Further, at a low cytostatic concentration (2 nM), ActD
causes ribosomal stress leading to a decrease in HDM2 activity and thereby p53 stabilization and activation.
At low cytostatic concentrations, ActD
causes ribosomal stress, which decreases MDM2 activity, resulting in p53 stabilization and activation.
Not exact matches
So the
ribosomal fragments that we were finding explain a lot of things, explains among other things the observation that CCD seems to be
caused by everything.
Mutations in TFIIH
causing trichothiodystrophy are responsible for defects in
ribosomal RNA production and processing.
Small subunit
ribosomal RNA sequences link the
cause of proliferative gill disease in channel catfish to Henneguya n. sp. (Myxozoa: Myxosporea).