Sentences with phrase «as myofibrillar»

This is known as myofibrillar hypertrophy, in which the contractile unit of the muscle gets stronger and larger, ultimately leading to greater force production.

This results in a different body response, a process known as myofibrillar hypertrophy.

Not exact matches

Most martial artists and non-heavy weight lifters will choose myofibrillar hypertrophy, since their goal is having strong muscles but keeping their weight down, while bodybuilders favor sarcoplasmic hypertrophy which increases muscle size but it's generally viewed as form over function.

Myofibrillar hypertrophy is the process of enlargement of the size / volume / protein content of the muscle tissue, also known as «real» growth.

It is believed that the nucleus releases increased amounts of mRNA as a response to muscle tension and myofibrillar damage, which happens because of insufficient cycling of the actin / myosin cross-bridges which happen during very intense muscle contractions, even though the exact mechanism is not yet understood in detail.

Studies show that the added microtrauma, or myofibrillar «damage,» you get from negatives requires bodyfat as energy to heal and regenerate.

The second one is called myofibrillar hypertrophy and it represents enlargement of the muscle fiber as it gains more myofibrils, which contracts and does all the functional work — creates tension in the muscle.With this type of hypertrophy, the area density of myofibrils increases and there is a significantly greater ability to exert muscular strength.

Now, there has been some back and forth as to whether or not you can exclusively train for sarcoplasmic or myofibrillar hypertrophy.

Myofibrillar hypertrophy is activated with low volume strength training, such as 3 - 6 reps (see the study here).

The argument goes that because sarcoplasmic hypertrophy isn't contractile, it is somehow an impediment to performance and that as rugby players we should only seek myofibrillar hypertrophy.

The heavy loads allow for myofibrillar protein synthesis to take place which, as discussed, will increase the size of the contractile proteins.

Modification of glucocorticoid - induced changes in myofibrillar protein turnover in rats by protein and energy deficiency as assessed by urinary excretion of Ntau - methylhistidine

The aim of the current study was to determine the effect of alcohol intake on anabolic cell signaling and rates of myofibrillar protein synthesis (MPS) in humans during recovery from a bout of strenuous exercise approximating stresses an athlete may experience in training and performance for various team sports such as various football and rugby codes, and court sports.

As such, it appears that the insulin / IGF - PI 3K pathway not only regulates myofibrillar cleavage through caspase 3, but also the ubiquitylation of the resulting protein fragments via modification of ubiquitin - ligase expression.