Signs of heartworm disease are caused
by damage to the heart and blood vessels as a result of the heartworms living in them.
These vibrations or murmurs are produced due to a disturbance in the blood flow through your pet's heart, usually caused
by damage to the heart valves or a change in the health of the heart valves.
Not exact matches
He opened my eyes and allowed me
to see the
damage I had been doing
to my
heart and mind
by putting Him off.
The oxidative
damage caused
to normal cells
by free radicals has been linked
to chronic conditions like cancer,
heart disease, and degenerative diseases related
to aging.
Furthermore antioxidant properties of Succinic Acid can help
to minimize disruptions of
heart rhythm and reduce
damage done
by free radicals.
Comparing the
heart rate or blood sugar levels of a given number of people might be beneficial in determining the range in which people maintain good health — and perhaps we can even say that
by comparing children's abilities and establishing a range of «normal,» we can determine which children have difficulties and how
to help them — but comparing ourselves with others, and in particular our children
to other children, can have very
damaging effects.
This practice is used
by doctors
to detect complications like low oxygen and
heart issues that can cause brain
damage, seizures and even death in babies.
Betty McBride, director of policy at the British
Heart Foundation (BHF), added: «Exposure to second - hand smoke by non-smokers for just half an hour is enough to damage the lining of the heart's arteries and increase the risk of a heart at
Heart Foundation (BHF), added: «Exposure
to second - hand smoke
by non-smokers for just half an hour is enough
to damage the lining of the
heart's arteries and increase the risk of a heart at
heart's arteries and increase the risk of a
heart at
heart attack.
Reperfusion involves opening the blocked artery
by surgery or pharmaceuticals, and has been shown
to significantly reduce
damage to the
heart.
Once inside, they could begin
to repair the
damage caused
by a
heart attack.
A cadre of scientists and companies is now trying
to prevent or reverse cardiac
damage by infusing a cocktail of stem cells into weakened
hearts.
Nitric Oxide protects the
heart from
damage caused
by ischemia, or blocked blood flow
to heart tissue.
The complaint, filed in a federal district court
by two prominent
heart researchers, offers the first indication of just what is amiss in two papers they co-authored, which describe the
heart's natural regenerative ability, and an effort
to heal
damaged hearts with stem cells.»
The study looked at 7,470 patients with PAD who took part in the COMPASS study, and found the combination reduced
heart attacks, stroke or cardiovascular death
by 28 per cent and
damage to limbs
by 46 percent.
By the time she was 55, she had suffered three more
heart attacks, and despite 13 stents
to prop open her arteries, the
damage has caused her left ventricle
to balloon in size.
As long ago as the 1950s, scientists discovered that many diseases — including
heart disease, strokes, cancer, diabetes, cataracts, arthritis and neurodegenerative disorders such as Parkinson's and Alzheimer's — were linked
to damage caused
by highly destructive chemicals called free radicals.
There is a lot of work still
to do, and many potential pitfalls before it could be applied
to human patients, but in principle almost any illness caused
by damaged or ageing tissue —
heart disease, diabetes, Alzheimer's and dozens of others — could be fixed this way.
A STEMI (ST segment elevation myocardial infarction)
heart attack happens when an artery is completely blocked
by the blood clot, which causes
damage to virtually all of the
heart muscle supplied
by that artery.
What's more,
by selectively deactivating certain PAMs within the matrix, the team mimicked the kind of
damage that happens
to the
heart muscle under certain disease conditions.
The finding, which was published today in the journal Nature, shows that it is possible
to limit the
damage wrought
by heart attacks, which strike nearly one million people in the United States each year.
A
heart attack occurs when blood flow
to an area of the
heart is blocked
by a narrowed or completely obstructed coronary artery, resulting in
damage of
heart muscle.
«Our findings uncover the direct coupling of these two important pathways and raise the prospect of therapeutic manipulation of the UPR
to lessen the
damage caused
by heart attack and stroke,» said Dr. Joseph A. Hill, Professor of Internal Medicine and Molecular Biology, and senior author of the study published in the March 13 issue of Cell.
Most of the time when the
heart is injured, these beneficial immune cells are supplanted
by immune cells from the bone marrow, which are spurred
to converge in the
heart and cause inflammation that leads
to further
damage.
Most importantly, these studies show that treatment with vitamin D3 can significantly restore the
damage to the cardiovascular system caused
by several diseases, including hypertension, atherosclerosis, and diabetes, while also reducing the risk of
heart attack.
Several studies show that this cuts
damage to heart muscle
by about one - third following surgery
to bypass blocked arteries, when the
heart's blood supply must be stopped for up
to an hour.
Whether caused
by an undetected birth defect or
by a
heart attack (myocardial infarction), when a
heart sustains
damage, it can be difficult
to repair.
According
to cardiologist Stefanie Dimmler at the University of Frankfurt in Germany, degraded telomeres might cause
heart disease
by impeding the ability of cells from the bone marrow
to repair
damaged parts of the arterial walls.
The polymer patch could one day lay down a pathway in areas
damaged by heart disease for cells
to regenerate and regrow, while the mesh itself slowly disintegrates within the body.
Researchers have found a way
to increase the effectiveness of a widely used cancer drug while decreasing the risk of
heart -
damaging side effects, according
to a new study
by researchers from The University of Texas Medical Branch at Galveston and Texas Tech University Health Sciences Center.
A TROPICAL disease that can fatally
damage the
heart may take hold in the human body
by causing important immune system cells
to commit suicide, according
to a Brazilian research team.
The team found that inflammatory
damage in the
heart can not occur without the help of macrophages and dendritic cells made
by the spleen, and that
heart failure is
to some degree an autoimmune disease.
«The long - term risk of
heart attack was insignificant, but risk of blood clots in the lung remained for years after surgery
to replace a hip or knee
damaged by osteoarthritis.»
We are working
to transform this game - changing research into therapies that help doctors repair
hearts damaged by heart attacks and disease.
Muscle
damaged by heart attacks can be repaired
by an injectable gel that forms scaffolding, attracting stem cells and blood vessels in a study that may lead
to a new method for reducing
heart failure.
«Scientists have tried for decades
to treat
heart failure
by transplanting adult
heart cells, but these cells can not reproduce themselves, and so they do not survive in the
damaged heart,» explained Yu Zhang, MD, PhD, lead author on the study and a postdoctoral scholar at the Gladstone Institutes.
By stimulating the growth of new blood vessels, promoting anti-inflammatory effects, recruiting cells toward tissue regeneration and inhibiting further cell death, adult stem cells can restore some function
to damaged or diseased
heart muscle.
With the loss of the cushioning effect provided
by free - moving collagen in the blood vessels, the force of the surge of blood that is driven into the arteries
by the pumping action of the
heart is carried directly
to organs like the kidneys and the brain,
damaging to the structures that filter our blood and that connect the functional regions of our brain, and putting us at risk of a stroke.
Okyanos cardiac cell therapy targets the restoration of blood flow
to the
heart and repairs
damaged heart tissue caused
by heart attack or ischemic and non-ischemic
heart disease.
The U.S. Patent and Trademark Office has announced that it will grant a patent
to MDI Biological Laboratory scientists Voot P. Yin, Ph.D., and Kevin Strange, Ph.D., and their collaborator Michael Zasloff, M.D., Ph.D., for use of the small molecule MSI - 1436
to stimulate the repair and regeneration of
heart tissue
damaged by injuries such as a
heart attack.
A child's
heart is more sensitive
to injury caused
by interrupting blood flow during surgery, and excess reactive oxygen species are a key source of this
damage.
The application of these therapies could range from the repair of
heart muscle
damaged by heart attack
to the repair of sports - related bone, cartilage and muscle injuries.
Deepak Srivastava writes about how
by helping cells switch their type, we may have discovered a new way
to repair
damaged hearts, and potentially revolutionize the future of medicine.
These penetrate tissue and heat up when excited
by radio - frequency waves; and they have been shown
to thaw arteries and
heart valve tissue from pigs, as well as human skin cells, rapidly and evenly, without the typical
damage.
But
heart cells never truly regenerate in the
damaged tissue, and myofibroblasts compensate for their absence
by forming a stiff, collagenous scar that interferes with the
heart's ability
to maintain stable
heart rhythms and
to expand and contract forcefully
to pump blood.
Philadelphia, PA (Scicasts)- Researchers may have found a way
to reduce ongoing
heart damage that occurs long after a severe
heart attack
by improving the longevity of stem cells injected into the
heart.
This ground - breaking research could lead
to a novel way
to repair
heart damage after a
heart attack using a patient's own cells
by converting them within the organ into new muscle.
This treatment further provides other anti-inflammatory, healing and restorative properties
to a
heart stressed or
damaged by ischemia.
Researchers and clinicians imagined using stem cells
to undo the
damage caused
by a
heart attack.
But the
damage caused
by a
heart attack can result in
heart failure — and patients diagnosed with
heart failure have an average life expectancy of less than five years, according
to Dr. Chuck Murry, director of the Center for Cardiovascular Biology at the University of Washington in Seattle.
CINCINNATI - Scientists used an experimental targeted molecular therapy
to block a matrix - forming protein in
heart cells
damaged by heart attack, reducing levels of scarred muscle tissue and saving mouse models from
heart failure.