When Lehningers says insulin inhibits fatty
acid mobilization that's pretty much the equivalent of what Williams is saying about insulin inhibiting lipolysis.
Doing so will keep your body in a perfect hormonal state for efficient fatty
acid mobilization, and will spare you from the negative effects of chronic high insulin levels.
What this means is that consuming alcohol will inhibit fatty
acid mobilization.
By depleting glycogen stores in several muscles, you enable your body to keep insulin levels under control the rest of the day so that fatty
acid mobilization can occur.
(B)-- Because your hormones and metabolism are all in the perfect alignment for fatty
acid mobilization
Focus on using exercise to create a favorable metabolic environment for fatty
acid mobilization, and then create a calorie deficit using your diet to lose fat.
«Lipolysis» (the conversion of fatty acids) and «ketogenesis» (creation of ketone bodies) increase significantly due to fatty
acid mobilization and oxidation.
Both HSL and ATGL are the rate - limiting enzymes needed for fatty
acids mobilization (Schweiger et al., 2006).
Not exact matches
It is also helpful for fat
mobilization purposes, so for those who are on a fat loss diet, you don't want to be missing out on this particular amino
acid.
These include elevations in non-esterified fatty
acid (NEFA)
mobilization (typically greater in women) and the secretion of counter-regulatory hormones, which act to oppose insulin action and impair glucose disposal.57 Previous works have noted similar deteriorations in glucose tolerance in response to a mixed test meal 58, intravenous and oral glucose loads 58, albeit following longer fasting intervals of 72 to 96 hours.
The actual effect on the body may differ, since body's own acidity level is a product of other factors and processes, such as the rate of mineral absorption, presence of other metabolites, and body's own
acid / alkaline regulatory actions, based on the selective excretion or
mobilization of acidic / alkaline substances.
Mobilization of visceral adipose tissue related to the improvement in insulin sensitivity in response to physical training in NIDDM: effects of branched - chain amino
acid supplements.
MCT digestion,
mobilization, metabolism, and conversion to ketone bodies: Unlike longer chain common fatty
acids, MCFA are not stored in adult adipose tissue, but rather, preferentially converted in the liver, to the ketone bodies (KBs) beta - hydroxybutyrate (BHB; technically not a KB because it does not have a keto molecular group) and acetoacetate (AcAc).
I think it is most appropriate to borrow the words from someone far smarter and well versed on this topic here (Dr. Kevin Hall), «A logical consequence of the carbohydrate - insulin model is that decreasing the proportion of dietary carbohydrate to fat without altering protein or calories will reduce insulin secretion, increase fat
mobilization from adipose tissue, and elevate oxidation of circulating free fatty
acids.
These powerful emotions trigger adrenal hormone release — the physiological effects of which include activation of adipocyte lipase (resulting in
mobilization of free fatty
acids) and partial inhibition of protein synthesis, i.e., the plasma amino
acids which are normally (readily) utilized by nonmalignant cells for protein synthesis are only partially used resulting in an increase in the availability of amino
acids to meet tumor cell metabolic needs.