Willet, Mills, and their colleagues believe the discovery that cells in different organs go through the same process to
become proliferative could lead to new potential targets for cancer treatment because the factors that initiate tumours could be the same in multiple organs.
But if, as this study suggests, the way that cells
become proliferative again is similar across many different organs, we can imagine therapies that interfere with cancer initiation in a more global way, regardless of where that cancer may appear in the body,» he explains.
Not exact matches
«When TET proteins are lost, iNKT cells that lack them apparently
become trapped in an immature, highly
proliferative state,» explains Tsagaratou.