Sentences with phrase «as tg»

Pride Dating is a gay dating site for gay men and gay women, bisexuals interested in bisexual dating and also transgender dating otherwise known as tg dating,.

The line of mice was known as Tg (BOPrP).

Conversely, Tg - hIAPP mice inoculated with WT pancreas homogenate (hereafter referred to as the Tg / WT group) only began to show small IAPP aggregates at around 20 wk of age (Fig. 3, A and B), in a manner similar to untreated Tg mice.

About Front Burner Brands Front Burner Brands (formerly known as TGS Restaurant Management) is a restaurant management company headquartered in Tampa, Fla..

Hopefully Ubisoft will be sending out some nice new quality screenshots of these areas during the coming days as TGS kicks off, in which case it's quite possible I'm going to need a bucket.

Not exact matches

Another biotech on Cramer's radar as one of the top five best performing biotechs of 2014 is TG Therapeutics.

In your 20s, all stock index fund investments might seem like a fine idea, as short - term volatility matters less than long - term appreciation when a portfolio has decades to grow, says Phillip J. Deerwester, portfolio analyst and chief compliance officer at TGS Financial Advisors in Radnor, Pennsylvania.

I do not see how it is possible, at least from a Christian or Western point of view, to avoid identifying Zen as a backward way tg nal or primordial Unity.

Citation: Kaye TG, Gaugler G, Sawlowicz Z (2008) Dinosaurian Soft Tissues Interpreted as Bacterial Biofilms.

TG is getting a national rep as a mock draft master.

When it was all said in the done Todd «TG» Goldner did have an impressive 18 points in the Loss and Matt «lebron» Emer got in on the scoring party as well.

Title contenders PSG temporarily go second on the Ligue 1 charts as Zlatan Ibrahimovic and three others score in their 4 - 0 thrashing of Evian TG.

Because DH - TGS is a nondestructive material diagnostic, Dennett said there are many systems one might envision studying as microstructural evolution is taking place.

The six benchmarks are: 1) HDR brachytherapy procedures are supported with the appropriate team as described in the report of the AAPM TG 59 and the American College of Radiology HDR Brachytherapy Practice Standard; 2) commissioning of the treatment unit, treatment planning system and each new source is performed by a qualified medical physicist and verified through a QA process; 3) assay of the HDR brachytherapy unit source is performed using a well - type ionization chamber with a calibration traceable to the National Institute of Standards and Technology, and this assay is performed or confirmed for each source change.

(A) Isolated islets from 3 - wk - old, female, Tg - hIAPP mice were cultured in presence of different concentrations of IAPP aggregates prepared in vitro from synthetic IAPP, as well as controls treated with other amyloidogenic proteins, including the Alzheimer's disease — associated protein Tau (the K18 fragment) and the bacterial amyloid Mcc.

As a model of IAPP aggregation, we used Tg mice overexpressing hIAPP at four - to fivefold levels of endogenous mouse IAPP (Janson et al., 1996).

Groups of male Tg - hIAPP mice were injected i.p. at 3 wk of age with 10 % pancreas homogenate from either 12 - mo - old, male, IAPP Tg mice bearing substantial islet amyloid aggregates (as shown in Fig. 1) or from age - matched, male, WT mice not expressing hIAPP.

Islets from Tg - hIAPP, as well as controls, were isolated using a previously published procedure (Li et al., 2009).

As a control, a group of age - and gender - matched Tg - hIAPP mice were i.p inoculated with buffer (PBS).

(C) To evaluate impairment in glucose tolerance, the i.p. glucose tolerance test was performed in 16 - wk - old, Tg - hIAPP mice inoculated with pancreas extracts from old Tg - hIAPP (red line) as compared with Tg - hIAPP mice inoculated with PBS (black line).

As a control, group of IAPP Tg mice were injected with PBS.

Fasting blood glucose level became significantly higher in animals injected with Tg - hIAPP pancreas homogenate containing IAPP aggregates, as early as 8 wk compared with mice treated with WT pancreas extracts (Fig. 4 A).

Fasting glycemia was measured periodically in Tg - hIAPP mice treated with synthetic IAPP aggregates or PBS as well as in WT mice injected with the same preparation of synthetic aggregates (Fig. 7 A).

As controls, groups of male Tg mice were injected with pancreas homogenate from age - matched non-Tg mice that did not have any detectable IAPP aggregates (Fig. 1).

The area of the islets occupied by β cells was significantly reduced in the Tg / Tg group, as compared with control mice.

Citation: Kaye TG, Gaugler G, Sawlowicz Z (2008) Dinosaurian Soft Tissues Interpreted as Bacterial Biofilms.

Using thermogravimetric (TG) and differential thermal analysis (DTA) coupled with mass spectrometry (MS) the thermal stability and the defect chemistry in compound semiconductors such as roquésite and clinobisvanite can be determined.

Examination of brain homogenates from immunized TG mice revealed no effect on monomeric human AS as compared with vehicle - treated animals, but a 45 % reduction in oligomeric AS and a trend toward reduction of human AS dimerAS as compared with vehicle - treated animals, but a 45 % reduction in oligomeric AS and a trend toward reduction of human AS dimeras compared with vehicle - treated animals, but a 45 % reduction in oligomeric AS and a trend toward reduction of human AS dimerAS and a trend toward reduction of human AS dimerAS dimers.

In 2012 she joined the laboratory of Dr. D.L. Konotyiannis at BSRC AL.Fleming, Greece as postdoctoral scientist, where she worked on the study of the RNA Binding Protein HuR during intestinal and lung inflammation and cancer, using inducible, KO and Tg mouse models and xenografts.

Animals were administered 10 mg / kg of candidate mAbs weekly for 6 mo; control animals (WT and TG) were immunized with IgG1 as a control Ab.

In addition to the reduction in total numbers of activated microglia in AFF 1 - treated TG mice, it is of note that costaining studies indicated that those activated microglia that did emerge in treated mice were highly colocalized with recalcitrant (proteinase K - resistant) AS aggregate as compared to untreated PDGF - AS micAS aggregate as compared to untreated PDGF - AS micas compared to untreated PDGF - AS micAS mice.

In contrast, no Slc6a14 − / − female with PyMT transgene developed tumours at 3 months of age; however, at 4 months of age, small tumour nodules became detectable (Figure 2B), indicating a marked delay in mammary tumour development in PyMT - Tg mice as a consequence of Slc6a14 deletion.

Total cholesterol and TG levels were determined enzymatically as described above.

While LPL - deficient patients had higher TG levels than were detected in the LPL - deficient mice, the TG levels in the mice were not dissimilar from those seen in patients who strictly adhered to a low - fat diet that might be viewed as the equivalent of mouse chow.

The iLpldf mice presented with elevated plasma TG and cholesterol levels (Supplemental Figure 5, C and D, respectively), along with a dramatic increase in VLDL TGs and a reduction in HDL cholesterol levels (Supplemental Figure 5, E and F), as observed in patients with LPL deficiency (16).

LPL is a pivotal player in TG metabolism, as mutations in LPL or in its cofactors (ApoC - II, glycosylphosphatidylinositol - anchored high - density lipoprotein — binding protein 1, lipase maturation factor 1) account for the majority of monogenic hypertriglyceridemia cases (25).

We used a previously described tamoxifen - inducible, Lpl - deficient mouse model (Lplfl / fl Actb - MerCreMer +, referred to herein as iLpldf mice) to further assess the in vivo contribution of LPL to the reduction of plasma TGs by ApoC - III ASO (22).

Administration of the ApoC - III ASO did not affect VLDL - TG secretion (Figure 7C) or chylomicron formation in vivo in mice as reported previously (15).

Furthermore, gender is important in the development of sucrose induced IR and hyper - TG in rats as females (unlike males) are typically not responsive to elevations in dietary sucrose (6).

To measure total lipase activity, plasma samples were incubated with 10 % Intralipid / [3H] TG emulsion (Hospira) as a substrate and human serum as the source of ApoC - III (24).

In contrast to rats and hamsters, the mouse is used less frequently as a model for sucrose / fructose - induced IR and hyper - TG as the commonly used C57BL / 6 mouse either does not develop IR or develops the phenotype more slowly (9, 14).

Again, ApoC - III ASO treatment had no effect on plasma TG levels in mice lacking both Lrp1 and Ldlr (Figure 5, A and C), as observed with animals fed a standard chow diet (Figure 1) and under postprandial conditions (Figure 3).

As reported by Noh et al. (43), tamoxifen transiently increases plasma TG levels, which return to normal 2 weeks after the last administration.

ApoC - III ASO treatment reduced TG levels in chylomicrons, VLDL, and remnant particles in all of the mutants as measured by size - exclusion fast protein liquid chromatography (FPLC)(Figure 2, A — E), but had no effect in Ldlr — / — Lrp1fl / fl Alb - Cre + mice (Figure 2F).

Clearance of chylomicrons derived from dietary TGs was measured by vitamin A excursion essentially as described previously (18).

Lowering of plasma TG levels after injection of [3H] triolein - labeled Liposyn particles was associated with a greater uptake of [3H] triolein in the liver, but not in organs such as the heart or skeletal muscle (Figure 8F), as would be expected if LPL activity were enhanced.

As i remember good in Thyroid Secret was mentioned that antibodies like tpo or tg should be as lower as possible — and even should be 1 or As i remember good in Thyroid Secret was mentioned that antibodies like tpo or tg should be as lower as possible — and even should be 1 or as lower as possible — and even should be 1 or as possible — and even should be 1 or 0!

Now, have you seen where TPO TG would be high at the same time as TSI?

Animal models strongly support this pathophysiological mechanism whereby iodine induces thyroid autoimmunity, as, «Excessive iodine intake can precipitate spontaneous thyroiditis in genetically predisposed animals, by increasing the immunogenicity of thyroglobulin (TG)» (19).

In fact, groups with the aforementioned thyroid diseases, as well as individuals with positive TG or TPO antibodies, have been demonstrated to exhibit significantly higher levels of urinary iodine, the main indicator of iodine nutritional status, compared to healthy controls (3).