Not exact matches
Nishimura's team proposes that the same process leads to the reduction in stem cells in the follicles of older
people, especially
as DNA damage
accumulates as we
age.
For the first time, scientists have confirmed the long - standing hypothesis that
as people age, they
accumulate gene mutations in their mitochondria — cells» energy source.
Amyloid — an abnormal protein whose accumulation in the brain is a hallmark of Alzheimer's disease — starts
accumulating inside neurons of
people as young
as 20, a much younger
age than scientists ever imagined, reports a surprising new Northwestern Medicine study.
Amyloid — an abnormal protein whose accumulation in the brain is a hallmark of Alzheimer's disease — starts
accumulating inside neurons of
people as young
as 20, a much younger
age than scientists ever imagined.
Health improvement (allowing to post - pone / escape the diseases and thus live, healthier / disease - free longer, but not above human MLSP of around 122 years; thus these therapies do not affect epigenetic
aging whatsoever, they are degenerative
aging problems not regular healthy
aging problem (except OncoSENS - only when you Already Have Cancer - which cancer increases epigenetic
aging, but cancer removal thus does not change anything / makes no difference about what happens in the other cells / about what happens in the normal epigenetic «
aging» course in Normal non-cancerous healthy cells) Although there is not such thing
as «healthy
aging» all
aging in «unhealthy» (
as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage
accumulating) that it does not affect their quality of life (enough yet), that is «healthy
aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making
people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their
age), and both are correlated to MLSP).
The RTM hypothesis is that the immune systems of
people who reach the
ages at which
AS, beta - amyloid, and other pathological aggregates begin to
accumulate would generate antibodies against their abnormal form, and that
people who remain free of these specific diseases might be producing antibodies that are particularly effective at keeping these aggregates at bay.
Cells with similar characteristics
accumulate during normal
aging as well
as in younger
persons infected with human immunodeficiency virus, suggesting that the process of replicative senescence is not an artifact of cell culture but is also occurring in vivo.
The criticism points out that,
as people age, plaque
accumulates in their arteries regardless of what they're eating.
Studies have shown that
as people age they continuously
accumulate toxic metals: lead, mercury, aluminum, iron, cadmium, and arsenic, among others.
Most
people of every
age would have trouble replacing their possessions if they were lost, but seniors are particularly vulnerable to loss, because they have
accumulated so much over time and because they are generally living on a pension or other fixed income and monthly expenses are already difficult enough to meet
as it is.