Sentences with phrase «after spinal cord injury»

The phrase "after spinal cord injury" refers to a situation where an individual has experienced some form of damage or trauma to their spinal cord. Full definition
Short - term peripheral nerve stimulation may be a new approach to preventing long - term changes in nerve and muscle function and improving rehabilitation outcomes after spinal cord injuries.
The findings may guide future efforts to restore other functions lost after spinal cord injury.
Simply put, this could mean restored activity and ability in parts of the body that stopped working after spinal cord injury in the near future.
Physical therapy aimed at rebuilding neural pathways can often improve a patient's level of functioning in the months and years after a spinal cord injury.
Deep brain stimulation is not the only form of electrical stimulation that could help restore movement after a spinal cord injury.
We're using a neural device to make long - term changes in neural circuits after spinal cord injury.
The symptoms and specific injuries that you will encounter after a spinal cord injury will depend on which part of your spinal cord is affected.
The results may guide future efforts to restore functions lost after spinal cord injury.
This image depicts extension of human axons into host adult rat white matter and gray matter three months after spinal cord injury and transplantation of human induced pluripotent stem cell - derived neurons.
While mammals lack the ability to regenerate nervous system tissue after spinal cord injury, zebrafish can regenerate such tissue.
The state's own Wadsworth Center in Albany will receive over $ 440,000 for research into urinary tract dysfunction after spinal cord injury.
New research in mice indicates that a drug commonly used to suppress the immune system in recipients of organ transplants may also reduce tissue damage and neuropathic pain after spinal cord injury.
«Interneurons can reroute after spinal cord injuries, which makes them a promising therapeutic target,» said senior author Todd McDevitt, PhD, a senior investigator at Gladstone.
Neuronal survival within spinal cord gray matter was determined using NeuN immuno - reactivity as a marker of surviving spinal cord neurons after spinal cord injury [34].
This work shows for the first time that IL - 37 suppresses the inflammatory response after a spinal cord injury and minimises spinal tissue degeneration and functional disabilities.
The CSNE is now recruiting more participants with challenges using their hands and arms after spinal cord injury, and the center will continue to test this novel form of stimulation as an innovative approach to improving hand and arm function.
«These findings highlight the need for a deeper understanding of glial cell function after spinal cord injury,» he said.
A new discovery at the University of Alberta will fundamentally alter how we view spinal cord function and rehabilitation after spinal cord injuries.
They also had less nerve cell death and lower levels of proteins known to induce inflammation after spinal cord injury and worsen neural damage (Neurobiology of Disease: DOI: 10.1016 / j.nbd.2010.04.003).
«Pressure ulcers are an unfortunately common complication after spinal cord injury and cause discomfort and functional limitations,» said co-author Gwendolyn A. Sowa, M.D., Ph.D., associate professor of physical medicine and rehabilitation, Pitt School of Medicine.
Rapamycin treatment four hours after spinal cord injury significantly improved locomotor function and reduced mechanical and thermal hypersensitivity in the hindpaws.
In their previous work, investigators at the Tohoku University Graduate School of Medicine in Japan found that rapamycin treatment can reduce nerve damage and locomotor impairment after spinal cord injury.
«Technique to promote nerve regeneration after spinal cord injury restores bladder function in rats.»
«Transplant drug may provide benefits after spinal cord injury
Neuroscientists found that spinal blood flow in rats was unexpectedly compromised long after a spinal cord injury (chronically ischemia), and that improving blood flow or simply inhaling more oxygen produces lasting improvements in cord oxygenation and motor functions, such as walking.
He also showed in 2012 that paralyzed rats could recover after spinal cord injury after a few weeks of rehabilitation, combining electro - chemical stimulation and physiotherapy that uses a robotic harness.
Here Dr. Reimer and his team asked the question, «what happens to mature oligodendrocytes after a spinal cord injury in adult zebrafish?».
To understand what molecules were potentially responsible for this remarkable process, the scientists conducted a molecular fishing expedition of sorts, searching for all of the genes whose activity abruptly changed after spinal cord injury.
Oligodendrocyte death, occurring after a spinal cord injury, activates a process called de-myelination that results first in damage to surviving neuronal connections and finally in death of the affected neurons.
A new study in mice published in The Journal of Neuroscience details a potential therapeutic strategy that uses stem cells to promote recovery of motor activity after spinal cord injury.
Researchers demonstrated that susceptibility to spontaneous pneumonia and severe lymphopenia after spinal cord injury resulted from a maladaptive sympathetic - neuroendocrine reflex involving the adrenal glands.
Stylized representation of a newly - discovered signaling pathway active after spinal cord injury, which sees the injured central nervous system use adrenal hormone production to potentially disrupt the immune system in a way that lead to severe infections.
Building upon previous research, scientists at the University of California, San Diego School of Medicine and Veteran's Affairs San Diego Healthcare System report that neurons derived from human induced pluripotent stem cells (iPSC) and grafted into rats after a spinal cord injury produced cells with tens of thousands of axons extending virtually the entire length of the animals» central nervous system.
WASHINGTON — Paralyzed rats can breathe a sigh of relief: A new treatment can restore lung function, even a year and a half after a spinal cord injury that takes it away.
The discovery of motor pattern generators, the neural networks underlying movements, in the spinal cord is already being used to re-establish locomotion in people paralysed after spinal cord injury
«This approach could help keep the body representation «awake» even after a spinal cord injury.
Bao, F., Brown, A., Dekaban, G.A., Omaña, V. and Weaver, L.C. Anti-CD11d integrin blockade reduces the systemic inflammatory response syndrome after spinal cord injury.
Dr. Dyson - Hudson is presenting the model system's research on preventing pneumonia after spinal cord injury, as well as results of a pilot study on the use of platelet - rich plasma therapy for shoulder pain in wheelchair users with spinal cord injury.
Bao, F., Bailey, C.S., Gurr, K.R., Bailey, S.I., Rosas - Arellano, M.P., Dekaban, G.A., and Weaver, L.C. Increased oxidative activity in human blood neutrophils and monocytes after spinal cord injury.
Dr. Fyffe is a co-author of a related article, «Longitudinal analysis of hospitalization after spinal cord injury: Variation based on race and ethnicity,» published online on August 2 by Archives of Physical Medicine & Rehabilitation (DOI: 10.1016 / j.apmr.2014.07.399).
The discovery of motor pattern generators, the neural network's underlying movements, in the spinal cord is already being used to re-establish locomotion in people paralyzed after spinal cord injury
«Row, row, row your bones: Can an FES rowing exercise program prevent osteoporosis after spinal cord injury
Reported MRI data from the SCiStar study that indicates AST - OPC1 cells have durably engrafted in patients post-implantation, demonstrating the potential for AST - OPC1 to prevent lesion cavity formation and possibly reducing spinal cord tissue deterioration after spinal cord injury.
Previous studies of neuron survival by the Priestley lab have confirmed with toluidine blue histochemistry that NeuN immuno - reactivity is a reliable marker of neuron survival after spinal cord injury [34].
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