Malaria is caused
by the parasite Plasmodium.
Avian malaria is mainly caused
by the parasite Plasmodium relictum, which reproduces in red blood cells.
Severe forms of malaria infection are caused
by the parasite Plasmodium falciparum, which is spread by mosquitoes.
Not exact matches
When a human host is bitten
by an infected mosquito, the
parasite (
Plasmodium) enters the blood and lays inactive in the liver.
The mosquito - borne disease caused
by the
Plasmodium parasite kills more than 700,000 people every year, the vast majority of them African children, and no vaccine has ever shown efficacy in trials.
Malaria is caused
by a handful of species of
parasites in the genus
Plasmodium through the bite of mosquitos and remains a widespread vector - borne infectious disease, sickening almost half a billion people every year around the planet.
Those who were not infected much
by the malaria
parasite Plasmodium while breeding also showed purer white cheek feathers in winter.
The
Plasmodium falciparum
parasite is responsible for most malaria infections and almost all deaths caused
by the disease worldwide.
Growing resistance to malaria drugs in Southeast Asia is caused
by a single mutated gene inside the disease - causing
Plasmodium falciparum
parasite, according to a study led
by David Fidock, PhD, professor of microbiology & immunology and of medical sciences (in medicine) at Columbia University Medical Center.
The gene codes for an immune receptor on red blood cells; lack of that receptor prevents infection
by Plasmodium vivax, a species of the malaria
parasite.
A serious and sometimes fatal infectious disease that is spread
by infected mosquitoes, malaria and its
parasite Plasmodium falciparum, is responsible for nearly 450,000 deaths every year, the majority of them children under the age of five.
Plasmodium falciparum, a blood - borne
parasite carried
by mosquitoes, is responsible for most of the estimated 219 million cases, and 655,000 deaths, from malaria per year.
Genetic variability of
Plasmodium falciparum underlies its transmission success and thwarts efforts to control disease caused
by this
parasite.
Transmitted
by Anopheles mosquitoes, the
Plasmodium falciparum
parasite infects hundreds of millions of people every year and may kill more than a million.
The researchers compared a modern - day map from 2007 that shows the spread of infection
by the malaria
parasite Plasmodium falciparum throughout the world to a similar map constructed in 1900.
By chance, she had discovered a malaria
parasite,
Plasmodium odocoilei — that infects white - tailed deer.
The Coban Team infected mice with a mutant
Plasmodium parasite producing less
by - products such as hemozoin, and discovered in this case bone loss did not occur, thereby confirming their findings.
Malaria is caused
by a single - celled
parasite called
Plasmodium that spreads from person to person through mosquito bites.
Malaria is caused
by Plasmodium parasites that are transmitted to humans
by a mosquito bite, leading to 219 million documented cases and 627,000 deaths worldwide in 2012.
Malaria is caused
by Plasmodium, a microscopic
parasite spread to humans
by mosquitoes.
An estimated 220 million people are infected each year
by malaria - causing
Plasmodium parasites, which are transmitted
by the bite of an infected mosquito.
A team led
by parasitologist Stefan Kappe at the Center for Infectious Disease Research in Seattle in Washington gave a rodent version of this «genetically attenuated
parasite,» or GAP, to mice and showed that they were completely protected when later infected with an unmodified — or wild - type — version of the same
Plasmodium strain.
Plasmodium cynomolgi, a non-human primate malaria
parasite first mentioned
by Mayer in 19071 and established as a separate species from P. inui
by Mulligan in 19352, has been used as a model
parasite species since its discovery.
The variant surface antigens expressed on
Plasmodium falciparum - infected erythrocytes are potentially important targets of immunity to malaria and are encoded, at least in part,
by a family of var genes, about 60 of which are present within every
parasite genome.
Malaria results from infection of human red blood cells (RBC)
by the
plasmodium parasite.
Scientists are making strides in understanding how the malaria
parasite Plasmodium falciparum disguises itself to avoid detection
by the immune system.
Many existing treatments for malaria - one of the deadliest infectious diseases - target only one stage of the complex life cycle of the
Plasmodium parasite that causes the disease, and are further limited
by the emergence of drug resistance in particular strains.
Here we show that
Plasmodium blood - stage
parasites actively respond to host dietary calorie alterations through rearrangement of their transcriptome accompanied
by substantial adjustment of their multiplication rate.
The publication of the P. falciparum genome project in 2002 [10] identified the full complement of
parasite proteins but progress in understanding the function of these proteins, including those displayed on the merozoite cell surface, has been hindered
by the technical difficulties in expressing
Plasmodium proteins in a functionally active form [11].
Plasmodium falciparum malaria parasitaemic blood, screened
by light microscopy, was depleted of leucocytes using CF11 cellulose columns and the
parasite genotype ascertained
by sequencing on the Illumina HiSeq platform.
In this study, we begin the empirical effort to determine the likely direction of immune - mediated virulence evolution
by performing experimental evolution of the rodent malaria
parasite,
Plasmodium chabaudi, in laboratory mice.
The disease is spread
by the female Anopheles mosquito, which transfers the
plasmodium parasite when it feeds on human blood.
Once a person is bitten
by an infected mosquito, the
plasmodium parasite is released into the bloodstream where it makes its way to the person's liver.
Malaria is caused
by species of single - celled
parasites in the genus
Plasmodium, vectored
by mosquitoes primarily in the genera Aedes and Anopheles between many vertebrate hosts, including humans.
Genome sequence and comparative analysis of the model rodent malaria
parasite Plasmodium yoelii yoelii, 41 9 Nature 5 12 - 5 19, Co-Authored
by Carlton, J. et al., 2002