Sentences with phrase «about cancer cells»

I know it's possible to get back into ketosis quickly but I worry about the cancer cells feeding on the carb up glucose

The conventional wisdom about cancer cells is that they are masters of camouflage, invisible to the immune system.

Before somebody comes along and says the same thing about a cancer cell, the big caveat is that if its a part of the body that is hurting you, then it should go.

Not exact matches

(There are good write - ups about this in the journals Science and Cell, and in Paul Goldberg's authoritative The Cancer Letter.)

That includes cell phone numbers of and sensitive information about cancer patients and their families.

Speaking of Novartis — the company's experimental CTL019, which is expected to be the first approved drug in a revolutionary new cancer treatment space that turns the body's own immune cells into cancer - killers, is already facing some apprehension from doctors and patient groups who are worried about its eventual pricing.

And while there may be benefits to reducing your cell phone use, there's no reason to panic about cancer.

About Nohla Therapeutics Nohla Therapeutics is a leading developer of off - the shelf cell therapies for the treatment of cancer and other critical diseases.

Hoping to learn something about how the human body defends itself against cancer, he had zeroed in on a complex regiment of lymphocytes called T cells, common to the immune systems in both mouse and man.

Swiss pharmaceutical giant Novartis has made waves with a drug pipeline that includes one of the most talked - about experimental cancer therapies in recent years — a treatment called Kymriah that reconfigures the body's own immune cells to become aggressive blood - cancer killers.

giant Novartis has made waves with a drug pipeline that includes one of the most talked - about experimental cancer therapies in recent years — a treatment called Kymriah that reconfigures the body's own immune cells to become aggressive blood - cancer killers.

This new kind of approach to fighting blood cancers is truly personalized; immune T - cells are extracted from patients, genetically tinkered to home in on an destroy cancerous cells, multiplied in a lab, and then jolted back into the patient's body within about two weeks.

As a cancer researcher, do you think the mechanisms of tumor growth are somehow changing to come into line with your perceptions, or is it possible that the process of our learning more about DNA mutations and cell architecture and nutrient exchange and epigenetic effects make it possible for us to inch ever closer to understanding that which is already going on under our noses?

Specifically, when capsaicin frequently binds to receptors within the human central nervous system's TRPV1 channel (the sensory receptor system for pain and heat detection), these receptors deplete and this depletion results in a whole host of benefits for the central nervous system at large, including terminating cancer cells, increasing the metabolic rate and digestive efficiency, increasing circulatory blood flow, and combatting inflammation, and making you feel better about the world.

You have probably heard a number of rumors about the effects of technology on your health; that having a cell phone next to your head can cause brain cancer, for example, or that pregnant women shouldn't stand near a microwave that is running because it may harm the fetus.

I had my follow - up CT scan about a month ago to check and make sure that I had no more growth and no more active cancer cells.

After being a nurse in the PICU, I thought about all of the genetic disorders my patients had encountered, (i.e. various cancers, cystic fibrosis, muscular dystrophy) and I wondered if there were anyway stem cells could have cured or at least improved their conditions.

As researchers learn more about genetic profile of various cancers, other work is charging ahead to deliver personalized vaccines targeted to a patient's own tumor cells

Learning more about how cancer cells move around the body is also providing scientists with new insights that could thwart the spread of the disease.

When Gupta's team added salinomycin to cultured human breast - cancer cells, it was about 100 times as effective at killing the cancer stem cells as popular anti-cancer drug, paclitaxel.

All are examples of translational researchers converting molecular knowledge about specific cancer cells into effective, targeted therapies.

Human papillomavirus 16 accounts for about half of all cervical cancers, but researchers reporting September 7 in the journal Cell have found that not all infections are equal.

The findings, in mice and in human stomach cells, also raise questions about how cancer cells may evade treatment.

But a new study suggests that mature cells also play a key role in initiating cancer — a finding that could upend the way scientists think about the origins of the disease.

Her next step is to use the technique to find, map and learn more about cancers and stem cells.

The important thing about the zebra mussel is to not consider each one as an individual organism but instead, like a cancer cell, part of a greater scourge that metastasizes as fast as currents flow.

This new insight into how chromosomes are disassembled and reassembled during cell division will allow researchers to begin answering basic questions about epigenetic inheritance, as well as human disease such as chromosome disorders and cancer.

The tissue barrier, about four cells deep, was made from «BeWo» human cancer cells.

In testing on two mouse cell lines, the drug combination reduced the viability of cancer cells by about 50 percent when compared with everolimus alone, according to the findings.

Porter and June are now participating in a new trial, funded by the National Cancer Institute, that will administer about a dozen patients a fifty - fifty mixture of cells from the Penn and Sloan - Kettering groups.

Óscar Llorca and his team have used this technique to learn about the structure and functioning of a complex system called R2TP, which is involved in various key processes for cell survival such as the activation of the kinases mTOR, ATR and ATM, proteins that are the target of various cancer drugs currently being developed.

However, in the wake of fatalities from gene therapy and other technologies, as well as the potential for cancers associated with stem cell transplants, governments are understandably nervous about safety issues — not to mention the ethical maze of tinkering with fledgling life.

About ten years ago, research results showed that things are not quite as simple as that: «Under most conditions, H2O2 is not an undesired side product but rather an essential chemical messenger that plays an important role in regulating the way in which body cells respond to signals from outside such as hormones and growth factors,» says Dr. Tobias Dick of the German Cancer Research Center (Deutsches Krebsforschungszentrum, DKFZ).

As cell phones and smart phones take over more communication, information and entertainment functions, concerns about their possible role in causing brain cancer are buzzing on the airwaves.

Three types of colon cancer cells grew faster, producing about 50 to 60 percent more cells within 24 hours, with the bacteria than they did when cultured with no bacteria or with a harmless, milk - fermenting bacterium called Lactococcus lactis.

The time needed for breast cancer metastases (secondary lesions caused by cells that have escaped from the original tumour) to develop varies between patients, and little is known about the mechanisms that govern latency (the dormant state of cells that have already spread through the body).

Until now, little was known about how oxygen - deprived cancer cells bound together and interacted to spread.

Learn about the latest approaches to assessing cancer - immune interactions («immune profiling») using FFPE tissue sections, multiplexed immunofluorescence, and cell phenotyping software

When The Cancer Genome Atlas Research Network reported its genomic profiling of clear - cell kidney tumours, about one - quarter of participants (126 patients) had been operated on at Memorial Sloan Kettering3.

Until now, little was known in preclinical models about the mechanisms that allow breast cancer cells to leave the latent state and even less is known in patients,» explains Roger Gomis, head of the Growth Control and Cancer Metastasicancer cells to leave the latent state and even less is known in patients,» explains Roger Gomis, head of the Growth Control and Cancer MetastasiCancer Metastasis Lab.

The papillary cell type represents only about 1 in 10 of these; other cancers are even less common.

This paracrine effect was brought about by the tumor suppressor Par - 4, which specifically kills cancer cells.

Lee's study results, which appear in the July 16, 2015 issue of Nature Communications, revealed new understanding about how 14 -3-3 sigma — a cell cycle «controller» - regulates cancer metabolic programming, thus protecting healthy cells from turning into tumor - producing factories.

When Kaufman, Zon and colleagues looked to see what was different about these early cancer cells, they found that crestin and the other activated genes are the same ones turned on during zebrafish embryonic development — specifically, in the stem cells that give rise to the pigment cells known as melanocytes, within a structure called the neural crest.

Lee believes that the study findings provide additional insight about the «crosstalk» between cancer metabolism and cell cycle.

It is difficult to detect cancer cells in the blood at an early stage: About one malignant cell is encountered per billion of healthy cells.

The team is also planning further research to find out more about why pancreatic cancer cells rely on PAK4 and what other proteins and pathways PAK4 is interacting with to drive cell growth and migration.

However, little is known about the specific stem cell - related effects by which ONC201 inhibits cancer stem cells from renewing.

The finding could reveal much about how cells control gene activity, and also illuminate cancer, multiple sclerosis, and other diseases spurred by faulty gene expression.

The three - year study included cell culture studies at Rice as well as a detailed analysis of gene - expression profiles of more than 500 patients from the Cancer Genome Atlas and protein - expression profiles from about 200 MD Anderson patients.