Sentences with phrase «brain reward activation»

Insulin sensitivity and brain reward activation in overweight Hispanic girls: a pilot study.

Not exact matches

Research has linked the positive feeling we often experience while talking about ourselves to higher levels of activation in areas of the brain associated with reward.

In brain - imaging studies, teen brains show more activation in regions that process rewards, motivations and emotions (the socioaffective circuitry in the subcortical, limbic regions) compared to children and adults.

«The self - expansion activities yielded significantly greater activation in a major reward region of the brain, which is associated with addictive behaviors, than did non-expanding conditions.

Activation of a reward - processing brain region peaks in the morning and evening and dips at 2 p.m., finds a study of healthy young men published in The Journal of Neuroscience.

Greg Murray and colleagues compared activation of the brain's reward system in 16 men during a gambling task at 10 a.m., 2 p.m. and 7 p.m..

In Callie's scans, Berns observes activation in the caudate, a brain region involved in processing rewards, after exposing her to the smell of his daughter.

At the same time, the obese girls sipping milk shakes showed decreased activation in the striatum, a region near the center of the brain that is studded with dopamine receptors and known to respond to stimuli associated with rewards.

The chronic dieters showed activations in areas of the brain associated with desire and expectation of reward, however.

Functional MR imaging taken while the animals received either a juice reward or VTA stimulation revealed that both induced activation of brain regions that previous studies in humans and other primates have associated with reward signaling by means of the neurotransmitter dopamine.

If obese individuals must eat more and more to overcome reduced activation of reward networks in the brain, that sounds a lot like tolerance.

As the study was conducted in a brain scanner at the LIFE&BRAIN Center in Bonn, researchers could also show that products labeled with this emblem led to increased activity in specific brain regions: For example, they observed increased activation in regions important for reward processing as well as frontal regions that process abstract product attributes (e.g. whether or not a product carries a Fair Trade logo, and the meaning of such a label).

Over the following year, those whose brains showed less activation in areas known to respond to natural rewards like foods ended up gaining more weight — though only if they had a particular genetic makeup.

The study's results suggested that girls whose brains showed less reward system activation to foods also gained more weight — if they had the DRD2 Taq1AQ1 or the DRD4 7 + repeat alleles.

Using further neurological studies, George and his colleagues tracked this compulsive behavior to the activation of «stress» and «reward» pathways in the brain.

This decreased reward response to alcohol arose via two mechanisms: an initial activation of stress hormone receptors and a subsequent increase in inhibitory signaling in the brain.

In different experiments we will examine the effects of exogenous (intranasal oxytocin) and endogenous (providing social support) oxytocin on fear and reward related brain activation patterns, functional connectivity, stress reactivity and trauma - related psychopathology symptoms.

They found that individuals with genetic variations to the RASGRF2 gene had higher activation of the ventral striatum area of the brain (closely linked to the VTA and involved in dopamine release) when anticipating reward in a cognitive task.

Brain activation (fMRI) was significantly altered in XR - NTX - treated individuals in regions associated with the integration of emotion, cognition, reward, punishment, and learning / memory.

Satiety and physiological hunger was assessed by measuring hormonal markers and perceived appetite sensations combined with psychological reward - driven eating motivation, making use of fMRI to determine brain activation in certain areas associated with food motivation and reward.

At the same time, magnetic resonance imaging (MRI) brain scans of those who drank the corn syrup drink showed increased activation in a region of the brain called the nucleus accumbens, which has been tied to reward and cravings in past research, compared with the men who drank the low - glycemic index drink.

Higher activation of this brain region has been found in obese compared with normal - weight subjects when anticipating food, suggesting that the rewarding quality of food is enhanced in obesity.

Studies show a link between high - fat and high - sugar foods and the increase in the activation of reward pathways in the brain, particularly dopamine receptors, says Erin Macdonald, R.D.

Sleep deprivation produces a «double hit» to the brain in this regard: there is a sharp reduction in frontal lobe activity leading one to misjudge hunger and eat more junk and also leading to an exaggerated activation of reward centers (mostly the amygdalae) after eating.

We found that breakfast skipping led to increased neural activation in brain regions controlling food motivation and food reward (i.e., hippocampus, amygdala, anterior cingulate, and parahippocampus) prior to lunch; however, the addition of a protein - rich breakfast led to reduced activation in these regions.

Higher plasma glucose levels correlated with greater brain activity in executive control centers in the ACC and ventromedial PFC, whereas higher levels of plasma cortisol, but not other hormones, were correlated with greater activation in reward regions, such as the insula and putamen (P < 0.01, corrected), in response to high - calorie food cues.

Scientists found that dog owners» aroma actually sparked activation in the «reward center» of their brains

Conduct - disordered youth exhibit a decreased dopamine response to reward and increased risk - taking behaviors related to abnormally disrupted frontal activity in the anterior cingulate cortex (ACC), orbitofrontal cortices (OFC), and dorsolateral prefrontal cortex (DLPFC) that worsens over time due to dysphoria activation of brain stress systems and increases in corticotropin - releasing factor (CRF).

Studies of neural processing show that risk - taking may be associated with reward - related brain activation, with risk - taking behaviors and the opposite of what normally should happen (ie, hypoactivation with disappointment losses).

For positive emotional faces, considering the spreading effects of activation of secure base schema, it was assumed that both levels (high and low) of attachment anxiety are associated with increased activities in brain circuits that are responsible for positive emotions and the regions belonging to the reward system.

During the outcome phase (during presentation of feedback), the contrast of interest was the effect of allelic groups on differences in brain activation for reward versus non-reward trials.

During the anticipation phase (the time between presentation of cues and potential reward receipt), the contrast of interest was the effect of allelic group on differences in brain activation for incentive versus non-incentive trials.