MCFAs produce ketones when they're digested — this is significant
because cancer cells lack the ability to use fat as a fuel.
This is
because some cancer cells consume calories, virtually robbing the kitty of nutrition.
Because cancer cells grow and divide more rapidly than most of the normal cells around them, radiation therapy can successfully treat many kinds of cancer.
The liver releases the glucose and cancer cells are likely to pick up this glucose
because cancer cells consume about 15 times more glucose than normal cells.
So there's a number of things that will target cancer cells
because cancer cells love sweets.
Because cancer cells have a poorer tolerance to heat than healthy cells, infrared light is just the ticket to kill them off — and stop potential cancers in their tracks.
In addition to killing cancer cells outright, infrared saunas combat cancer by helping to improve oxygen - carrying blood flow — important
because cancer cells can't thrive in high - oxygen environments.
Because cancer cells prefer to grow well in the acidic environment.
Also, when using things that kill microbes and can get inside of cancer cells by themselves, if a person quits eating and drinking at 6:00 PM, then takes the liquid cancer products first thing in the morning and continues the liquid and food fast until NOON, these products should be more effective
because the cancer cells get «thirsty» and gobble up the liquid product that kills microbes (e.g. colloidal silver).
This is likely
because cancer cells primarily burn sugar for energy and ketosis does not provide that for them.
Because cancer cells can not use these ketones, they starve to death.13
This is especially important for cancer treatment,
because cancer cells thrive on glucose.
Said simply,
because cancer cells are weaker than normal cells, they are more susceptible to damage from heat.
Our underlying premise is that the tumor microenvironment is immune suppressive
because cancer cells elicit responses characteristic of wound healing and tissue regeneration.
Because cancer cells reproduce at a furious pace, they quickly reach the ends of their telomeric «ropes,» and need to find a way to lengthen them again in order to keep going.
* The fact is that
because cancer cells reproduce more rapidly than any other cell in the body, mutations occur in cancer cells in far greater numbers.
Now, scientists at the Salk Institute have uncovered details about how cancer is able to become drug resistant over time, a phenomenon that occurs
because cancer cells within the same tumor aren't identical — the cells have slight genetic variation, or diversity.
Part of that is
because cancer cells in solid tumors appear to have learned how to «hide» better.
Not surprisingly, the more aggressive a tumor, the more likely it is to engraft — probably
because those cancer cells are particularly adept at growing and spreading.
Because cancer cells acquire mutations in oncogenes — genes that can transform cells into cancer cells — to support their growth and survival, a great deal of research has focused on identifying oncogenes that could be targeted by cancer drugs.
But
because cancer cells contain telomerase and also live forever, scientists worried that the newly immortal cells would become malignant when implanted in humans.
Tumors go mostly unmolested by the body's natural defenses, partly
because cancer cells are descendents of normal body cells.
Because cancer cells grow and divide rapidly, they use a lot of energy, sucking up glucose and giving themselves away; the red coloring denotes disease in the patient's liver and shoulder area.
The discovery might also apply to cancer,
because cancer cells often have abnormal numbers and arrangements of chromosomes.
Yet despite all the fanfare about them, these drugs rarely cure anyone,
because cancer cells» high mutation rate allows them to dodge the pathway concerned.
The HER2 pathway is mutated in many cancers, which drives tumors, but inhibitors of this pathway, such as lapatinib, have only limited success
because cancer cells quickly adapt.
Because cancer cells also make telomerase, scientists have suspected that the enzyme allows cancer cells to keep on dividing without dying.
«This is important
because some cancer cells are more resistant to one type of treatment than another.
DIPGs are known as one of the most challenging tumors to treat
because cancer cells are intimately intermingled with normal brain cells in a part of the brain that can not be surgically resected.
Because cancer cells in general are metabolically inflexible, we are able to take advantage of ketone metabolism as a way of placing cancer cells in a weakened state.
This feat is possible
because cancer cells emit a different kind of metabolic waste than healthy cells.
Because cancer cell metabolism relies upon glucose, minimizing a tumor's access to sugar as a means to manage or even limit cancerous cell growth has been the subject of several recent cancer therapy studies.
«That's
because a cancer cell is hyperactive and has a different biochemistry on its surface and degree of activity that it exhibits.
Not exact matches
I was sedated, but my wife, who was there, told me that when they viewed the
cells under a microscope, the doctors started crying
because it turned out to be a very rare form of pancreatic
cancer that is curable with surgery.
One reason that taller people are more likely to die of many types of
cancer is
because they tend to have bigger organs, so there is a greater chance that one
cell in the organ will become cancerous, Batty said.
Davies and Lineweaver suggest that genes active in embryogenesis and switched off later may be reactivated
because of damage, causing the accelerated
cell division of these rogue
cancer cells.
DMBA is another
cancer - generating drug used in research
because it causes mutations in
cells.
You have probably heard a number of rumors about the effects of technology on your health; that having a
cell phone next to your head can cause brain
cancer, for example, or that pregnant women shouldn't stand near a microwave that is running
because it may harm the fetus.
Breech Twins and higher order multiples Previous CS Pre-Eclampsia Placenta praevia Cervical incompetence Previous late stillbirth Previous premature birth Grand multiparty Age under 18 Age over 35 Smoking Drug use Severe mental health issue Epilepsy Type 1 diabetes Type 2 diabetes Gestational diabetes Asthma GBS positive Abnormal antibodies Transplant recipient Congenital heart disease Known foetal abnormality Immunosuppressive medication MS Physical disability Intellectual disability Hypothyroidism Hyperthyroidism Previous shoulder dystocia Previous 3rd or 4th degree tear Sickle
Cell anaemia BMI under 18 or over 35 at conception Previous massive PPH APH in current pregnancy HIV / AIDS Hepatitis B or C Active TB IUGR Oligohydramnios Polyhydramnios Child previously removed from custody
because of abuse Uterine abnormalities such as uterine septum or double uterus Previous uterine surgery for fibroids Chronic renal problems Hypertension Auto immune condition Previous stroke or blod clot
Cancer Domestic violence or abusive home Prisoners Homeless women
(borrowed from Dr Kitty) Breech Twins and higher order multiples Previous CS Pre-Eclampsia Placenta praevia Cervical incompetence Previous late stillbirth Previous premature birth Grand multiparty Age under 18 Age over 35 Smoking Drug use Severe mental health issue Epilepsy Type 1 diabetes Type 2 diabetes Gestational diabetes Asthma GBS positive Abnormal antibodies Transplant recipient Congenital heart disease Known foetal abnormality Immunosuppressive medication MS Physical disability Intellectual disability Hypothyroidism Hyperthyroidism Previous shoulder dystocia Previous 3rd or 4th degree tear Sickle
Cell anaemia BMI under 18 or over 35 at conception Previous massive PPH APH in current pregnancy HIV / AIDS Hepatitis B or C Active TB IUGR Oligohydramnios Polyhydramnios Child previously removed from custody
because of abuse Uterine abnormalities such as uterine septum or double uterus Previous uterine surgery for fibroids Chronic renal problems Hypertension Auto immune condition Previous stroke or blod clot
Cancer Domestic violence or abusive home Prisoners Homeless women
In one respect the first look is unnerving,
because the chemical mechanisms that seem to drive the
cancer cell astray are not different in kind from mechanisms at work in the normal
cell.
«
Because this binding causes EphA2 internalization, we also sought to conjugate 123B9 with paclitaxel and thus direct the drug to migrating
cancer cells,» said Pellecchia, who holds the Daniel Hays Chair in Cancer Research a
cancer cells,» said Pellecchia, who holds the Daniel Hays Chair in
Cancer Research a
Cancer Research at UCR.
The robots could be useful in
cancer treatments,
because they can target specific
cells.
Because prostate
cancer does not express SLC13A5, the growth of prostate
cancer cells was unaffected by suppressing SLC13A5 expression.
Because they multiply quickly and spread throughout the body,
cancer cells require more energy than normal
cells.
Belov already knew that devils in eastern Tasmania are vulnerable
because their immune system mistakes foreign
cancer cells for «self»
cells.
The hope is that 3BP specifically kills certain
cancer cells — while leaving normal
cells alone —
because they rely more on glucose metabolism than on an alternative pathway called oxidative phosphorylation.
Now a University of Colorado
Cancer Center study published online ahead of print in the journal Oncogene offers compelling evidence explaining this failure and offering a possible strategy for the use of retinoic acid or other retinoids against some breast cancers: Because early clinical trials are often offered to patients who have already tried other more established therapies, breast cancer cells may have been pushed past an important tipping point that offers retinoic acid resis
Cancer Center study published online ahead of print in the journal Oncogene offers compelling evidence explaining this failure and offering a possible strategy for the use of retinoic acid or other retinoids against some breast
cancers:
Because early clinical trials are often offered to patients who have already tried other more established therapies, breast
cancer cells may have been pushed past an important tipping point that offers retinoic acid resis
cancer cells may have been pushed past an important tipping point that offers retinoic acid resistance.
However, new research from the University of Copenhagen reveal that the infections may even benefit the
cancer cells,
because the toxins in certain staphylococcus bacteria make healthy
cells produce substances that in fact advance their growth.
But T -
cells appear to be even more promising,
because they can react to abnormalities inside
cancer cells rather than just on their surfaces.