FWGE appears to be so effective
against cancer cells because is literally acts as a «biological bounty hunter» for them, especially cancer cells that may be «hidden» from the view of our body's own natural killer cells.
Not exact matches
Now a University of Colorado
Cancer Center study published online ahead of print in the journal Oncogene offers compelling evidence explaining this failure and offering a possible strategy for the use of retinoic acid or other retinoids against some breast cancers: Because early clinical trials are often offered to patients who have already tried other more established therapies, breast cancer cells may have been pushed past an important tipping point that offers retinoic acid resis
Cancer Center study published online ahead of print in the journal Oncogene offers compelling evidence explaining this failure and offering a possible strategy for the use of retinoic acid or other retinoids
against some breast
cancers:
Because early clinical trials are often offered to patients who have already tried other more established therapies, breast
cancer cells may have been pushed past an important tipping point that offers retinoic acid resis
cancer cells may have been pushed past an important tipping point that offers retinoic acid resistance.
Because the different
cancer cells within a tumorous tissue also develop different modes of defense
against therapeutic measures, the treatment of patients is extraordinarily difficult.
The frenzied reproduction of
cancer cells is also a key part of their ability to evade our assaults,
because it is essential to their capacity to evolve new solutions to the challenges that we throw up
against them.
This widespread lack of the need for telomerase is used by evolution as a key component of our defense
against cancer,
because having a limit to the size and renewal of telomeres prevents our
cells from replicating themselves indefinitely — the crucial hallmark of
cancer.
«These new results are significant
because they represent the discovery of a number of more potent variations of the natural product as cytotoxic agents
against cancer cells,» Nicolaou said.
Because it more effectively induces
cancer cell apoptosis in p53 - deficient tumors, FL118 is especially effective
against late - stage
cancers, which usually lose functional p53 and are resistant to DNA - damage drugs.
Knowing the answer is important both as a matter of basic immunology and
because NK
cells, as crucial members of the body's first line of infection defense, are often the subjects of efforts to harness the immune system in protection
against infections and
cancer.
«This is
because the stress led to poor function
against the
cancer by T -
cells, which are very important in the immune system's control and surveillance of tumours and are a major target in many immunotherapy treatments.»
Cancer stem
cells (CSCs) in the hypoxic BM regions are blamed for the incurability of MM,
because CSCs are often resistant to drugs currently used
against BM
cancers (including proteasome inhibitors and immunomodulatory agents).
While orange sweet potatoes have anti-carcinogenic properties, it was found that purple sweet potatoes have better
cancer - fighting abilities
because they have cyanidins and peonidins, which have a positive effect
against cancer cell growth.
Vitamin D also has
cell - normalizing (anti-proliferation) properties, and
because cancer is
cell proliferation gone mad, it protects
against many
cancers.