Sentences with phrase «brain disease called»

This is one reason football players have high rates of the chronic degenerative brain disease called chronic traumatic encephalopathy.

Last year Ionis had a massive success with an ASO for another brain disease called spinal muscular atrophy (SMA).

Meanwhile, researchers have treated another inherited childhood brain disease called Canavan disease with gene therapy.

For example, there is growing awareness of a particularly severe degenerative brain disease called chronic traumatic encephalopathy (CTE).

HARD KNOCKS A degenerative brain disease called chronic traumatic encephalopathy turns up in athletes like football players who take many hits to the head.

Head impacts, not just concussions, may lead to the degenerative brain disease called chronic traumatic encephalopathy (CTE), according to new research.

The test could enable earlier diagnosis of a group of degenerative brain diseases called synucleinopathies, which include Parkinson's disease, Lewy Body dementia, and multiple system atrophy.

Not exact matches

Unlike some of the promising treatments that have failed in 2017 that deal with the so - called «amyloid hypothesis» (the treatments target amyloid beta deposits in the brain that accumulate in people with Alzheimer's disease), approaches that try to prevent nerve cells from dying wouldn't have any impact on that buildup.

This drug is designed to reduce the build up of a neural protein in the brain called alpha - synuclein that is associated with the disease.

satan the christian created foe appears to be in u. this disease called religion has your brain mashed like taters

This research noticed a strong correlation between insulin resistance in the brain and early Alzheimer's Disease, suggesting that Alzheimer's might be considered a neuroendocrine disorder of the brain or so - called «type 3 diabetes».

«BPA - free» means that the bottle was manufactured without the industrial chemical called bisphenol A. BPA has been connected to negative health effects on the brain, heart diseases, reproductive disorders and impotence for men.

The goal of treatment is to lower the bilirubin level to prevent the buildup of toxic levels in the baby's brain (a disease called kernicterus).

Although the prevailing idea has been that the devastating disease, which strikes some 1 percent of U.S. adults, is primarily caused by something going wrong with neurons, the scientists suspected the brain's support cells, called glia.

HARD KNOCKS By studying the brains of former football players, researchers are finding clues about how a neurodegenerative disease called chronic traumatic encephalopathy, or CTE, progresses, with the hopes of one day preventing it.

Degenerative brain diseases like mad cow disease (officially known as bovine spongiform encephalopathy, or BSE), scrapie in sheep, and vCJD in humans are thought to be caused by prions, misfolded versions of a normal cellular protein called PrPC.

All these diseases share a common feature: abnormal buildup of a protein called tau in the brains of patients.

V: An addiction has been classically understood as a disease of the primitive limbic brain, not of the cortical areas that are involved in what we call executive function.

Beyond PSP, other brain diseases are also marked by abnormal tau clumps — among them a rare movement disorder called corticobasal degeneration, an inherited form of frontotemporal dementia with Parkinson's - like features, and Alzheimer's disease.

The new findings suggest a simple blood test can accurately predict levels of a protein called amyloid beta in the brain that begins appearing early in the course of the disease before symptoms appear.

In humans, Huntington's is an inherited disease caused by a gene encoding a toxic protein, called mutant huntingtin, which causes brain cells to die.

At the same time, researchers have found that much smaller protein clusters called oligomers — made of only a few copies of these proteins — can be highly toxic to motor neuron - like cells grown in the lab and thus are more likely to be the chief causes of brain - cell death in these diseases.

According to a leading theory, proteins called amyloid beta and tau build up in the brain and choke nerve cell communication, setting the disease in motion years before people suspect anything is wrong with their recall.

Previous studies of people with Huntington's disease point to a link between low levels of a neurotrophin called brain - derived neurotrophic factor (BDNF) and symptoms of the disorder.

Before winning his Nobel prize, Stanley Prusiner was ridiculed for suggesting that something he called a prion caused spongiform brain diseases

According to the proposal, called the amyloid hypothesis, Alzheimer's disease, estimated to affect more than 5 million people in the United States alone, is caused by abnormal buildup of A-beta protein in the brain.

The fatal disease, which they have called bovine spongiform encephalopathy, causes degeneration of the brain.

They found that although the protein stayed soluble for a week or two, it eventually polymerized into long fibers resembling those in so - called prion diseases — brain diseases such as scrapie in sheep, «mad cow disease» in cattle, and Creutzfeldt - Jakob disease in humans.

The dye, called Congo red, breaks apart hallmark protein clumps in the brain, adding to evidence that these globs are to blame for symptoms of the disease.

This week in Science we profile Yale School of Medicine neuropathologist Laura Manuelidis, who has spent her career fighting the consensus that misfolded proteins called prions cause «mad cow disease» and other related brain diseases.

Misshapen proteins called prions lie at the root of mad cow disease and similar brain ailments, but the role of these molecules in their normal form remains unclear.

These chemicals, called cytokines, drive the inflammation in the brain, attracting more immune cells, and causing the debilitating disease marked by loss of neurological function.

The brains of people with Alzheimer's show several signs of the disease: plaques made of a protein called amyloid - β, tangles of a protein called tau and the loss of neurons.

Mouse brain nerve cells (green) making a disease - causing version of the tau protein were grown in lab dishes with supporting brain cells called glia.

The two main abnormalities of the disease are microscopic lesions called plaques and tangles, which occur in the brains of patients.

Brains of patients with Alzheimer's disease clog up too, but with plaques made from a different protein called amyloid beta peptide.

Several factors have been implicated in Alzheimer's, including the build - up of an abnormal protein called beta amyloid, fibrous tangles in the brain involving abnormal forms of a protein called tau, and — most recently — an association between the disease and a gene called ApoE.

Called craniotomy, this skull - cutting technique is still used today during brain surgery to treat neurological diseases, injuries, tumors, and blood clots.

The technique used is called deep brain stimulation, and is already used to treat the tremors and movement problems of some people with severe Parkinson's disease.

Charles DeCarli, director of the Alzheimer's Disease Center and an author of the study, said it is a wake - up call that, just as people can influence their late - life brain health by limiting vascular brain injury through controlling their blood pressure, the same is true of getting a handle on their serum cholesterol levels.

In Alzheimer's disease, an abnormal protein called amyloid beta begin s to appear on the neurons, forming plaques and compromising brain activity.

Various studies have linked Alzheimer's disease to the accumulation of two particular proteins in the brain called amyloid - beta and tau.

In 1993, Poirier and his Montreal - based team co-discovered an important genetic risk factor involved in the most common form of the disease: a defective gene, called Apolipoprotein E type 4 (ApoE4), that prevents the normal transport of cholesterol and phospholipids to the brain.

Auriel Willette, a researcher in food science and human nutrition at Iowa State University, found evidence that an elevated presence of a protein called neuronal pentraxin - 2 may slow cognitive decline and reduce brain atrophy in people with Alzheimer's disease.

They focused on three different types of PVD: arterial disease in the lower extremities, called peripheral artery disease; carotid artery stenosis, which is blockage in the carotid arteries, the major blood vessels in the neck that supply blood to the brain, neck and face; and abdominal aortic aneurysm, an enlargement of the lower aorta, the major blood vessel that supplies blood to the body.

This is largely due to life - threatening infections, as well as brain inflammation, activated by the body's natural disease - fighting immune response, called «complement».

The disease, which afflicts 270,000 people in the United States, has been linked to a mutation in a gene, called HD, that is thought to help the brain develop.

For nearly 30 years, researchers have gathered evidence that a group of bizarre, fatal brain diseases — including mad cow and its human equivalent, Creutzfeldt - Jakob disease — are caused not by a virus or bacterium but by an abnormal form of a protein, called a prion.

Auriel Willette, a researcher in food science and human nutrition, found evidence that an elevated presence of a protein called neuronal pentraxin - 2 may slow cognitive decline and reduce brain atrophy in people with Alzheimer's disease.

The protein, called neuregulin - 1, has many forms and functions across the brain and is already a potential target for brain disorders such as Parkinson's disease, amyotrophic lateral sclerosis and schizophrenia.