Sentences with phrase «xanthoria elegans»

The entire genome of the tiny nematode (Caenorhabditis elegans) also has been sequenced as a ta - ngen = tial study to the human genome project.

We might think that a chimpanzee is exercising free will when it chooses to chomp on a banana, or a cat when it rips up your sofa, but what about the roundworm called Caenorhabditis elegans — a simple creature made of only 959 cells?

Her interest was piqued: Upon graduating in 1999 she joined the lab of Ding Xue at the University of Colorado, Boulder, to study how cell death is regulated in the model organism Caenorhabditis elegans.

Many of the most dramatic forms of selfishness, the murderous cheats, come from bacteria, so Werren welcomes the C. elegans scam as a rare case discovered in animals.

The big community of researchers regularly studying C. elegans had missed discovering the selfish role for a simple reason: The main lab strain of nematodes carries the selfish element, explains study coauthor Eyal Ben - David.

POISONED BY MOM A C. elegans nematode that inherits a gene for the antidote to a maternal toxin grows a normal feeding tube (shown first).

Biologists have for decades discussed how two genes in the familiar lab nematode Caenorhabditis elegans might help embryos build their organs.

That's how many lab C. elegans nematodes grow a normal body (left) with a wide, inner feeding tube (partly visible toward worm top).

Led by graduate student Patrick O'Hern, the team at Brown and the University of Cologne in Germany uncovered a complex cause - and - effect sequence in both C. elegans worm and mouse models of SMA.

MOMMIE DEAREST Covertly poisoning offspring appears to be a common part of motherhood in the C. elegans nematode (shown) that stars in biology labs worldwide.

A maternal - effect selfish genetic element in Caenorhabditis elegans.

Lichens Hang In There Lichens like Xanthoria elegans thrive in harsh environments like Antarctica and the high peaks of the Himalayas.

X. elegans survived 18 months strapped to the outside of the ISS in the EXPOSE experiment.

Additionally, C. elegans continued to change its behavior even after the fear - chemical was removed.

In findings published today in the journal Cell, postdoctoral fellow Hongyun Tang and Professor Min Han, both of CU Boulder's Department of Molecular, Cellular and Developmental Biology and the Howard Hughes Medical Institute, detail how fat levels in a tiny soil - dwelling roundworm (C. elegans) can tip the balance between whether the worm makes eggs or sperm.

«Radical signal to the progeny: Hlobin protein found in roundworm model Caenorhabditis elegans that is able to generate free radical signals.»

The research was performed on C. elegans, tiny roundworms that typically live an average of two weeks.

Magnified, the slime comes alive as hundreds of translucent worms, known as Caenorhabditis elegans, slither to and fro.

In 1990, they sent her to Syracuse University in New York state, her father's alma mater, where she was introduced to C. elegans and majored in biology and ceramics — the pot - throwing kind, not materials science.

«I saw a picture of the C. elegans embryo with the microtubules all lit up,» she says.

Sasha De Henau, a researcher at the Biology Department of Ghent University in Belgium recently discovered a globin protein in the roundworm model Caenorhabditis elegans that is able to generate such free radical signals.

In plants and Caenorhabditis elegans, two distinct populations of small RNAs have been proposed to participate in RNAi: «Primary siRNAs» (derived from DICER nuclease - mediated cleavage of the original trigger) and «secondary siRNAs» [additional small RNAs whose synthesis requires an RNA - directed RNA polymerase (RdRP)-RSB-.

Data published by the International Human Genome Sequencing Consortium indicate that somewhere between 113 and 223 genes present in bacteria and in the human genome are absent in well - studied organisms — such as the yeast Saccharomyces cerevisiae, the fruit fly Drosophila melanogaster and the nematode Caenorhabditis elegans — that lie in between those two evolutionary extremes.

C. elegans, which contains only 302 neurons, has a natural predator — another worm called Pristionchus pacificus, which bites and kills C. elegans.

The researchers discovered that by exposing C. elegans to chemicals that are excreted by P. pacificus, they could elicit a fear - like response.

The two postembryonic touch receptor neurons in the nematode Caenorhabditis elegans arise from essentially identical cell lineages and have the same ultrastructural features.

Strains of the lab workhorse roundworm C. elegans that lived longer added more time being frail and had the same portion of their lives being healthy as normal worms.

The team at Salk started with a simple creature, the microscopic worm called Caenorhabditis elegans.

When it encounters these predator - excreted chemicals, C. elegans rapidly reverses direction and crawls away.

C. elegans comes in two sexes: males and hermaphrodites.

The fungal toxin binds to specific sugar structures which occur on the surface of intestinal cells of the nematode Caenorhabditis elegans.

To understand how a single cell knows when, where, and how to divide, Skop studies the embryos of Caenorhabditis elegans, the roundworm.

«In this study, we used the small roundworm C. elegans as a model to show that autophagy in the intestine is critical for lifespan extension,» said Malene Hansen, Ph.D., associate professor in SBP's Development, Aging, and Regeneration Program and senior author of the study.

For the study, the researchers incubated eggs from the Trachemys scripta elegans, a semi-aquatic turtle, under different temperature and moisture regimes to study the effect of the two environmental factors on developmental rate, egg mass, embryo mass and length, and sex ratio.

As California Institute of Technology neuroscientist Christof Koch noted in narrating the wiring diagram of the entire nervous system of Caenorhabditis elegans, we are clueless in understanding how this simple roundworm «thinks,» much less in explicating (and reproducing in a computer) a human mind billions of times more complex.

A comparative analysis of the genomes ofDrosophila melanogaster, Caenorhabditis elegans, and Saccharomyces cerevisiae — and the proteins they are predicted to encode — was undertaken in the context of cellular, developmental, and evolutionary processes.

Many of these future studies will also employ C. elegans.

Nath is studying sleep in the worm Caenorhabditis elegans, but whenever he presented his work at research conferences, other scientists scoffed at the idea that such a simple animal could sleep.

Researchers studying the nematode worm C. elegans have discovered a new mechanism by which the millimeter - long critter flexes its pooping muscles.

With worms as his subjects, he plumbs the cellular mechanisms driving the complex protein interactions regulating lifespan, some of which — remarkably — have been conserved through evolution all the way from his microscopic Caenorhabditis elegans to us.

But to my mind that is not how it has turned out — rather the reverse with a near equivalence of a modest 20,000 genes across the vast range of organismic complexity from the millimetre - long Caenorhabditis elegans to the 60 - trillion - celled Homo sapiens.

One of his colleagues studies aging in nematodes (Caenorhabditis elegans), and he's considering changing his model organism.

Using one of the great advantages of C. elegans, they were able to visualize the influx of calcium that occurred in a neuron in response to stimulus in a living worm.

These results showed that different genes control the life span of C. elegans and the length of reproductive time.

C. elegans with a green fluorescent protein (GFP) marker in the ASH neuron, the neuron that senses high pH.

Already, researchers have used CRISPR / Cas9 to edit genes in human cells grown in lab dishes, monkeys (SN: 3/8/14, p. 7), dogs (SN: 11/28/15, p. 16), mice and pigs (SN: 11/14/15, p. 6), yeast, fruit flies, the worm Caenorhabditis elegans, zebrafish, tobacco and rice.

While completing her postdoc, Murphy began to study C. elegans mutants that could live and reproduce twice as long as normal worms.

Members of OIST's Information Processing Biology Unit, led by Prof. Ichiro Maruyama, have recently uncovered the sensor for alkaline environments, or high pH, in C. elegans.

Most mutations in C. elegans affect both life span and reproduction, which had led scientists to believe that body cells and female reproductive cells aged according to the same clock.

In a new study, Murphy, a molecular biologist at Princeton University, showed that long - lived bodily, or somatic, cells in Caenorhabditis elegans, a one - millimeter nematode commonly used as a model for aging studies in labs, activate genetic pathways completely separate from those found in long - lived egg, or oocyte, cells.