Sentences with phrase «b allele»

In breeds where the A locus does not come into play, any animal that has at least one B allele (and is not «ee»), will be black in pigmented coat.
Regardless of other loci, any animal with at least one B allele will have a black nose and pads, while those with any two b alleles will have a liver nose and pads.
That would mean that both the Sire and the Dam would have to contribute a recessive b allele on to the puppy in order for it to be a Chocolate Labrador.
It would take a bb gene for the puppy to be chocolate, because the b allele is a recessive trait.
Genetic tests revealed that all ants in a monogyne colony have two copies of the B allele, but among the polygynes, at least 10 % are heterozygous, carrying a mutant allele, b, which Krieger says appears to code for a faulty protein.
HLA - B is the most polymorphic gene in the MHC class I region, comprised of 4,765 HLA - B alleles (IPD - IMGT / HLA Database Release 3.28).
Red or cream dogs that carry two b alleles remain red or cream but have brown noses and footpads.
Those dogs, which have two copies of any of several b alleles will be brown.

Not exact matches

Several variants, such as the HLA B * 5701 allele, have been associated with the pace of HIV disease progression in both controllers and «normals.»
In the second tree (B) the grandfather is ff, so all his children are female, they have the ff combination of alleles because their father and mother were both ff.
«This research is very important, but it remains to be clarified what the role of these two loci are,» says Mark Connors, an immunologist at the National Institute of Allergy and Infectious Diseases in Bethesda, Maryland, whose lab first reported that HIV - infected people who remain unharmed by the virus for many years often have the HLA - B * 5701 allele.
The team uncovered strong signs of this imbalance between rare alleles and heterozygosity in two populations — one, in people living today in the Middle East, and the other in the Yakut, who live near the Bering Strait.
Rescue of hemizygous XpdTTD / KO survival by Xpd † XPCS and Xpd † XP alleles is illustrated by arrows marked A and B, respectively.
(B) TALEN targets for the generation of the CCHa2 - RTAL - 34 allele [1L, 1R, 2L, and 2R; underlined].
(B) The CCHa2 - RTAL - 34 allele was created using TALENs.
We did not identify the JAK2V617F disease allele in B - lineage ALL (n = 83), T - cell ALL (n = 93), or CLL (n = 45).
(B) Deletions in CCHa2 mutant alleles and the resulting amino acid sequences.
(B) DNA sequence of mutated alleles from MSTN - mutant colonies.
Yet each one tells a different story: (a), how novel - to - dbSNP variants were rare; (b), how nonsynonymous variant frequencies are shifted to lower values relative to those of synonymous variants, (c), how this shift in allele frequencies is more pronounced for damaging nsSNPs, consistent with natural selection, and (d), how the sizes of observed indels are enriched for non-frameshift events divisible by 3.
Individuals with the HLA - B * 5701 allele were found to be more likely to have a hypersensitivity reaction to abacavir.
Testing for the human leukocyte antigen HLA - B * 5801 allele before initiating allopurinol was found to be cost effective for black and Asian patients but not for white or Hispanic patients...
Testing for the human leukocyte antigen HLA - B * 5801 allele before initiating allopurinol was found to be cost effective for black and Asian patients but not for white or Hispanic patients, according to results of a cost - effectiveness analysis published in Seminars in Arthritis and Rheumatism.
In individuals found to have the HLA - B * 5701 allele, abacavir is avoided, and alternative HIV treatments are given.
If the puppy has a Bb gene, then they will be black, because B is a black allele and it is dominant.
Multiplex PCR was designed to amplify the normal and mutated alleles of canine ADAM9 (Table 7, Figure 4A, B).
The alleles involved with color are B and E (or their recessive version b and e)B and E (or their recessive version b and e)b and e).
For example, if you have a black lab with the genetic makeup of EEBB then that dog will always pass on one E allele and one B. On the other hand, if your black lab has the genetic makeup of EeBb, then your puppy might receive an E or e and a B or bB. On the other hand, if your black lab has the genetic makeup of EeBb, then your puppy might receive an E or e and a B or bB or bb.
Eventually it would be possible to mate two animals that are both heterozygous for the desired allele and produce homozygous progeny, with very little change in genetic composition (B) After five generations of backcrossing to the desired breed, the proportion of the genotype contributed by this breed is 96.9 %, while by ten generations it is 99.9 %.
The dog could be a carrier with a false allele (but because of how markers are inherited it will never be a Pattern B; it's still a Pattern C).
Pattern B and C uncertainties are due to the test detecting a false allele.
The locus is given the designation B (if we refer to a more recessive allele we use the subcase b) and you can remember that by associating it with the word Brown, if that helps your memoryB (if we refer to a more recessive allele we use the subcase b) and you can remember that by associating it with the word Brown, if that helps your memoryb) and you can remember that by associating it with the word Brown, if that helps your memory.
(b) In the mediation regression where both S allelic frequency and cultural value of individualism — collectivism were included as predictors of global depression prevalence across 12 nations, individualism — collectivism remained a reliable predictor (β = − 0.21, p < 0.05), and the effect of S allele frequency decreased significantly (from r (12) = − 0.51 to r (12) = − 0.03; Sobel test Z = − 1.92, p < 0.05).
Simple slope tests with a cutoff of ± 2 SD indicated that in the condition of very high negative parenting (b = 0.00, t = 0.01, p >.05) or very low negative parenting (b = − 0.00, t = − 0.11, p >.05), A1 allele was also not related to depressive symptoms at T3.
Adolescents living in high - risk families who carried at least one DRD4 long allele and were assigned to the control condition evinced greater escalations in drug use than did (a) adolescents who lived in high - risk families, carried the DRD4 long allele, and were assigned to AIM, or (b) adolescents assigned to either condition who carried no DRD4 long alleles.
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