However, once opiate addiction had developed, the scientists observed a functional switch to a separate molecular memory pathway, controlled by a molecule
called calmodulin - dependent kinase II or «CaMKII.»
For starters, a key protein
called calmodulin, which helps spindles form, failed to bind to the spindles properly.
Not exact matches
Cunningham and her postdoc at the time, Christine Kirvan, found that the antibodies literally bind to human neurons, activating an enzyme
called calcium /
calmodulin - dependent protein (CaM) kinase II.
When calcium floods a neuron as a memory is formed, it turns on an enzyme
called CaMKII (calcium /
calmodulin - dependent protein kinase).