After transmission, Anaplasma phagocytophilum bacteria invade white blood
cells called neutrophils.
The clinical signs may include anemia, thrombocytopenia (relatively few platelets in blood) and neutropenia (presence of abnormally low number of a type of white blood
cells called neutrophils).
Analysis of sputum samples revealed that those who had eaten the burger meal had an increased number of immune
cells called neutrophils in their airways.
The saliva triggers an immune response in which white blood
cells called neutrophils and myeloid cells rush to the site.
The round white cells are specialized immune
cells called neutrophils.
Normally, immune
cells called neutrophils kill microbes by gobbling them up or releasing toxic chemicals.
Cells called neutrophils, which are considered the immune system's «first line of defense» because they react strongly to foreign invaders that enter the skin through a cut or other injury, were also surprisingly important.
IL - 17 attracted other immune
cells called neutrophils — the most abundant type of white blood cell — to the conjunctiva and induced the release of anti-microbial proteins into the tears.
Research published on August 7th in PLOS Pathogens comparing the two pathogens reveals how S. Typhi avoids recognition and elimination by patrolling immune
cells called neutrophils, allowing it to disseminate throughout the patient's body.
Further experiments suggested that the production of high levels of cell wall - associated GAG makes A. fumigatus more resistant than A. nidulans to neutrophil killing by neutrophil extracellular traps, or NETs (NETs are are networks of extracellular fibers, composed of DNA and proteins secreted by immune
cells called neutrophils, that bind and trap various pathogens).
This process helps to form both a bacteria - killing NET — which is comprised of infection - combatting white blood
cells called neutrophils — and the fluffy, scattered ball that comprises a blood clot.
In their report published in Science Immunology they describe how expression of a specific molecule — complement C5a — is required to cause the immune
cells called neutrophils to adhere to joint surfaces and migrate into the joint, a process known to set off the inflammatory cascade.
Patients who develop this specific fungal infection are overwhelmingly adults who are immunocompromised, Kumar explains, including those with diabetes, transplant recipients, patients with cancer and those who have abnormally low concentrations of immune
cells called neutrophils in their blood.
The enzyme is produced by white blood
cells called neutrophils, which play an important role in inflammatory and immune responses and may be a key contributor to the development of muscular dystrophy.
In the 2012 trial, for example, eight people had serious side effects, such as a sharp decrease in immune
cells called neutrophils, and 14 people had comparatively mild side effects like nausea.
Not exact matches
In both diabetic models, high blood sugar levels caused a significant increase in the blood counts of certain immune
cells called monocytes and
neutrophils.
The investigators also discovered that
neutrophils secrete a molecule
called S100A8 / S100A9 that interacts with a receptor named RAGE (the Receptor for Advanced Glycation Endproducts) on bone marrow
cells.
Once the inflammatory process has been initiated,
neutrophils within the joint space release interleukin - 1, which induces
cells lining the joint space to produce chemoattractants
called chemokines that further facilitate the movement of
neutrophils into the joint space.
In another experiment, the research team transferred infection - combatting white blood
cells —
called neutrophils — from normal mice to genetically mutated mice.
Current thinking regarding type III hypersensitivity is that immune
cells within tissues sense the presence of these immune complexes (ICs) through specific receptor molecules and release inflammatory factors
called cytokines that activate the endothelial
cells lining adjacent blood vessels to promote the recruitment of
neutrophils.
In a new report appearing in the November 2016 issue of the Journal of Leukocyte Biology, scientists show that nicotine activates certain white blood
cells,
called neutrophils, which in turn release molecules that lead to increased inflammation.
Immune
cells known as
neutrophils can eject their DNA, forming a mesh of sticky strands
called a
neutrophil extracellular trap (NET) that captures and kills microbes.
The team went on to show that Haemophilus somehow recruited host immune
cells,
called neutrophils, to the site of colonization.
White blood
cells,
called neutrophils, are a primary player in the immune system, fending off infection from foreign bacteria and viruses, anything from colds and strep throat to ear and sinus infections.
The immune system, by sending in special
cells called macrophages and
neutrophils, plays a big role in cleaning up this damage (17).
As pathologists began to examine these lesions they noted that they were composed of inflammatory
cells called eosinophils,
neutrophils and scar tissue.
For example, the presence of increased numbers of immature white blood
cells,
called band
neutrophils or bands for short, indicate a serious infection.
As pathologists began to examine these lesions they noticed that they all contained abnormal numbers of inflammatory
cells called eosinophils, accompanied by
neutrophils and scar tissue.
You have these little soldier
cells in your body
called neutrophils, and when they spend too long going around looking for something to do, they become grumpy and pro-inflammatory.