Sentences with phrase «cell memory makes»

This parallels prior observations that on the resistant BALB / c background, pre-existing CD8 T cell memory makes disease permissive (10).

Not exact matches

The company claims the open cell construction makes for a more durable, long - lasting memory foam, which is how they can afford a whopping 25 - year warranty.

Intel and Micron researchers have made the wires out of a new material as well as the wells that make up the memory cell where each bit of data is stored.

This damages some of the nerve cells in the brain, and affects my dad's memory, decision - making and personality.

The EverTrue Elite Mattress is made with open cell, pressure reduction, viscoelastic memory foam for your baby's comfort and safety.

Sleep soundly knowing your littlest one is resting on the EverTrue ™ Elite: a crib mattress made with open cell, pressure reduction, viscoelastic memory foam for your baby's comfort and safety.

It's an open - cell memory foam pillow (for enhanced airflow and less heat retention), which makes up for a lack of a cooling gel.

In animal models, the researchers found that using microRNA to disrupt cells in the brain's prefrontal cortex — a region associated with complex planning and decision - making — affected learning and memory later in life.

But he added, «We need to improve the memory responses of T and B cells to make longer - lasting vaccines.»

When injected into mice engineered with a tau mutation that makes the protein clump abnormally in brain cells, triggering memory and motor problems, the antibodies reduced the clumping and improved the animals» behavior.

Scientists have long experimented with organs - on - chips: tiny representations of human organs, such as lungs, hearts and intestines, made from cells embedded on plastic about the size of a computer memory stick.

In another group, the disabled gene made it difficult for fly brain cells to reinforce new connections that encode memories.

They aren't yet competition for Intel, but bioengineers have created a one - bit «memory» made of DNA that can record, erase and rewrite data within living cells.

Study casts doubt on whether adult brain's memory - forming region makes new cells.

T cells don't make antibodies, but certain T cells hold on to a memory of foreign molecules seen before.

And a bit surprisingly, it seems that the cells that make antibodies in the mucosa and the memory cells in the mucosa are not created at the same time,» says Mats Bemark.

It is the sparseness of the signals in the Kenyon cell neurons that makes studying memory formation in flies so promising to Turner and his team.

If so, perhaps you could make more resident memory cells for that particular organ to bolster the immune response.»

Artificial muscles made with carbon nanotubes or a shape memory alloy can be designed to act as fuel cells, thus alleviating a need for a remote battery power source.

At the heart of the «bit» of memory is essentially a fragment of DNA called a promoter, which enables the machinery of a cell to make a given protein.

The problem is, Wilson says, that a person has relatively few memory B cells — «on the order of one in thousands» — making the process of extracting antibodies from them a time - consuming task, because they first must be located.

Those plaques are dense deposits of protein and other cell material that attach themselves to and eventually kill neurons responsible for memory and decision making.

She and colleagues examined DNA from individual brain cells taken from three donated human brains and tested bulk samples from the hippocampus (an area important for learning and memory) and the frontal cortex (where most thinking and decision making is thought to happen).

Unraveling what happens in nerve cells during memory formation can help illuminate critical glitches that occur in short - term memory and lead to impaired decision - making in a range of neuropsychiatric and neurodegenerative disorders.

In one instance, he made the memory especially easy to read by engineering the cells to mutate an antibiotic resistance gene in response to light.

«The main achievement was the fact that we used the inhibition of the inhibitory neurons» to stimulate the memory patterns, says physicist Eshel Ben - Jacob, senior author of a paper on the findings published in the May issue of Physical Review E. «We probably made [the cell culture] trigger the collective mode of activity that... [is]... possible.»

In mice and humans alike, the cerebral cortex — the outermost layer of brain tissue associated with high - level functions such as memory and decision - making — starts out as a spherical sheet of tissue made up of only neural stem cells.

Most important, he adds, it only tells us that certain cells, in some regions at specific times, make memories — not how they contribute to doing so.

Taking the mice off the drug allowed cells that fire while they explored a new environment to make the proteins, effectively «tagging» the memory for that environment.

«Stimulating brain cells can make false memories.»

A study comparing children between 7 and 11 years of age who have moderate or severe obstructive sleep apnea to children the same age who slept normally, found significant reductions of gray matter — brain cells involved in movement, memory, emotions, speech, perception, decision making and self - control — in several regions of the brains of children with sleep apnea.

When the researchers modeled the effects in mice, they found it strengthened the connections between neurons that make learning possible — what is known as synaptic plasticity — by increasing the action of a cell receptor critical to forming memories.

The scientists implanted neural stem cells into the hippocampus, which is a brain region involved in making new memories and connecting them to emotions.

Cells that become cancerous may be making use of these early memories to power their ability to metastasize, or spread to distant sites in the body, newly published research reveals.

Health improvement (allowing to post - pone / escape the diseases and thus live, healthier / disease - free longer, but not above human MLSP of around 122 years; thus these therapies do not affect epigenetic aging whatsoever, they are degenerative aging problems not regular healthy aging problem (except OncoSENS - only when you Already Have Cancer - which cancer increases epigenetic aging, but cancer removal thus does not change anything / makes no difference about what happens in the other cells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to Mcells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to Mcells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to MCells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to Mcells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to MLSP).

Immunization with gp33 - loaded DCs led to the development of significant numbers of CD8 + T cells capable of making rapid IFN - γ responses to gp33 restimulation, consistent with a memory response, which was absent in control immunized mice (Fig. 1A).

While the exact contribution adult - born neurons make to overall learning and memory remains mysterious, Song notes that these results are promising for any future nerve stem cell therapy.

Cells that become cancerous may be making use of molecular memories imprinted during their earliest days to power their ability to metastasize, or spread to distant sites in the body.

He and his colleagues have made significant discoveries about how immune memory cells are created and how long they survive; understanding these mechanisms is crucial to the development of vaccines for HIV and other infectious agents.

This strategy takes advantage of the cells «somatic memory of origin» and novel reprogramming strategies to make these cells an effective and safe source of cells for the treatment of cartilage defects and osteoarthritis.

The idea that the activity of a place cell is predictive and can be modified by learning and the behavior of other animals may make it easier to conceptualize an all - encompassing framework that ties together the hippocampus» role in spatial navigation with its well - established role in learning and memory formation.

Describing the brain as a big circuit board in which each new experience creates a new circuit, Hopkins neuroscience professor Richard Huganir, Ph.D. says that he and his team found that during emotional peaks, the hormone norepinephrine dramatically sensitizes synapses - the site where nerve cells make an electro - chemical connection - to enhance the sculpting of a memory into the big board.

The results, which are published in the journal Cell, add to the understanding of how the brain's frontal lobes work and control behaviour.The frontal cortex of the brain plays a crucial part in cognitive functions, including everyday mental processes such as attention, memory, learning, decision - making and problem - solving.

At eight weeks of development, either a testosterone surge kills off cells in the communication center of the brain, and grows more cells in the sex and aggression areas (making it a «male brain,») or a surge of estrogen promotes brain growth in parts handling communication, feelings, emotional memory and anger - suppression (making it a «female brain»).

One of Frankland's own studies in mice, for example, found that as new brain cells are formed in the hippocampus — a region of the brain associated with learning new things — those new connections overwrite old memories and make them harder to access.

EPA protects against nerve - cell death and promotes nerve - tissue growth in the hippocampus, a part of the brain responsible for happiness, decision making, learning, and memory.

At some level, the cells hold on to a memory of what they used to be — when made from pancreatic beta cells, which are responsible for producing insulin, these pluripotent cells turn out to be better than their embryonic counterparts for generating insulin - producing cells.

In this episode Rhonda talks about how heat stress from using the sauna makes the body more resilient to the stresses of aging, possible reasons why one study associated sauna use with up to a 40 % lower all - cause mortality as well as a 50 % lower cardiovascular disease related mortality, how it enhances athletic endurance, staves off muscle atrophy, improves regrowth of muscle after disuse, and some of the profound effects on the brain, including the growth of new brain cells, improvement in focus, learning, and memory, and even potentially ameliorating depression and anxiety.

The point is, not only do your muscles carry memories so do your cells on every level, including the patterns you make in your brain.

Studies show that nitric oxide signaling and the blood flow increases it stimulates play a central role in nerve cell maintenance, growth and repair.,, Most pertinent to anyone looking to enhance their aptitude for learning, nitric oxide - induced blood flow also makes forming new memories physically possible as it plays a key role in what neurologists call long - term potentiation, a process required for assembling and reinforcing new synaptic connections throughout the entire cerebral cortex, striatum, and hippocampus.