Here Dr. Reimer and his team asked the question, «what happens to mature oligodendrocytes
after a spinal cord injury in adult zebrafish?».
Not exact matches
Physical therapy aimed at rebuilding neural pathways can often improve a patient's level of functioning
in the months and years
after a
spinal cord injury.
The 1948 British Olympics was the first year to see disabled athletes participating
after the German refugee, Ludwig Guttmann, hosted a competition at Stoke Mandeville Hospital where he was
in charge of Second World War patients with
spinal cord injuries.
The Regenerative Research Foundation
in Rensselaer, an affiliate of the Neural Stem Cell Institute, will receive nearly one - fifth of the funds, or almost $ 1.1 million, for work aimed at promoting
spinal cord regeneration
after injury.
Albert Einstein College of Medicine
in the Bronx received the largest grant, of nearly $ 1.2 million, for research intended to improve urological function
after spinal cord injury.
Here, mice that lack protein kinase C gamma (PKCγ) displayed normal responses to acute pain stimuli, but they almost completely failed to develop a neuropathic pain syndrome
after partial sciatic nerve section, and the neurochemical changes that occurred
in the
spinal cord after nerve
injury were blunted.
Short - term peripheral nerve stimulation may be a new approach to preventing long - term changes
in nerve and muscle function and improving rehabilitation outcomes
after spinal cord injuries.
«Many of the current Phase I trials for
spinal cord injury are run
in the acute phase, which is right
after injury.
Eight people who have spent years paralyzed from
spinal cord injuries have regained partial sensation and muscle control
in their lower limbs
after training with brain - controlled robotics, according to a study published Aug. 11
in Scientific Reports.
A team of researchers led by the University of South Carolina's Jeff Twiss just reported an innate repair mechanism
in central nervous system axons that might be harnessed to regenerate nerves
after brain or
spinal cord injuries.
«Nanoparticles limit damage
in spinal cord injury: Injection
after an
injury reduces inflammation and scarring.»
Together, studies
in zebrafish and mammalian models could inform new ways to manipulate glial cells
after human
spinal cord injury.»
New research
in mice indicates that a drug commonly used to suppress the immune system
in recipients of organ transplants may also reduce tissue damage and neuropathic pain
after spinal cord injury.
Rapamycin treatment four hours
after spinal cord injury significantly improved locomotor function and reduced mechanical and thermal hypersensitivity
in the hindpaws.
«Technique to promote nerve regeneration
after spinal cord injury restores bladder function
in rats.»
Published
in Nature Medicine on May 1, 2017, the study demonstrates chronic ischemic hypoxia (lack of blood and oxygen)
after spinal cord injury and how blood flow plays a key role
in the cause and treatment of motor disorders.
Neuroscientists found that
spinal blood flow
in rats was unexpectedly compromised long
after a
spinal cord injury (chronically ischemia), and that improving blood flow or simply inhaling more oxygen produces lasting improvements
in cord oxygenation and motor functions, such as walking.
He also showed
in 2012 that paralyzed rats could recover
after spinal cord injury after a few weeks of rehabilitation, combining electro - chemical stimulation and physiotherapy that uses a robotic harness.
Partially paralyzed rodents walk almost normally
after human embryonic or fetal brain stem cells repaired their
spinal cord injuries in recent studies.
After other researchers reported that bone marrow stem cell therapy led to a modest but promising 5 percent improvement
in the regeneration of sensory nerve fibers
in rats with
spinal cord injuries, he performed a critical reality check.
The study introduced here focused on a population of support cells
in the
spinal cord that helps to protect surviving nerve cells (neurons)
after injury: oligodendrocytes and their precursor cells.
Oligodendrocyte death, occurring
after a
spinal cord injury, activates a process called de-myelination that results first
in damage to surviving neuronal connections and finally
in death of the affected neurons.
Some examples of the scholars» projects are: effects of estrogen on cardiac fibrosis
after myocardial infarction, depression and the growth - hormone axis, substance abuse, stress and nicotine, cardiovascular risk
in spinal cord injury, and pharmacogenomics and the treatment of breast cancer
in elderly women.
In the years
after the visit, his foundation granted about $ 4 million to Edgerton's lab to find a way to train human patients with complete
spinal cord injuries to walk again.
«Given the effectiveness of inosine
in promoting cortical plasticity, axonal sprouting, and dendritic branching, the present evidence of efficacy
after cortical
injury in a non-human primate, combined with a long history of safe use, indicates a need for clinical trials with inosine
after cortical
injury and
spinal cord injury,» noted Dr. Moore.
When scientists gave mice an oral drug called LM11A - 31 (designed to prevent neural degeneration
in Alzheimer's patients) just hours
after a complete
spinal cord injury, it limited the death of cells that protect nerve fibers, allowing test rodents to retain their ability to walk and swim.
A new study
in mice published
in The Journal of Neuroscience details a potential therapeutic strategy that uses stem cells to promote recovery of motor activity
after spinal cord injury.
Stylized representation of a newly - discovered signaling pathway active
after spinal cord injury, which sees the injured central nervous system use adrenal hormone production to potentially disrupt the immune system
in a way that lead to severe infections.
After eight years of work, we were able to identify an entirely new mechanism for how
spinal cord injury weakens the immune system,» said principal investigator Dr. Jan M. Schwab, neurologist and physician at Ohio State's Neurological Institute, who collaborated with researchers from several institutes
in Germany, along with the University of Alabama
in Birmingham, Harvard Medical School and Boston's Children's Hospital.
Pneumonia is the main cause of death
in patients both
after acute and chronic
spinal cord injury.
Neuroscientists have long believed that scar tissue formed by glial cells — the cells that surround neurons
in the central nervous system — impedes damaged nerve cells from regrowing
after a brain or
spinal cord injury.
Here, the research team led by Associate Professor Yukio Nishimura, National Institute for Physiological Sciences (NIPS), Natural Institutes of Natural Sciences (NINS), and Masahiro Sawada, a former graduate student of Kyoto University, and Dr. Hirotaka Onoe, a team leader at RIKEN Center for Life Science Technologies found that the nucleus accumbens, that control motivation
in the brain, activates the activity of the motor cortex of the brain, and then promotes recovery of motor function during the early stage of recovery
after spinal cord injury.
Dr. Nishimura said, «Our result suggests that
in the early stage
after brain
injury including
spinal cord injury, it is important to motivate the patients for promoting functional recovery
in rehabilitation.
In the intact monkey, and also in the fully recovered stage (3 months after spinal cord injury), such deficit in hand movement by blocking the activity of the nucleus accumbens was not observe
In the intact monkey, and also
in the fully recovered stage (3 months after spinal cord injury), such deficit in hand movement by blocking the activity of the nucleus accumbens was not observe
in the fully recovered stage (3 months
after spinal cord injury), such deficit
in hand movement by blocking the activity of the nucleus accumbens was not observe
in hand movement by blocking the activity of the nucleus accumbens was not observed.
He and colleagues reveal that eight Brazilians paralyzed because of
spinal cord injuries regained some small but significant sensation and muscle control
in their lower limbs
after many months of training with the robotic exoskeleton, and by a virtual reality avatar also controlled by brain signals.
Second - order
spinal cord pathway contributes to cortical responses
after long recoveries from dorsal column
injury in squirrel monkeys
Treatment with the neurosteroid dehydroepiandrosterone promotes recovery of motor behavior
after moderate contusive
spinal cord injury in the mouse.
NEWARK, Calif., May 31, 2016 (GLOBE NEWSWIRE)-- StemCells, Inc. (NASDAQ: STEM)(the «Company») today announced its decision to terminate the Company's Phase II Pathway Study
in spinal cord injury following an
in - depth review of data from the study and
after obtaining the concurrence of the study's Interim Analysis Data Monitoring Committee (the «IA - DMC»).
After all, nanomaterials currently represent our best hope for developing innovative strategies
in the treatment of
spinal cord injuries».
The signaling molecule CD95L, known as «death messenger,» causes an inflammatory process
in injured tissue
after spinal cord injuries and prevents its healing.
The discovery of motor pattern generators, the neural network's underlying movements,
in the
spinal cord is already being used to re-establish locomotion
in people paralyzed
after spinal cord injury.»
Reported MRI data from the SCiStar study that indicates AST - OPC1 cells have durably engrafted
in patients post-implantation, demonstrating the potential for AST - OPC1 to prevent lesion cavity formation and possibly reducing
spinal cord tissue deterioration
after spinal cord injury.
For example, we are using electrical
spinal stimulation synchronized with residual muscle activity or movement
in order to produce lasting improvements
in hand and arm function
after spinal cord injury.
«Humans have very limited capacity for regeneration, while other species like salamanders have the remarkable ability to functionally regenerate limbs, heart tissue and even the
spinal cord after injury,» said lead researcher Karen Echeverri, PhD, assistant professor
in the department of genetics, cell biology and development at the University of Minnesota.
WEST ORANGE, N.J. — August 11, 2015 — The New Jersey Commission on
Spinal Cord Research awarded $ 198,000 over two years to Zhiguo (Tony) Jiang, Ph.D., research scientist at Kessler Foundation, to examine the efficacy of using diffusion tensor imaging (DTI) in assessing recovery after spinal cord injury
Spinal Cord Research awarded $ 198,000 over two years to Zhiguo (Tony) Jiang, Ph.D., research scientist at Kessler Foundation, to examine the efficacy of using diffusion tensor imaging (DTI) in assessing recovery after spinal cord injury (S
Cord Research awarded $ 198,000 over two years to Zhiguo (Tony) Jiang, Ph.D., research scientist at Kessler Foundation, to examine the efficacy of using diffusion tensor imaging (DTI)
in assessing recovery
after spinal cord injury
spinal cord injury (S
cord injury (SCI).
In September of last year, DARPA reported the case of a 28 year - old man who had been paralyzed for a decade,
after suffering a
spinal cord injury.
We're using a neural device to make long - term changes
in neural circuits
after spinal cord injury.
Matt's Testimonial
After a
spinal cord injury left Matt with major disabilities, Matt describes how a change
in attitude allowed him to conceive of a life fully healed.
Many people are also involved
in an auto accident or other trauma that can often sustain a brain or
spinal cord injury — sometimes so apparently minor that many doctors or hospitals say you're fine, even
after an MRI or CT scan.
Furthermore, alternate - day fasting leads to increased functional recovery
after experimentally induced
spinal cord injuries in rats, independently if the alternate - day fasting regimen is implemented prior or
after the
spinal cord is injured (27,50).