Weak associations
between pubertal development and psychiatric and behavioral problems.
The current study uses a prospective, longitudinal sample of 1,185 girls (47.8 % Caucasian) to examine the relationships
between pubertal timing, childhood depressive symptoms, and adolescent depressive symptomatology.
Previous research has investigated the relationship
between pubertal timing and depression in girls, with most results suggesting that earlier menarche predicts more depression in adolescence.
In this way, the behaviors of peers in response to the girl's early maturity could be said to mediate associations
between pubertal timing and sexual outcomes (Baron & Kenny, 1986; Holmbeck, 1997, 2002).
Finally, we explored whether the association
between pubertal status and neural response to peer rejection and acceptance would differ for depressed youth and healthy controls.
Resilience factors may moderate the associations
between pubertal timing, body mass and emotional symptoms in adolescence.
This study examined two such factors, romantic competence and romantic experiences, and their role in the cross-sectional and longitudinal associations
between pubertal timing and depressive symptoms among 83 early adolescent females (89 % Caucasian).
To examine whether the relationship
between pubertal status and neural response to social evaluation differed for healthy youth and controls, similar whole brain regression analyses were conducted to identify areas showing group × pubertal status interaction effects.
Summary of hierarchical regression analyses testing peer - and teacher - reported popularity among boys as a mediator of the link
between pubertal timing and rumors
These effects were qualified by group × pubertal status interaction effects (P < 0.001, 30 voxels contiguity) indicating that, contrary to hypotheses, the relationship
between pubertal maturation and response to rejection in the left amygdala and caudate / sgACC was stronger among healthy controls than MDD youth.
Not exact matches
Since timing of puberty is not a simple function of chronological age, and also greatly differs
between the sexes, the
pubertal phase at first drink may therefore represent a stronger and better indicator for subsequent alcohol - related problems than simply the age.»
Pubertal onset marks the transition period
between childhood and adulthood during which adult reproductive capacity is attained.
Our faculty study a wide range of topics, including health outcomes and quality of life for children with diabetes, and the link
between childhood obesity and its long - term endocrine consequences such as
pubertal maturation.
Despite the widely reported link
between early
pubertal timing and internalizing symptoms among girls, less is known about the peer reputation of earlier maturing girls.
In spite of the large literature supporting the link
between early
pubertal timing and depression in adolescent girls, there are some exceptions.
Thus, the current study capitalizes on multiple informants as well as a 3 - wave longitudinal design to test associations
between early
pubertal timing, peer reputation, and psychological distress among an ethnically diverse sample of girls transitioning through the emotionally «risky» period of early adolescence.
Most noteworthy, though, is the novel evidence the current study provides for the role of rumors and gossip in helping to explain the link
between early
pubertal timing and internalizing symptoms.
The association
between early
pubertal timing and internalizing symptoms among girls has gained substantial support in the adolescent development literature.
While off - time
pubertal development has emerged as a potential risk factor for both symptoms of depression and anxiety in youth, the literature is mixed and inconsistent as to (1) how early versus late
pubertal timing confers risk for both boys and girls, (2) if the conferred risk is distinct
between symptoms of anxiety and depression, and (3) under what social contexts (e.g., family environment, peer relationships) off - time
pubertal development may emerge as a potent risk factor for these symptoms.
The interaction
between earlier
pubertal timing and greater harsh discipline was significantly related to higher youth anxiety but not depressive symptoms.
In line with biopsychosocial models, results indicate that the effect of
pubertal timing on depressive symptoms must be conceptualized through complex interactions
between characteristics of adolescents» interpersonal relationships and prepubertal vulnerabilities.
The first model (dashed arrows) predicts that popularity among boys at the start of middle school will partially mediate the link
between earlier
pubertal development and rumor nominations received from peers in the spring of 6th grade.
Interestingly, contrary to our hypotheses, the relationship
between left amygdala and caudate / sgACC activity with
pubertal maturation was stronger among healthy youth than depressed youth.
Additional analyses indicated that
pubertal timing moderated the association
between synchrony and depressive symptoms at age 20, such that girls who exhibited asynchronous development had the highest levels of depressive symptoms when they matured later than peers.
This study investigated how
pubertal development may influence the relation
between aspects of the family environment and diabetes adjustment.
Results showed that although the four - way interaction
between NCS, stressors, gender, and age was significant while the four - way interaction with
pubertal status was marginally significant, the interpretation of these interactions was largely similar, i.e., NCS and depressive symptoms were significantly related only in the presence of many stressors in middle to late adolescent boys (or in boys reporting a high
pubertal status).
The purpose of this report is to provide evidence of an association
between within - person variability in diurnal testosterone over 1 year, lifetime exposure to violence, and the manifestation of antisocial behavior in 135
pubertal - aged adolescents across 1 year.
Moderation by
pubertal status instead of age yielded slightly different results, that is, in the model with stress - reactive rumination, the relationship
between negative cognitive style and depressive symptoms was stronger in adolescents who perceived their
pubertal status as high, whereas age did not moderate this relationship.
Pearson correlations
between aspects of the family environment and diabetes adjustment revealed many significant findings for the total sample and separately by
pubertal development.
The four - way interaction
between NCS, stressors,
pubertal status, and gender approached significance (β = −.10, p =.08), indicating that the interaction
between NCS and stressors was only significant in boys who perceived their
pubertal status as high (β =.42, p =.007).
Some support has been found for an association
between hormonal concentrations and negative affect20 - 24; however, social factors, including negative life events and their interaction with
pubertal status (but not hormonal status), account for more of the variance in negative affect than biological factors alone.25 Early
pubertal timing and its social implications have also been postulated as an important risk factor in girls.26 - 31 Two recent studies, however, report that
pubertal status has a greater influence in predicting female depression than age32, 33 or the timing of puberty.32
Because of the marked collinearity
between age and
pubertal status, we were unable to estimate the effects of these 2 variables simultaneously.
Our goals were (1) to compare the trajectory of depressive symptoms among boys and girls from childhood into adolescence; (2) to analyze the role of genetic, shared, and unique environmental factors in depression among prepubertal and
pubertal male and female twins; and (3) to investigate a possible common etiology
between liability to depression and one salient index of the child's environment: past - year life events.
Exposure to peer delinquency as a mediator
between self - report
pubertal timing and delinquency: A longitudinal study of mediation
Thus, we controlled for three level 1 variables (age,
pubertal status, and treatment delivery method), two level 2 variables (baseline social status and baseline BMI), and the interaction
between age and BMI in cross-sectional multilevel models.