Sentences with phrase «called tumor necrosis factor»

These human astrocytes apparently did so, the scientists suggest, by secreting a protein called tumor necrosis factor - α, which mouse astrocytes produce at much lower levels.

Inflammation is a known feature of heart failure, and it is associated with high levels of inflammatory proteins called cytokines — including one called tumor necrosis factor alpha or TNF - a — that ramp up immune cell responses.

Normally, the VHL gene acts to suppress another gene called tumor necrosis factor alpha or TNF - alpha.

Infliximab reduces symptoms and inflammation by targeting one of the proteins — called tumor necrosis factor (TNF)-- that leads to inflammation in IBD.

They act by dampening part of the immune system called tumor necrosis factor (TNF).

This particular cytokine, called tumor necrosis factor alpha, seems to be damaging to taste buds, the researchers found.

Not exact matches

Specifically A20 appears to call off a substance known as tumor necrosis factor (TNF).

A Mayo Clinic study is shedding light on why some rheumatoid arthritis patients respond poorly when treated with tumor necrosis factor inhibitors, part of a class of drugs called biologics.

Researchers found that patients with a higher amount or higher proportion of an inflammatory protein called type 1 interferon beta compared with another inflammatory protein, type 1 interferon alpha, do not respond as well to tumor necrosis factor inhibitors as others.

Researchers in Sweden sought to determine if use of tumor necrosis factor inhibitor drugs to treat RA would result in a reduced risk of acute coronary syndrome (commonly called ACS), defined as a diagnosis of a heart attack or unstable angina (the worsening or increasing cardiac symptoms)

Tumor necrosis factor inhibitor drugs (commonly called Anti-TNFs) modestly reduce the risk of acute coronary syndrome, such as heart attacks and angina, in rheumatoid arthritis patients whose inflammation places them at higher risk of developing coronary heart disease, according to new research findings presented this week at the American College of Rheumatology Annual Meeting in San Diego.

They have detected, for example, revved up signaling molecules involved in inflammation, such as tumor necrosis factor α (TNFα) and other cytokines; skewed populations of natural killer cells and other immune cells; imbalances in the protein - destroying enzymes called proteases; and a shortening of the telomeres, the «end caps» on chromosomes, which indicates prematurely aged cells.

Yongwon Choi, with Ralph Steinman and others, identifies a new tumor necrosis factor family member TRANCE (later called RANKL), [i] and subsequently demonstrates its role in the immune and bone system, and also in bone - residing cancers such as multiple myelomas.