Sentences with phrase «by damage to the heart»

Signs of heartworm disease are caused by damage to the heart and blood vessels as a result of the heartworms living in them.
These vibrations or murmurs are produced due to a disturbance in the blood flow through your pet's heart, usually caused by damage to the heart valves or a change in the health of the heart valves.

Not exact matches

He opened my eyes and allowed me to see the damage I had been doing to my heart and mind by putting Him off.
The oxidative damage caused to normal cells by free radicals has been linked to chronic conditions like cancer, heart disease, and degenerative diseases related to aging.
Furthermore antioxidant properties of Succinic Acid can help to minimize disruptions of heart rhythm and reduce damage done by free radicals.
Comparing the heart rate or blood sugar levels of a given number of people might be beneficial in determining the range in which people maintain good health — and perhaps we can even say that by comparing children's abilities and establishing a range of «normal,» we can determine which children have difficulties and how to help them — but comparing ourselves with others, and in particular our children to other children, can have very damaging effects.
This practice is used by doctors to detect complications like low oxygen and heart issues that can cause brain damage, seizures and even death in babies.
Betty McBride, director of policy at the British Heart Foundation (BHF), added: «Exposure to second - hand smoke by non-smokers for just half an hour is enough to damage the lining of the heart's arteries and increase the risk of a heart atHeart Foundation (BHF), added: «Exposure to second - hand smoke by non-smokers for just half an hour is enough to damage the lining of the heart's arteries and increase the risk of a heart atheart's arteries and increase the risk of a heart atheart attack.
Reperfusion involves opening the blocked artery by surgery or pharmaceuticals, and has been shown to significantly reduce damage to the heart.
Once inside, they could begin to repair the damage caused by a heart attack.
A cadre of scientists and companies is now trying to prevent or reverse cardiac damage by infusing a cocktail of stem cells into weakened hearts.
Nitric Oxide protects the heart from damage caused by ischemia, or blocked blood flow to heart tissue.
The complaint, filed in a federal district court by two prominent heart researchers, offers the first indication of just what is amiss in two papers they co-authored, which describe the heart's natural regenerative ability, and an effort to heal damaged hearts with stem cells.»
The study looked at 7,470 patients with PAD who took part in the COMPASS study, and found the combination reduced heart attacks, stroke or cardiovascular death by 28 per cent and damage to limbs by 46 percent.
By the time she was 55, she had suffered three more heart attacks, and despite 13 stents to prop open her arteries, the damage has caused her left ventricle to balloon in size.
As long ago as the 1950s, scientists discovered that many diseases — including heart disease, strokes, cancer, diabetes, cataracts, arthritis and neurodegenerative disorders such as Parkinson's and Alzheimer's — were linked to damage caused by highly destructive chemicals called free radicals.
There is a lot of work still to do, and many potential pitfalls before it could be applied to human patients, but in principle almost any illness caused by damaged or ageing tissue — heart disease, diabetes, Alzheimer's and dozens of others — could be fixed this way.
A STEMI (ST segment elevation myocardial infarction) heart attack happens when an artery is completely blocked by the blood clot, which causes damage to virtually all of the heart muscle supplied by that artery.
What's more, by selectively deactivating certain PAMs within the matrix, the team mimicked the kind of damage that happens to the heart muscle under certain disease conditions.
The finding, which was published today in the journal Nature, shows that it is possible to limit the damage wrought by heart attacks, which strike nearly one million people in the United States each year.
A heart attack occurs when blood flow to an area of the heart is blocked by a narrowed or completely obstructed coronary artery, resulting in damage of heart muscle.
«Our findings uncover the direct coupling of these two important pathways and raise the prospect of therapeutic manipulation of the UPR to lessen the damage caused by heart attack and stroke,» said Dr. Joseph A. Hill, Professor of Internal Medicine and Molecular Biology, and senior author of the study published in the March 13 issue of Cell.
Most of the time when the heart is injured, these beneficial immune cells are supplanted by immune cells from the bone marrow, which are spurred to converge in the heart and cause inflammation that leads to further damage.
Most importantly, these studies show that treatment with vitamin D3 can significantly restore the damage to the cardiovascular system caused by several diseases, including hypertension, atherosclerosis, and diabetes, while also reducing the risk of heart attack.
Several studies show that this cuts damage to heart muscle by about one - third following surgery to bypass blocked arteries, when the heart's blood supply must be stopped for up to an hour.
Whether caused by an undetected birth defect or by a heart attack (myocardial infarction), when a heart sustains damage, it can be difficult to repair.
According to cardiologist Stefanie Dimmler at the University of Frankfurt in Germany, degraded telomeres might cause heart disease by impeding the ability of cells from the bone marrow to repair damaged parts of the arterial walls.
The polymer patch could one day lay down a pathway in areas damaged by heart disease for cells to regenerate and regrow, while the mesh itself slowly disintegrates within the body.
Researchers have found a way to increase the effectiveness of a widely used cancer drug while decreasing the risk of heart - damaging side effects, according to a new study by researchers from The University of Texas Medical Branch at Galveston and Texas Tech University Health Sciences Center.
A TROPICAL disease that can fatally damage the heart may take hold in the human body by causing important immune system cells to commit suicide, according to a Brazilian research team.
The team found that inflammatory damage in the heart can not occur without the help of macrophages and dendritic cells made by the spleen, and that heart failure is to some degree an autoimmune disease.
«The long - term risk of heart attack was insignificant, but risk of blood clots in the lung remained for years after surgery to replace a hip or knee damaged by osteoarthritis.»
We are working to transform this game - changing research into therapies that help doctors repair hearts damaged by heart attacks and disease.
Muscle damaged by heart attacks can be repaired by an injectable gel that forms scaffolding, attracting stem cells and blood vessels in a study that may lead to a new method for reducing heart failure.
«Scientists have tried for decades to treat heart failure by transplanting adult heart cells, but these cells can not reproduce themselves, and so they do not survive in the damaged heart,» explained Yu Zhang, MD, PhD, lead author on the study and a postdoctoral scholar at the Gladstone Institutes.
By stimulating the growth of new blood vessels, promoting anti-inflammatory effects, recruiting cells toward tissue regeneration and inhibiting further cell death, adult stem cells can restore some function to damaged or diseased heart muscle.
With the loss of the cushioning effect provided by free - moving collagen in the blood vessels, the force of the surge of blood that is driven into the arteries by the pumping action of the heart is carried directly to organs like the kidneys and the brain, damaging to the structures that filter our blood and that connect the functional regions of our brain, and putting us at risk of a stroke.
Okyanos cardiac cell therapy targets the restoration of blood flow to the heart and repairs damaged heart tissue caused by heart attack or ischemic and non-ischemic heart disease.
The U.S. Patent and Trademark Office has announced that it will grant a patent to MDI Biological Laboratory scientists Voot P. Yin, Ph.D., and Kevin Strange, Ph.D., and their collaborator Michael Zasloff, M.D., Ph.D., for use of the small molecule MSI - 1436 to stimulate the repair and regeneration of heart tissue damaged by injuries such as a heart attack.
A child's heart is more sensitive to injury caused by interrupting blood flow during surgery, and excess reactive oxygen species are a key source of this damage.
The application of these therapies could range from the repair of heart muscle damaged by heart attack to the repair of sports - related bone, cartilage and muscle injuries.
Deepak Srivastava writes about how by helping cells switch their type, we may have discovered a new way to repair damaged hearts, and potentially revolutionize the future of medicine.
These penetrate tissue and heat up when excited by radio - frequency waves; and they have been shown to thaw arteries and heart valve tissue from pigs, as well as human skin cells, rapidly and evenly, without the typical damage.
But heart cells never truly regenerate in the damaged tissue, and myofibroblasts compensate for their absence by forming a stiff, collagenous scar that interferes with the heart's ability to maintain stable heart rhythms and to expand and contract forcefully to pump blood.
Philadelphia, PA (Scicasts)- Researchers may have found a way to reduce ongoing heart damage that occurs long after a severe heart attack by improving the longevity of stem cells injected into the heart.
This ground - breaking research could lead to a novel way to repair heart damage after a heart attack using a patient's own cells by converting them within the organ into new muscle.
This treatment further provides other anti-inflammatory, healing and restorative properties to a heart stressed or damaged by ischemia.
Researchers and clinicians imagined using stem cells to undo the damage caused by a heart attack.
But the damage caused by a heart attack can result in heart failure — and patients diagnosed with heart failure have an average life expectancy of less than five years, according to Dr. Chuck Murry, director of the Center for Cardiovascular Biology at the University of Washington in Seattle.
CINCINNATI - Scientists used an experimental targeted molecular therapy to block a matrix - forming protein in heart cells damaged by heart attack, reducing levels of scarred muscle tissue and saving mouse models from heart failure.
a b c d e f g h i j k l m n o p q r s t u v w x y z