Sentences with phrase «brain of a patient»

The link between depression and the immune system has been shown using positron emission tomography using a tracer for the translocator protein (TSPO) showing increased immune activation in the brains of patients with major depressive disorder compared with control subjects.
«New findings about stem cells in the brain of patients with epilepsy.»
Most recently, he noted, researchers reported in Science that sleep functions as a kind of «sewer system» for the brain, at least in mice, by flushing beta - amyloid, which is known to accumulate in the brains of patients with Alzheimer's disease.
A new study in Biological Psychiatry: Cognitive Neuroscience and Neuroimaging using magnetic resonance imaging (MRI) reports a link between reduced functional activation and reduced cortical thickness in the brains of patients with bipolar disorder.
Using a different type of MRI imaging, researchers at the University of Iowa have discovered previously unrecognized differences in the brains of patients with bipolar disorder.
Using an MRI technique that is sensitive to certain byproducts of cell metabolism, including levels of glucose and acidity, University of Iowa researchers discovered previously unrecognized differences in the brains of patients with bipolar disorder.
The work should help scientists understand precisely what goes wrong in the brains of patients and help them design better therapies, Costa says.
James Ironside, a pathologist at the National CJD Surveillance Unit, discovered that the brains of these patients showed unusual spongiform symptoms.
All these diseases share a common feature: abnormal buildup of a protein called tau in the brains of patients.
Beginning in 2009, they used functional magnetic resonance imaging (fMRI) to scan the brains of patients prior to treatment for depression; they then followed the patients through the course of therapy, generally for four weeks.
«They may not look like a big deal from the outside, but if you listen in to the brain of a patient having one of these seizures, you can hear that the brain is in seizure,» says Josef Parvizi, a Stanford neuroscientist and epilepsy specialist who developed the brain stethoscope with colleague Chris Chafe, a music researcher at Stanford.
A recent study published in Annals of Neurology reports that healthy human tissue grafted to the brains of patients with Huntington's disease in the hopes of treating the neurological disorder also developed signs of the illness, several years after the graft.
Once the electrodes were placed on the brains of each patient, Shestyuk and her colleagues conducted a series of eight tasks that included visual and auditory stimuli.
In fact, several national guidelines for doctors specifically discourage scanning the brains of patients who complain of headache and migraine.
In another study, the brain of a patient who appeared to be in a vegetative state responded just like a healthy person's when asked to imagine playing a game of tennis.
Before and after treatment the brains of patients were examined with magnetic resonance imaging (MRI).
German physician Aloysius «Alois» Alzheimer (left) first described the disease that would bear his name after finding altered proteins in the brain of his patient Auguste Deter (right).
A few molecules of the protein, they found, can trigger the formation of long protein fibers similar to those in the brains of patients with neurological disorders such as Creutzfeld - Jakob disease.
Accumulations of plaques and tangles in the brains of patients with Alzheimer's disease were first observed more than a century ago.
Dr Ilse Pienaar, a neuroscientist at Sussex University, said: «At present, treatments are aimed at the whole brain of patient's with Parkinson's disease.
After just nine weeks of internet - delivered cognitive behavioral therapy, the brain of patients suffering from social anxiety disorder changes in volume.
The UCLA researchers, led by David Eisenberg, director of the UCLA - Department of Energy Institute of Genomics and Proteomics and a Howard Hughes Medical Institute investigator, report the first application of this technique in the search for molecular compounds that bind to and inhibit the activity of the amyloid - beta protein responsible for forming dangerous plaques in the brain of patients with Alzheimer's and other degenerative diseases.
«Although previous research has shown that some head injury patients have these amyloid plaques shortly after the incident, these findings suggest these plaques are still present in the brains of patients over 10 years later.
Let's say I understand precisely what is going wrong in the brain of a patient.
By the time we get to look at the brain of a patient with Alzheimer's, it's really end - stage disease.
The two main abnormalities of the disease are microscopic lesions called plaques and tangles, which occur in the brains of patients.
Brains of patients with Alzheimer's disease clog up too, but with plaques made from a different protein called amyloid beta peptide.
He contends the vitamins neutralize an oxidized compound that causes hallucinations when it accumulates in the brains of patients.
«We need to understand what these antibodies do in the brains of patients better,» he says.
The ever - growing crowd of misfolded proteins form the aggregates seen in the brains of patients with neurodegenerative disorders such as Parkinson's and Alzheimer's diseases.
Fried implanted hair - thin wires in the brains of patients at the David Geffen School of Medicine at U.C.L.A..
«Beta - amyloid dimers found in brains of patients with Alzheimer's.»
We have found that the dimers are preserved during the tissue extraction procedure used in the lab and are consequently identical to those in the brains of patients with AD.
Researchers at Rice's Center for Theoretical Biological Physics used computer models to analyze proteins suspected of misfolding and forming plaques in the brains of patients with neurological diseases.
In the case of Parkinson's disease, it is found that the expression of MAO - B, but not MAO - A, is significantly enhanced in the brain of patients and increases with age.
In a June paper in the Proceedings of the National Academy of Sciences, the Lund team described the brain of a patient who'd received a unilateral transplant — where only one side of the brain received the treatment — 24 years earlier.
In the brains of patients with Alzheimer's, amyloid peptides aggregate to form oligomers and plaques that are thought to be responsible for the disease symptoms.
Penn State researchers were the first to discover increased iron levels in the brains of some patients with the late - onset neurodegenerative disorders Parkinson's disease and Alzheimer's disease.
«When studying magnetic resonance images of the brains of patients suffering from CRPS, we noticed that the choroid plexus was nearly one - fifth larger in patients than in healthy control subjects,» says Postdoctoral Researcher Guangyu Zhou from Aalto University Department of Neuroscience and Biomedical Engineering (NBE), who analysed the images.
In the brains of patients with Alzheimer's disease (AD), amyloid precursor protein is broken apart, and the resulting fragments — β - amyloid peptides, or Aβ peptides — aggregate to form plaques.
A new study casts doubts on a long - held theory about what goes awry in the brains of patients with Huntington's disease.
We have just started a multicenter trial using simvastatine to reduce inflammation in the brain of patients with schizophrenia.
Combined with previous studies on other disorders, the new study suggests that L1 genes are indeed more active in the brain of patients with neuropsychiatric diseases, Gage says.
For the first time ever, thin electrical wires were surgically implanted into the frontal lobes of the brains of patients with Alzheimer's disease to determine if using a brain pacemaker could improve cognitive, behavioral, and functional abilities in patients with this form of dementia.
So we documented plasticity in the brain of the patient.
Elongated fibres (fibrils) of the beta - amyloid protein form the typical senile plaques present in the brains of patients with Alzheimer's disease.
Brains of patients with epilepsy may have smaller overall areas and intensity of activation in their alertness networks, which keep brains ready for incoming stimuli.
In one instance, they showed structures that plan and then activate movement, which tend to interact in one direction in control subjects, may have abnormal bidirectional interactions in the brains of patients with temporal lobe epilepsy.
A recent human study also indicated a genetic association of the αCaMKII gene with bipolar disorder, and decreased expression of αCaMKII has been observed in postmortem brains of patients with bipolar disorder.
For decades, most researchers have agreed that these plaques and tangles are the key malefactors of dementia, and that if you could clear them from the brains of patients, you would halt or reverse illness.
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