Since more Cx43 means less room in the bladder, the team speculates that
although normal mice didn't stop producing urine when asleep, their bladders were able to hold more due to decreased Cx43 concentrations.
Not exact matches
Although the
mice seemed
normal at birth, after about 3 months they had less bone growth and bone mass compared to
normal mice.
Although the Nppb - knockout
mice lived a
normal lifespan, when the researchers killed the receptor cells in the spinal cord, the
mice died prematurely, suggesting that it could be dangerous to try humans, Hoon says.
Obese
mice gained less weight after exercise or WBV than obese
mice in the sedentary group,
although they remained heavier than
normal mice.
Bacterial counts from rodents» poop showed that
although B. fragilis did not establish lasting colonies in the
mice, they did «shake up the community,» of microorganisms, bringing it closer to that of the
normal mice, Hsiao says.
Although the
mice without Bax started out with the same number of oocytes as
normal mice, their ovaries harbored about three times as many oocyte - containing follicles shortly after puberty.
Although muscle cells did not reduce in size or number in
mice lacking a protective antioxidant protein, they were weaker than
normal muscle cells, researchers from the Barshop Institute for Longevity and Aging Studies at The University of Texas Health Science Center San Antonio found.
But Hecht notes that
although CNP breaks down within minutes in the body, decoy FGFR3 lasts for hours and produces
normal growth in the
mice with just twice - weekly injections.
Puzzlingly, however,
mice that lack ghrelin don't eat less: They have
normal appetites,
although they burn excess fat.
Although the behavior of the mutant
mice seemed mostly
normal, further testing showed that they had deficiencies in spatial learning, confirming the enzyme's key role in that process.
Although mice completely lacking SLC6A14 are viable and exhibit
normal mammary gland development, these animals are highly resistant to mammary tumour initiation and progression driven by potent oncogenes.