Sentences with phrase «cell treatments because»

Luan said the chemo drug Brandon took was much more affordable than his stem cell treatments because it was covered after the family met their $ 5,000 deductible.

Not exact matches

Lola's Secret Beauty Blog can't stop praising The Organic Pharmacy Jasmine Night Conditioner because this amazing nighttime treatment eliminates dead skin cells and it hydrates the skin!

Usually, no treatment is needed — avoiding the triggers is normally enough to prevent a crisis, or removing them is all that's necessary to eliminate symptoms, because the body then starts to create new red blood cells naturally.

The robots could be useful in cancer treatments, because they can target specific cells.

The cells are also lost naturally because of the aging process, especially in the face, which leads to permanent, deep wrinkles, something anti-aging treatments can't fix in a cosmetically satisfactory way.

«This is important because some cancer cells are more resistant to one type of treatment than another.

Because the CAR - T cells do not eradicate all cancer cells, the researchers think the immune therapy will need to be combined with other treatments.

Myc inhibitors are thought to be promising treatments for multiple myeloma because they shut down a gene required for cell proliferation.

CAR - T cell therapy is particularly exciting because it works well in people whose cancers haven't responded to other available treatments, says Renier Brentjens, an oncologist at Memorial Sloan Kettering Cancer Center in New York City.

The development of targeted therapies has significantly improved the survival of melanoma patients over the last decade; however, patients often relapse because many therapies do not kill all of the tumor cells, and the remaining cells adapt to treatment and become resistant.

Immune therapy for ovarian, breast and colorectal cancer — treatments that encourage the immune system to attack cancer cells as the foreign invaders they are — has so far had limited success, primarily because the immune system often can't destroy the cancer cells.

Another problem with Kobinger's approach is that patients will require repeated treatments, because the added gene ends up in surface cells that die off after a few months.

Because the different cancer cells within a tumorous tissue also develop different modes of defense against therapeutic measures, the treatment of patients is extraordinarily difficult.

«The iPS cells may also help us identify treatments for more common diseases, such as atherosclerosis and vascular calcification, because the same bone morphogenetic protein pathways are involved in these medical conditions.»

The treatment stopped the degeneration but the trial was halted in 2015 because genetic mutations were detected in another batch of iPS cells intended for another patient.

Metastasis, or cancer spread by the formation of tumors at new sites, is generally what makes cancers deadly because surgery and other treatments are unlikely to find and destroy every cancer cell.

Current chemotherapy treatments cure the disease in fewer than 10 % of patients who have a FLT3 mutation — partly because toxic chemotherapy drugs can be used for only a few days at a time, not killing as many cancer cells as they might.

Because of this lack of receptors, common cancer drugs can't «find» the cells, and doctors must treat the cancer with extremely aggressive and highly toxic treatment strategies,» said Salman Hyder, the Zalk Endowed Professor in Tumor Angiogenesis and professor of biomedical sciences in the College of Veterinary Medicine and the Dalton Cardiovascular Research Center.

Another is that the transplanted bits of tumor act nothing like cancers in actual human brains, Fine and colleagues reported in 2006: Real - life glioblastomas grow and spread and resist treatment because they contain what are called tumor stem cells, but tumor stem cells don't grow well in the lab, so they don't get transplanted into those mouse brains.

«This is why some cancers can be so difficult to treat with chemotherapy, because the cells can be in different states — some sensitive to treatment and some resistant to treatment, all in the same tumor,» says Sandro Santagata, a former visiting scientist in the lab of Whitehead Member Susan Lindquist.

Treatment is difficult because pancreatic tumours are protected by an armour of connective tissue, blood vessels and immune cells, known collectively as the stroma.

Cancer stem cells, a type of self - renewing cell found in tumors, are of particular interest because they are the main cell type responsible for tumor progression and for resistance to chemotherapy and radiotherapy, and therefore a major cause of tumor recurrence after treatment.

Because biologists like Daley are convinced that embryonic stem cells — the most generic, versatile type — may not only lead to dramatically different new treatments but can also uniquely illuminate the origins of disease in a way adult stem cells never will.

Because p53 is often mutated or lost in cancer cells, a number of compounds have been designed to increase its levels as a possible anti-cancer treatment.

Because AML is difficult to treat, standard treatment commonly involves multiple chemotherapy medicines that are given at a higher dose in order to kill the cancer cells.

A new gene therapy treatment has restored some sight in a handful of blind patients suffering from Leber's congenital amaurosis, a syndrome in which, because of a broken or missing gene called RPE65, retinal photoreceptor cells malfunction and eventually die.

Geschwind, a professor in the Russell H. Morgan Department of Radiology and Radiological Science at the Johns Hopkins University School of Medicine and its Kimmel Cancer Center, and others at Johns Hopkins have been studying the experimental drug as a cancer treatment for over a decade because of its ability to block a key metabolic pathway of cancer cells.

Previous attempts to do the same in monkeys, however, have failed — a disappointment because monkeys are more similar than mice to humans, and thus likely a better harbinger of how stem cell treatments will fare in people.

This has the potential to be a less toxic and more effective treatment than more standard approaches because it can specifically target cancer cells.

Those two papers were groundbreaking because they put forward a method for generating stem cells far simpler than any previously reported, a development that could advance regenerative medicine, in which scientists try to grow replacement tissues as a treatment for diseases and injuries.

Wang explained that, because ECMs are composed of collagen, elastin, carbohydrates and signaling molecules and have no cell surface markers, DNA or RNA from the donor, the recipient is less likely to reject the treatment.

«Cells respond differently to different ways of treatment because of the mutations of their genes.

«This is because the stress led to poor function against the cancer by T - cells, which are very important in the immune system's control and surveillance of tumours and are a major target in many immunotherapy treatments.»

In her presentation, Ann Tsukamoto, StemCells» vice president for research, said the company chose to test its neural stem cell approach on PMD because there is currently no treatment for the condition and a diagnosis can be confirmed by genetic testing and magnetic resonance imaging.

Treatment of brain tumors is particularly challenging because regulatory T - cells accumulate in brain tumors and suppress an immune attack.

Drugs can not reach the tissue at lethal doses because its blood supply is so poor, while radiation treatments depend on oxygen to trigger cell death.

Treatment has focused on electronic devices like hearing aids or cochlear implants because once lost, human auditory hair cells do not grow back.

Treatment that targets the DNA in HIV - infected cells has been challenging because the persistent, incurable human immunodeficiency virus is able to insert its own DNA into the DNA of any infected cell while disabling that cell's ability to die to save other cells from a viral invasion.

Because the treatment uses the patient's immune cells as a sort of T - cell training force, it is an immunotherapy.

Why it matters: Because the secretome is important for tissue function and equilibrium as well as cell communication, proliferation and organization, studying these proteins may lead to new treatments and biomarker discovery.

Because scientists could use the new stem cells to make any tissue in a test tube, they might have an easier time studying certain diseases and treatments.

\ n \ nI am very concerned because my Grandson has Spinal Muscular Atrophy and he needs the embryonic stem cell treatment to survive, before he dies.

A new breast cancer clinical trial is testing the idea a major reason why breast cancer returns after treatment and spreads to other parts of the body is because current chemotherapy and radiation treatments do not kill the cancer stem cells.

In some countries, this phenomenon traditionally has been called stem cell tourism because it involved travel to another country with less stringent regulations to obtain treatment.

(This is important because the more parts of a cell a treatment hits, the harder it is for cancer cells to mutate to avoid it.)

Because treatment with rapamycin also led to decreased AKT phosphorylation in ALK + ALCL cells in our in vitro study, it is tempting to speculate that an effector protein downstream of mTOR - raptor may contribute directly or indirectly to AKT activation.

Willet, Mills, and their colleagues believe the discovery that cells in different organs go through the same process to become proliferative could lead to new potential targets for cancer treatment because the factors that initiate tumours could be the same in multiple organs.

Because the drug's mode of action was known and patients» response to treatment could be precisely monitored, Michor and colleagues used modeling methods and data from patients in a large clinical trial to identify a group of copiously self - renewing stem cells, which persist within the tumor, resist the drug, and sustain the cancer.

Because they stimulate the immune system rather than introducing an agent or process that can kill healthy cells along with the malignant ones, these therapies — which include commonly prescribed treatments such as trastuzumab (Herceptin), pembrolizumab (Keytruda) and nivolumab (Opdivo)-- have been noteworthy not only for their effectiveness but for their tolerability.

Because organoids are easy and relatively inexpensive to grow and can be created from a particular person's cells, they might also be extremely useful in personalized medicine, helping tailor a treatment the way some cancer treatments can be targeted to the genetic makeup of a tumor.