When the parathyroid glands release more parathyroid hormones, it causes the bones to release more calcium into the blood and reduces the amount of
calcium released by the kidneys.
Inhibition of both oxytocin - induced contraction and
calcium release by the traditional NAADP - signaling disrupter bafilomycin and the NAADP receptor antagonist Ned - 19 clearly demonstrated a role for NAADP in oxytocin - induced signaling.
Regulation of inositol 1, 4, 5 - trisphosphate receptor - mediated
calcium release by the Na / K - ATPase in cultured renal epithelial cells.
Not exact matches
When elevating
calcium ion concentration directly at the presynaptic terminal with the use of caged
calcium, cleavage of SNAP - 25
by botulinum toxin A (BoNT / A) produced a strong reduction in the
calcium sensitivity for
release, whereas cleavage of syntaxin using BoNT / C1 and synaptobrevin using tetanus toxin (TeNT) produced an all - or - nothing block without changing the kinetics of remaining vesicles.
This solution would then be reacted with limestone, neutralizing the CO2
by converting it to
calcium bicarbonate, after which it would be
released into the ocean.
In a new study, biomedical engineers found that
by attaching microscopic beads to receptors on the cell's surface, they can produce the technique's cell - stretching,
calcium -
releasing effects much more safely.
Stimulating the plant, such as
by touch or wind pressure, triggers the
release of this
calcium into the main body of a cell, where sensitive
calcium receptors register the nature of the stimulus and mount a biochemical response to it.
Astrocytes also form their own long - distance communication networks
by «talking» via waves of
calcium ions, and, like neurons, they can receive and
release neurotransmitters.
While most cases of osteoporosis are caused
by normal aging, another leading cause of the bone - loss disease is a condition called hyperparathyroidism, in which the parathyroid glands
release an excessive amount of a hormone that regulates the body's
calcium levels.
Rome and colleagues therefore measured the amount of
calcium released and pumped
by midshipman swimbladder muscles as they repeatedly contract and relax.
Nothing was known about neurotransmitter
release except that it was triggered
by calcium.
Increasing
calcium avidity, for instance, decreased
by approximately the same amount how much
calcium was needed for neurotransmitter
release.
Bartonella - induced endothelial cell proliferation is mediated
by release of
calcium from intracellular stores.
Responses were correlated to
calcium release in cultured rat uterine smooth muscle cells measured
by fluorescence microscopy.
Blood
calcium is very tightly self - regulated, but if
calcium is low in the blood either due to low
calcium in your diet or a deficiency caused
by another illness,
calcium is
released from the bones.
That
calcium - binding protein will then pick up a free
calcium ion, transport it through the intestinal mucosa and
release it into the blood.1 Manganese and magnesium have similar carriers and their absorption, retention and excretion is likewise governed
by complex feedback mechanisms involving other nutrients and hormonal signals.
PTH has a variety of effects including raising blood
calcium levels (
by breaking down and
releasing calcium in our bones), increasing
calcium absorption in the digestive system and it also affects our sense of well being.
Higher doses increase the force of muscle contractions, possibly
by stimulating more
calcium release from the sarcoplasmic reticulum of muscle cells.
For optimal health, our body's pH should be neutral to slightly alkaline; when it is too acidic the body buffers this acidity
by releasing calcium and magnesium from bones and breaking down muscle to produce ammonia (strongly alkaline).
Titin is activated
by calcium ions (Labeit et al. 2003; Cornachione et al. 2016), which are
released in response to the action potentials that cause activation of muscle fibers.
Boron deficiency causes the parathyroids to become overactive,
releasing too much parathyroid hormone which raises the blood level of
calcium by releasing calcium from bones and teeth.
Updated recommendations for
calcium intake based on the optimization of bone health were
released by the Food and Nutrition Board (FNB) of the Institute of Medicine in 2011 (9).
Dental Diet made
by Royal Canin also binds
calcium in the mouth, preventing it from being used to form tartar, and then
releases the
calcium in the gut where it can be used nutritionally.
High blood
calcium levels have been reported to cause increased urination, perhaps through its ability to interfere with the action of antidiuretic hormone (ADH, AVP, vasopressin)
released by your pet's pituitary gland.
The glands do that
by constantly monitoring the pet's blood
calcium level and
releasing PTH hormone when levels drop too low.