Sentences with phrase «cause mediator release»

Diet - induced inflammatory reactions cause mediator release (cytokines, histamine, leukotrienes, prostaglandins, etc.) from various white blood cells (lymphocytes, neutrophils, monocytes, eosinophils).

Not exact matches

Flavonoids help maintain a healthy collagen structure by (1) decreasing blood vessel permeability, thereby decreasing the influx of inflammatory mediators into areas of damage; (2) preventing free radical damage by means of their potent antioxidant properties; (3) inhibiting damage to collagen tissue caused by enzymes that break down collagen; (4) inhibiting the release of inflammatory chemicals; and (5) reinforcing the natural cross-linking of collagen fibers to make them stronger.

In fact, medicines that combat the inflammatory mediators released from stress, such as TNF and IL - 6, are currently in clinical trials for the treatment of cancer.29 Once again, we don't have proof that stress causes cancer, but big pharma is putting their money where their mouth is and targeting the same inflammatory mediators that stress causes in order to treat cancer.

The release of these mediators causes symptoms connected to food sensitivities.

These mediators are released from cells and cause inflammatory reactions like headaches, skin eruptions, sinus and eye irritation, digestive problems such and IBS and pain such as fibromyalgia.

When cells become senescent, they release inflammatory mediators, which can damage neighbouring cells and cause inflammation and disease.

It measures the inflammatory response and subtle changes in our immune cells (lymphocytes, neutrophils, monocytes, and eosinophils) as they release chemical mediators (a substance released from cells, e.g., cytokines, histamine, leukotrienes, prostaglandins, etc.), that regulate or cause physiologic consequences during an immune response to an antigen.

While there is little doubt that some of these systemic effects, such as systemic lipodystrophy, are caused by circulating pancreatic enzymes, recent data would suggest that other systemic sequelae are a consequence of the release of inflammatory mediators in response to pancreatic inflammation.

Release of active pancreatic enzymes and inflammatory mediators from the infl amed pancreas, such as Tumor Necrosis Factor - α (TNF - α) interleukin - 1 (IL - 1) and phospholipid platelet activating factor (PAF), amplifies the severity of pancreatic inflammation, and adversely affects the function of many organs (systemic inflammatory response), and cause derangement in fl uid, electrolyte and acid - base balance.

Injury to a joint — especially to the articular cartilage connecting the bones of a joint - causes release of a number of chemical substances, collectively called inflammatory mediators.

«It stimulates the release of endorphins, causes vasodilation, and promotes lymphatic flow, which brings more blood to the area and flushes out inflammatory mediators and swelling, and accelerates tissue repair by increasing the rate of cell division and activating cells needed for repair.

The irritant, such as the grass or infection inflames the lining of the stomach (gastric mucosa), causing the release of of mediators that cause the secretion of acids and a change in the way the stomach functions.