This study, by Professor Jeremy Henley and co-workers reports a new type of LTP that is controlled
by kainate receptors.
Not exact matches
Publication of Proof that Cot Death Babies Show Physiological Effects of Gaseous Poisoning: «Decreased
Kainate Receptor Binding in the Arcuate Nucleus of the Sudden Infant Death Syndrome», Journal of Neuropathology and Experimental Neurology 1997; 56:1253 - 61: proof that cot death babies have neurochemical deficits consistent with poisoning
by nerve gases.
The whole - cell current response to glutamate and
kainate [a non-NMDA (N - methyl - D - aspartate) receptor agonist] was enhanced
by forskolin, an activator of adenylate cyclase.
Functional expression of the cDNAs in cultured mammalian cells generated receptors displaying alpha - amino -3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)- selective binding pharmacology (AMPA = quisqualate greater than glutamate greater than
kainate) as well as cation channels gated
by glutamate, AMPA, and
kainate and blocked
by 6,7 - dinitroquinoxaline -2,3-dione (CNQX).