There's a known link
between inflammation of the brain and depression, which can be treated and reversed through an anti-inflammatory diet that includes foods such as turmeric.
Not exact matches
`... Interactions
between in
inflammation and the
brain appear to drive the development
of depression and may contribute to non-responsiveness to current antidepressant therapies... [The] inevitability
of our evolutionary past is apparent in the high rates
of depression that are seen in society today.»
Analysis
of their
brains revealed
inflammation and decreased numbers
of dendrites, which transmit signals
between nerves.
This type
of inflammation between 18 and 32 weeks
of gestation in humans has been linked to preterm birth as well as an imbalance
of immune cells in the
brain of the offspring and even death
of nerve cells in the
brains of those children.
In a recent paper in the Journal
of Alzheimer's Disease, UTMB's research team detailed their investigation on the relationship
between inflammation, toxic tau and Alzheimer's onset by performing systematic analyses
of brain and retina samples from people with Alzheimer's and a mouse model
of Alzheimer's.
Because
of the work
of several other collaborators, Haughey says, his team knew that some sort
of inflammation - promoting molecule was released from
brain and targeted to the liver after
brain injury to send immune system cells to the damaged area, but the identity
of this go -
between had been elusive for years.
More recently we have been funded by the NIH and Simon's Foundation (USA) to study the interactions
between systemic
inflammation,
brain metabolism and cognitive function in models
of hypocholinergic function,
brain amyloidosis and autism.
«This study suggests a link
between increased
inflammation in middle - aged people and shrinkage in areas
of the
brain that are known to be affected by Alzheimer's disease.
Kinney is leading a team researching the role
of several risk factors and subtle changes in cell function associated with Alzheimer's disease, including interactions
between diabetes and
inflammation in the
brain in the onset and progression
of Alzheimer's disease.
Hundreds
of studies have proven the relation
between eggs consumption and improved
brain health, decreasing
inflammation and the improvement
of cardiovascular health.
One study showed a significant correlation
between the death
of nerve cells in the
brain and chronic
inflammation.
Neurologist David Perlmutter, MD, author
of Brain Maker, believes that
Inflammation is the likely connection
between various food triggers.
This is bad news because research shows an inflamed and unhealthy gut is directly linked to depression, giving post-concussive patients a double whammy
of depression - inducing
inflammation that travels back and forth
between the gut and the
brain.
Because
of the strong correlation
between gut health and
brain health, I recommend stool testing to check for pathogens and parasites that contribute to systemic
inflammation.
And insulin resistance increases
inflammation in the
brain, driving the formation
of clumps
of protein clumps
between nerve cells, too.
Several candidate mechanisms
of CRCI have been proposed, including
inflammation and cytokine dysregulation, chemotherapy - induced epigenetic changes, blood —
brain barrier disruption, hormone deficiencies, oxidative DNA damage and shortened telomere length, and genetic susceptibility.21 — 26 However, the heterogeneity and design limitations within the emerging body
of evidence have led to recommendations for harmonising study methodologies and moving towards multisite, longitudinal research.3 27 Such approaches would support the demonstration
of robust relationships
between measurable biological processes and cognitive outcomes.
These toxic stress - induced changes in
brain structure and function mediate, at least in part, the well - described relationship
between adversity and altered life - course trajectories (see Fig 1).4, 6 A hyper - responsive or chronically activated stress response contributes to the
inflammation and changes in immune function that are seen in those chronic, noncommunicable diseases often associated with childhood adversity, like chronic obstructive pulmonary disease (COPD), cirrhosis, type II diabetes, depression, and cardiovascular disease.4, 6 Impairments in critical SE, language, and cognitive skills contribute to the fractured social networks often associated with childhood adversity, like school failure, poverty, divorce, homelessness, violence, and limited access to healthcare.4, 19,58 — 60 Finally, behavioral allostasis, or the adoption
of potentially maladaptive behaviors to deal or cope with chronic stress, begins to explain the association
between childhood adversity and unhealthy lifestyles, like alcohol, tobacco, and substance abuse, promiscuity, gambling, and obesity.4, 6,61 Taken together, these 3 general classes
of altered developmental outcomes (unhealthy lifestyles, fractured social networks, and changes in immune function) contribute to the development
of noncommunicable diseases and encompass many
of the morbidities associated epidemiologically with childhood adversity.4, 6