In his study, some of the cells his team initially flagged
as young neurons turned out to be mature cells upon further investigation.
Not exact matches
Healthy people in their 70s have just
as many
young nerve cells, or
neurons, in a memory - related part of the brain
as do teenagers and
young adults, researchers report in the April 5 Cell Stem Cell.
While these brain rhythms, occurring hundreds of times a night, move in perfect lockstep in
young adults, findings published in the journal
Neuron show that, in old age, slow waves during non-rapid eye movement (NREM) sleep fail to make timely contact with speedy electrical bursts known
as «spindles.»
«Although running induces both substantial changes in number and morphology of
young neurons as well
as significant changes in learning behavior, this does not prove a causal relationship,» noted Professor Bischofberger, «Nevertheless, our results, together with previous findings, suggest that the enhanced pattern separation during memory testing is most likely mediated via running - induced increase in adult neurogenesis.»
Amyloid — an abnormal protein whose accumulation in the brain is a hallmark of Alzheimer's disease — starts accumulating inside
neurons of people
as young as 20, a much
younger age than scientists ever imagined, reports a surprising new Northwestern Medicine study.
Amyloid — an abnormal protein whose accumulation in the brain is a hallmark of Alzheimer's disease — starts accumulating inside
neurons of people
as young as 20, a much
younger age than scientists ever imagined.
When the induced pluripotent stem cell method was used,
as expected, the patterns in the
neurons were indistinguishable between
young and old derived samples.
Health improvement (allowing to post - pone / escape the diseases and thus live, healthier / disease - free longer, but not above human MLSP of around 122 years; thus these therapies do not affect epigenetic aging whatsoever, they are degenerative aging problems not regular healthy aging problem (except OncoSENS - only when you Already Have Cancer - which cancer increases epigenetic aging, but cancer removal thus does not change anything / makes no difference about what happens in the other cells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing
as «healthy aging» all aging in «unhealthy» (
as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories /
neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are
younger brain for their age), and both are correlated to MLSP).
«We found that older people have similar ability to make thousands of hippocampal new
neurons from progenitor cells
as younger people do,» lead author Maura Boldrini and associate professor of neurobiology at Columbia Universtiy says.
Overall, the study found, older and
younger brains had similar numbers of «intermediate» progenitor cells and «immature»
neurons — signaling that older people had a similar capacity for generating new cells
as young people.
The discovery that healthy, older adults produce just
as many new
neurons as young people could provide clues to how to keep our brains sharper for longer