80 per cent — by 2025, according to a Europe - wide survey, the UK end of which was led by Queen's University Belfast.

Lead UK author on the study, Professor Stuart Elborn, Dean of the School of Medicine, Dentistry and Biomedical Sciences at Queen's University Belfast, said: «The estimations we have made show very positive news for cystic fibrosis patients as the average survival age is increasing.

Now, scientists at Boston University's Center for Regenerative Medicine (CReM) have announced two major findings that further our understanding of this process: the ability to grow and purify the earliest lung progenitors that emerge from human stem cells, and the ability to differentiate these cells into tiny «bronchospheres» that model cystic fibrosis.

To better understand the role of streptococci in cystic fibrosis, Jessica Scoffield of the University of Alabama at Birmingham and colleagues grew several biofilms in dishes and in fruit flies.

Diana W. Bianchi, a reproductive geneticist at Tufts University School of Medicine in Boston, Mass., agrees that fetal genomes could inform treatment in the future, but she questions the need to replace conventional genetic testing with whole - genome sequencing to diagnose genetic diseases such as beta - thalassaemia and cystic fibrosis.

Francis Collins of the University of Michigan, who was a co-discoverer of the cystic fibrosis gene, told a working group on genetics that if it was not «very forward looking», society could be caught off guard by the rapid pace of progress.

Ben Wilfond, a paediatrician from the University of Arizona, asked: «If there's not much interest in terminating these pregnancies, do we want to spend our health care resources on cystic fibrosis screening?»

The chances of that are so miniscule that it suggests alternative explanations, says medical geneticist Garry Cutting of Johns Hopkins University in Baltimore, Maryland, who has found mutations that reduce the severity of cystic fibrosis through a different approach.

Over the last few years our lab at the University of California, San Francisco (UCSF) has used HTS to discover activators and inhibitors of the cystic fibrosis transmembrane conductance regulator (CFTR) chloride channel.

«Inhibiting DGKi seems to reverse the effects of cystic fibrosis, but not block ENaC completely,» says Margarida Amaral from the University of Lisboa, «indeed, inhibiting DGKi reduces ENaC activity enough for cells to go back to normal, but not so much that they cause other problems, like pulmonary oedema.»

Professor Stuart Elborn, Dean of the School of Medicine, Dentistry and Biomedical Sciences at Queen's University, and the European lead on the study, said: «This is a very significant breakthrough for people with cystic fibrosis.

Scientists at the European Molecular Biology Laboratory (EMBL) in Heidelberg and Regensburg University, both in Germany, and the University of Lisboa, in Portugal, have discovered a promising potential drug target for cystic fibrosis.

The research team included Scott H. Randell, Ph.D., a leading expert in the culture of primary lung cells, from the Cystic Fibrosis / Pulmonary Research and Treatment Center at the University of North Carolina at Chapel Hill.

One of the study's coauthors, John Engelhardt of the University of Iowa, made the first nearly 10 years ago to study cystic fibrosis.

Michael J. Welsh, MD Howard Hughes Medical Institute Investigator Roy J. Carver Biomedical Research Chair in Internal Medicine and Molecular Physiology and Biophysics Director, Cystic Fibrosis Research Center Director, Institute for Biomedical Discovery University of Iowa

The Cystic Fibrosis Center at the University of Michigan C.S. Mott Children's Hospital has the largest program of its kind in the state, offering a multidisciplinary approach for complete care.

Dr. Welsh is a Howard Hughes Medical Institute Investigator and Roy J. Carver Biomedical Research Chair in Internal Medicine and Molecular Physiology and Biophysics, Professor of Neurosurgery, Director of the University of Iowa Cystic Fibrosis Research Center, and Director of the University of Iowa Institute for Biomedical Discovery at the Roy J. and Lucille A. Carver College of Medicine of the University of Iowa.

The University of Michigan CF Center was a recipient of the 2008 - 2009 Cystic Fibrosis Foundation's Quality Care Award recognizing outstanding QI processes and accomplishments.

Mathis participated in the half marathon with his wife in support of their friend's son who is a patient at Augusta University's Cystic Fibrosis Center.

The University of Michigan's Cystic Fibrosis Center supports CF patients over 21 years of age and is accredited by the Cystic Fibrosis Foundation.

A team of scientists from the UNC School of Medicine and North Carolina State University (NCSU) has developed promising research towards a possible stem cell treatment for several lung conditions, such as idiopathic pulmonary fibrosis (IPF), chronic obstructive pulmonary disease (COPD), and cystic fibrosis — often - fatal conditions that affect tens of millions of Americans.

The Adult Cystic Fibrosis Clinic, located at the University of Michigan's Cystic Fibrosis Center is accredited by the Cystic Fibrosis Foundation and was created to support patients over 21 years of age to provide dedicated, comprehensive, and coordinated care by a multidisciplinary team of medical professionals that includes doctors, nurses and other health professionals, including dieticians, social workers and physical therapists.

The University of Michigan's Adult Cystic Fibrosis Clinic at U-M's Cystic Fibrosis Center supports CF patients over 21 years of age and is accredited by the Cystic Fibrosis Foundation.

THE MOUSE MODEL FOR CYSTIC FIBROSIS, as with models for many diseases, owes its existence to a technique called gene targeting, which was developed in the 1980s by Mario Capecchi, a professor of human genetics and biology at the University of Utah who won the 2007 Nobel Prize in Physiology or Medicine for his work.

A team of researchers, including a number from the University of North Carolina at Chapel Hill School of Medicine, have pinpointed regions of the genome that contribute to the debilitating lung disease that is the hallmark of cystic fibrosis.