The links
between early life stress, alterations in biological stress regulatory systems, and health outcomes likely depend on neural regulation of stress responses in the brain.
Not exact matches
The «rapid about - face» began in the
early 1960s under the impulse of the Second Vatican Council and «its willingness to address non-Catholic Christians as «brothers,» to acknowledge that blame lay on both sides for the ecclesiastical ruptures of the Reformation, to
stress the unique role of Christ as mediator
between God and humanity, and to urge ordinary lay Catholics to
live lives of practical Christian holiness.»
Parental responsiveness moderates the association
between early -
life stress and reduced telomere length.
I completed my MA thesis on the association
between early life adversity and developing
stress physiology in free - ranging juvenile rhesus macaques on Cayo Santiago.
A few interesting articles in
early life human microbiome, plus: A comparison
between Staphylococcus epidermidis commensal and pathogenic lineages from the skin of healthy individuals
living in North American and India; A new tool to reconstruct microbial genome - scale metabolic models (GSMMs) from their genome sequence; The seasonal changes in Amazon rainforest soil microbiome are associated with changes in the canopy; A specific class of chemicals secreted by birds modulates their feather microbiome; chronic
stress alters gut microbiota and triggers a specific immune response in a mouse model of colitis; and evidence that the short chain fatty acids profile in the gut reflects the impact of dietary fibre on the microbiome using the PolyFermS continuous intestinal fermentation model.
A collaboration
between investigators at Massachusetts General Hospital and Khyber Medical University in Pakistan may have discovered how chronic
stress experienced
early in
life increases vulnerability to post-traumatic
stress disorder (PTSD) later in
life.
While this article discusses the impact of
early life stress combined with the exposure to (bad) bacteria, it continues to confirm the connection
between «happy gut, happy brain,» which is something that is very important to the success of resetting your weight.
In humans, both the HPA system and the autonomic nervous system show developmental changes in infancy, with the HPA axis becoming organized
between 2 and 6 months of age and the autonomic nervous system demonstrating relative stability by 6 to 12 months of age.63 The HPA axis in particular has been shown to be highly responsive to child - caregiver interactions, with sensitive caregiving programming the HPA axis to become an effective physiological regulator of
stress and insensitive caregiving promoting hyperreactive or hyporeactive HPA systems.17 Several animal models as well as human studies also support the connection
between caregiver experiences in
early postnatal
life and alterations of autonomic nervous system balance.63 - 65 Furthermore, children who have a history of sensitive caregiving are more likely to demonstrate optimal affective and behavioral strategies for coping with
stress.66, 67 Therefore, children with histories of supportive, sensitive caregiving in
early development may be better able to self - regulate their physiological, affective, and behavioral responses to environmental stressors and, consequently, less likely to manifest disturbed HPA and autonomic reactivity that put them at risk for
stress - related illnesses such as asthma.
ECD programmes can take many forms, including promotion of good health and nutrition, support for safe and stimulating environments, protection from risks such as violence or abandonment, parenting support and
early learning experiences, media, preschools and community groups.4 Poverty is the key underlying cause of poor child development; children
living in poverty are exposed to many negative influences, including poor physical environments, inadequate nutrition, parental
stress and insufficient cognitive stimulation.5 Undernutrition can influence brain development directly by affecting brain structure and function, or indirectly via poor physical or motor development, in addition to other pathways.6 — 8 Exposure to multiple co-occurring risks most likely contributes to greater disparities in developmental trajectories among children with differential exposure.9 — 12 This paper focuses on associations
between specific aspects of children's physical environments — access to improved water and sanitation (W&S)-- and childhood development as measured by performance on a test of receptive language.
This review will give an overview of animal and human studies that have focused specifically on the relation
between prenatal
stress exposure and offspring behaviour or temperament in
early life.
Interactions
between BDNF Val66Met polymorphism and
early life stress predict brain and arousal pathways to syndromal depression and anxiety
Research links
early life trauma with insecure attachment (e.g. Murphy et al. 2014) and research also suggests an association
between attachment insecurity and parenting
stress (Kwako et al. 2010).