For instance, «
What immune genes do tortoises have to fight pathogens?
Not exact matches
Scientists believe that is
what happened during a 1999 French
gene therapy trial on a group of 10 young children with X-SCID, an
immune deficiency disorder known as boy - in - the - bubble syndrome.
«As you look for methods to discern complex
immune responses in human cells, more and more people look at
what genes are turned on with infections or vaccination procedures.»
To understand
what was unique about the LAD patients» disease, the researchers examined their
immune system - related
genes and proteins.
Scientists believe that's
what happened during a 1999 French
gene therapy trial on a group of 10 infants with X-SCID, an
immune deficiency disorder known as boy - in - the - bubble syndrome.
What's more, Discher says, unpublished work from his lab suggests that adding the molecular passports to viruses that deliver
genes in
gene therapy also helps them avoid
immune detection.
When the team looked at
what gene signatures the RORγt - dependent ILCs expressed, they found high expression of major histocompatibility complex class II (MHCII) protein, which some cells use to display foreign proteins to the
immune system and directly interact with T cells.
But
what about the
genes behind the
immune system?
In this case, as in a recent case of
gene borrowing between weeds (ScienceNOW, 13 November 2008), «humans were indirect agents in promoting these events,» says Enrico Coen, a geneticist at the John Innes Centre in Norwich, U.K. Geneticist Sheila Schmutz of the University of Saskatchewan in Canada wonders
what else the dogs might have contributed to wolves, say, to metabolism or
immune system function.
She found a
gene for
what's known as the major histocompatibility complex (MHC)-- cell surface molecules that help the
immune system recognize foreigners — that was remarkably similar to one in humans that allows infected people to keep the virus in check for decades.
The cells then produce more of
what the
genes code for: cytokines, which are messenger molecules that circulate in the blood and activate
immune cells.
«Following the trajectory of type 1 diabetes in the mice, and looking at
what genes become active in the microbes and in the
immune system, we think we can pinpoint some of these mechanisms that are important, if indeed the microbiome is causing diabetes,» Kostic says.
The more I look at how people's
immune response happens based on this presence of this
gene or that
gene, the more complex it is and the more it's really apparent that
what you do with your environment is so much more important than the
genes you have, for most
genes.
Dr. Peter Osborne:
What genetic testing does, HLA - DQ2 — there are two genes, there's HLA - DQ2, alpha 1 and HLA - DQ beta 1 — these are immune genes and they're job is — and they're on Chromosome 6, their job is to produce an antenna that sits on the surface of the white blood cell, and the — the job of this antenna is to identify what is good versus what is
What genetic testing does, HLA - DQ2 — there are two
genes, there's HLA - DQ2, alpha 1 and HLA - DQ beta 1 — these are
immune genes and they're job is — and they're on Chromosome 6, their job is to produce an antenna that sits on the surface of the white blood cell, and the — the job of this antenna is to identify
what is good versus what is
what is good versus
what is
what is bad.
Reduce the inflammation, reduce the activation of your
immune system, which is
what turns on the
genes.