Sentences with phrase «cancer cells process»

This study shows that cancer cells process sugar differently from the rest of the cells in our body.

Not exact matches

CAR - T approaches involve reengineering T - cells to find and destroy cancer cells, but that process can cause cytokine release syndrome (CRS), a life - threatening immune response NantKwest hopes to sidestep.

As a cancer researcher, do you think the mechanisms of tumor growth are somehow changing to come into line with your perceptions, or is it possible that the process of our learning more about DNA mutations and cell architecture and nutrient exchange and epigenetic effects make it possible for us to inch ever closer to understanding that which is already going on under our noses?

Cell replication allows our bodies to grow and develop, yet can result in cancer when natural processes misfire.

Processed sugars can be very damaging to the immune system and can even feed cancer cells.

Free radicals oxidize and damage our body cells, as a main process causing aging and cancers.

It is completely different in the most important ways, which is it does NOT feed cancer cells in the way highly processed white sugar does and it does NOT cause chemical reactions in the brain (exactly like heroin)-- keeping you craving more sugary foods / drinks like highly processed white sugar does.

The body wears out, mutations accumulate faster than can be repaired just through the natural process of cell division, cancer grows, arthritis wracks the world worn joints, the child born with tetralogy of Fallot away from surgical care dies.

Cancer cells travel around the body and begin to attack healthy tissue in a process called metastasis.

Carcinogens may cause cancer by altering cellular metabolism or damaging DNA directly in cells, which interferes with normal biological processes.

Joe W. Ramos, PhD, deputy director of the UH Cancer Center and collaborators focused on investigating how these oncogenes and related signals lead to dysregulation of normal processes within the cell and activate highly mobile and invasive cancer cell behCancer Center and collaborators focused on investigating how these oncogenes and related signals lead to dysregulation of normal processes within the cell and activate highly mobile and invasive cancer cell behcancer cell behavior.

«Used in cancer therapy, this process could increase the impact of a treatment by heating the cancer cells while introducing the drug compound into the tumor.»

They're also used as biological probes to image cancer and to study processes inside cells,» Pentelute says.

Cancer stem cells can reproduce themselves through a process called self - renewal and sustain the growth of a tumor.

Furthermore, it has been shown that the body's natural process of healing the wound created by surgery can actually spur these residual cancer cells to metastasize to distant parts of the body and form new growths.

Over the past 15 years, the GFP gene has enabled scientists to watch a plethora of previously murky biological processes in action: how nerve cells develop in the brain, how insulin - producing beta cells form in the pancreas of an embryo, how proteins are transported within cells, and how cancer cells metastasize through the body.

The findings by a team of Massachusetts General Hospital (MGH) investigators, which will be published in the April 24 issue of Cell and are receiving advance online release, support the importance of epigenetics — processes controlling whether or not genes are expressed — in cancer pathology and identify molecular circuits that may be targeted by new therapeutic approaches.

The Salford team set out to assess the bioenergetics of cancer stem cells — the processes which allow the cells to live and thrive — with a view to disrupting their metabolism.

Metastasis, the process that allows some cancer cells to break off from their tumor of origin and take root in a different tissue, is the most common reason people die from cancer.

But Whitehead Institute researchers have found a mechanism underlying this resistance — a mechanism that naturally occurs in many diverse cancer types and that may expose vulnerabilities to drugs that spur the natural cell - death process.

The study also suggests that targeting the machinery that makes cells mobile, rather than targeting the tissue - clearing process — which has been tested in patients but has not been very effective — may be a better treatment strategy to stop cancers from spreading.

If these processes are defective, cells acquire the wrong identity — which can ultimately lead to cancer.

They chose drugs that cut off the cancer's ability to burn glucose and shut down protective processes that help cancer cells survive.

Tumours grow through a process of Darwinian evolution, where cancer cells develop an advantageous mutation that allows them to survive and multiply, producing a population of cells which can mutate further.

«With this discovery in our hands, we'd now like to try to find out which additional immune - cell properties cancer cells have and study how they affect the metastatic process,» says Dr Fuxe.

«The knowledge we have gained may be applicable for future intervention of this process to block the replication of viral pathogens and cancer cells.»

This process often goes wrong in cancer cells, resulting in chromosomal instability.

PARP inhibitors also cause PARP to become trapped in the cancer cells» DNA, a process that is greatly enhanced when a DMNT inhibitor is added, Dr. Rassool says.

«The protein PITPNC1 regulates a process whereby the cancer cells are secreting molecules, which cut through a network of proteins outside the cells, like scissors.

The team also showed that the cellular process macropinocytosis, which participates in cell scavenging nutrients and vesicles, contributes to exosomes uptake in cancer cells with mutant KRAS.

The researchers observed the effect of the synthetically produced molecule, JK - 31, on the growth and proliferation of a model human breast cancer cell line and found that it effectively blocked the protein cyclin - dependent kinase 1 (CDK1), which plays a key part in the process of the division of cancer cells, and therefore inhibited the proliferation of the cells.

The biopsy removes fluid from the cysts, which is then analyzed for cancer cells and other telltale signs of the disease, a process called cytology.

Although this is a normal process in embryogenesis to build new tissues, when it occurs in cancer cells it generates metastasis.

A team from the Spanish National Cancer Research Centre (CNIO) has determined for the first time the high - resolution structure of a complex (R2TP) involved in key processes for cell survival and in diseases such as cCancer Research Centre (CNIO) has determined for the first time the high - resolution structure of a complex (R2TP) involved in key processes for cell survival and in diseases such as cancercancer.

In a process called cellular reprogramming, researchers at Icahn School of Medicine at Mount Sinai have taken mature blood cells from patients with myelodysplastic syndrome (MDS) and reprogrammed them back into iPSCs to study the genetic origins of this rare blood cancer.

Cancer cell illustration courtesy of iStockphoto / Eraxion Chemotherapy treatment for cancer is a nasty prCancer cell illustration courtesy of iStockphoto / Eraxion Chemotherapy treatment for cancer is a nasty prcancer is a nasty process.

Óscar Llorca and his team have used this technique to learn about the structure and functioning of a complex system called R2TP, which is involved in various key processes for cell survival such as the activation of the kinases mTOR, ATR and ATM, proteins that are the target of various cancer drugs currently being developed.

Autophagy is a process by which cancer cells recycle essential building blocks to fuel further growth.

Center for Nuclear Receptors & Cell Signaling (CNRCS) Assistant Professor Daniel Frigo and his research team recently published a study investigating the processes through which androgen receptors affect prostate cancer progression.

The new research, which studied the immortalization process using genome - engineered cells in culture and also tracked skin cells as they progressed from a mole into a malignant melanoma, suggests that telomerase plays a more complex role in cancer.

The cancer cells, she explains, seem to hijack metabolic processes so they — not the healthy cells — get the energy they need to thrive.

«Two - step process leads to cell immortalization and cancer: Clearer view of the role telomere length and telomerase play in cell immortalization.»

To see whether cancer stem cell renewal involves a chain of events similar to that used by embryonic stem cells, and whether the process was affected by oxygen levels, Semenza and graduate student Chuanzhao Zhang focused their studies on two human breast cancer cell lines that responded to low oxygen by ramping up production of the protein ALKBH5, which removes methyl groups from mRNAs.

The IFNGR receptor is one of these membrane proteins and undertakes to activate genes involved in a huge variety of cell processes, including defence against pathogens and cancer.

Moffitt Cancer Center researchers have found that a protein called TBK1 plays an important role in the process of cell division, especially at a stage called mitosis.

Based on the pioneering work of Dr. Claire Lugassy and Dr. Raymond Barnhill at UCLA's Jonsson Comprehensive Cancer Center, a new study provides additional support for a process by which melanoma cells, a deadly form of skin cancer, can spread throughout the body by creeping like tiny spiders along the outside of blood vessels without ever entering the blood stream, and that this process is exacerbated by exposure to ultraviolet (UV) Cancer Center, a new study provides additional support for a process by which melanoma cells, a deadly form of skin cancer, can spread throughout the body by creeping like tiny spiders along the outside of blood vessels without ever entering the blood stream, and that this process is exacerbated by exposure to ultraviolet (UV) cancer, can spread throughout the body by creeping like tiny spiders along the outside of blood vessels without ever entering the blood stream, and that this process is exacerbated by exposure to ultraviolet (UV) light.

«But mounting evidence confirming angiotropism and EVMM has revolutionized the knowledge of how cancer spreads through the body to the point that other scientists have confirmed the process in other solid tumor cell types such as pancreatic cancer.»

Healthy cells without cancer markers would remain unaffected by this process.

Autophagy — an essential process cancer cells need to fuel their growth — is a key troublemaker spurring tumor growth.

In a bid to progress beyond the shotgun approach to fighting cancer — blasting malignant cells with toxic chemicals or radiation, which kills surrounding healthy cells in the process — researchers at the Harvard - MIT Division of Health Sciences and Technology (HST) are using nanotechnology to develop seek - and - destroy models to zero in on and dismantle tumors without damaging nearby normal tissue.