parabens: Parabens are
chemicals with estrogen - like properties, and estrogen is one of the hormones involved in the development of breast cancer.
One study compiled evidence that suggests approximately 455 plastics commercially available to industry, leach
chemicals with estrogen activity (EA).
Not exact matches
BPA is a
chemical with estrogenic activity (EA), meaning it mimics the
estrogen hormone.
The findings come as the U.S. Environmental Protection Agency faces opposition from the pesticide industry after expanding its Endocrine Disruptor Screening Program, which requires testing of about 200
chemicals found in food and drinking water to see if they interfere
with estrogen, androgens or thyroid hormones.
«The brain along
with the reproductive system and every other cell in your body is exquisitely sensitive to exceedingly small changes in
estrogen and other sex hormones, and the fact that the environment is full of
chemicals that can activate
estrogen receptors means this phenomenally sensitive system is being perturbed constantly by environmental factors.»
In 1952, renowned mathematician Alan Turing was convicted of gross indecency in the U.K. after admitting to a sexual relationship
with a man, and was forced to submit to
estrogen injections as a form of «experimental
chemical castration.»
Women in Greece diagnosed
with polycystic ovary syndrome (PCOS)-- which causes irregular menstrual periods, infertility, weight gain and excessive hair growth — were more likely to have higher blood levels of the
estrogen - mimicking
chemical bisphenol A than women without the disease, according to a study published last year.
Susan Amara, USA - «Regulation of transporter function and trafficking by amphetamines, Structure - function relationships in excitatory amino acid transporters (EAATs), Modulation of dopamine transporters (DAT) by GPCRs, Genetics and functional analyses of human trace amine receptors» Tom I. Bonner, USA (Past Core Member)- Genomics, G protein coupled receptors Michel Bouvier, Canada - Molecular Pharmacology of G protein - Coupled Receptors; Molecular mechanisms controlling the selectivity and efficacy of GPCR signalling Thomas Burris, USA - Nuclear Receptor Pharmacology and Drug Discovery William A. Catterall, USA (Past Core Member)- The Molecular Basis of Electrical Excitability Steven Charlton, UK - Molecular Pharmacology and Drug Discovery Moses Chao, USA - Mechanisms of Neurotophin Receptor Signaling Mark Coles, UK - Cellular differentiation, human embryonic stem cells, stromal cells, haematopoietic stem cells, organogenesis, lymphoid microenvironments, develomental immunology Steven L. Colletti, USA Graham L Collingridge, UK Philippe Delerive, France - Metabolic Research (diabetes, obesity, non-alcoholic fatty liver, cardio - vascular diseases, nuclear hormone receptor, GPCRs, kinases) Sir Colin T. Dollery, UK (Founder and Past Core Member) Richard M. Eglen, UK Stephen M. Foord, UK David Gloriam, Denmark - GPCRs, databases, computational drug design, orphan recetpors Gillian Gray, UK Debbie Hay, New Zealand - G protein - coupled receptors, peptide receptors, CGRP, Amylin, Adrenomedullin, Migraine, Diabetes / obesity Allyn C. Howlett, USA Franz Hofmann, Germany - Voltage dependent calcium channels and the positive inotropic effect of beta adrenergic stimulation; cardiovascular function of cGMP protein kinase Yu Huang, Hong Kong - Endothelial and Metabolic Dysfunction, and Novel Biomarkers in Diabetes, Hypertension, Dyslipidemia and
Estrogen Deficiency, Endothelium - derived Contracting Factors in the Regulation of Vascular Tone, Adipose Tissue Regulation of Vascular Function in Obesity, Diabetes and Hypertension, Pharmacological Characterization of New Anti-diabetic and Anti-hypertensive Drugs, Hypotensive and antioxidant Actions of Biologically Active Components of Traditional Chinese Herbs and Natural Plants including Polypehnols and Ginsenosides Adriaan P. IJzerman, The Netherlands - G protein - coupled receptors; allosteric modulation; binding kinetics Michael F Jarvis, USA - Purines and Purinergic Receptors and Voltage-gated ion channel (sodium and calcium) pharmacology Pain mechanisms Research Reproducibility Bong - Kiun Kaang, Korea - G protein - coupled receptors; Glutamate receptors; Neuropsychiatric disorders Eamonn Kelly, Prof, UK - Molecular Pharmacology of G protein - coupled receptors, in particular opioid receptors, regulation of GPCRs by kinasis and arrestins Terry Kenakin, USA - Drug receptor pharmacodynamics, receptor theory Janos Kiss, Hungary - Neurodegenerative disorders, Alzheimer's disease Stefan Knapp, Germany - Rational design of highly selective inhibitors (so call chemical probes) targeting protein kinases as well as protein interaction inhibitors of the bromodomain family Andrew Knight, UK Chris Langmead, Australia - Drug discovery, GPCRs, neuroscience and analytical pharmacology Vincent Laudet, France (Past Core Member)- Evolution of the Nuclear Receptor / Ligand couple Margaret R. MacLean, UK - Serotonin, endothelin, estrogen, microRNAs and pulmonary hyperten Neil Marrion, UK - Calcium - activated potassium channels, neuronal excitability Fiona Marshall, UK - GPCR molecular pharmacology, structure and drug discovery Alistair Mathie, UK - Ion channel structure, function and regulation, pain and the nervous system Ian McGrath, UK - Adrenoceptors; autonomic transmission; vascular pharmacology Graeme Milligan, UK - Structure, function and regulation of G protein - coupled receptors Richard Neubig, USA (Past Core Member)- G protein signaling; academic drug discovery Stefan Offermanns, Germany - G protein - coupled receptors, vascular / metabolic signaling Richard Olsen, USA - Structure and function of GABA - A receptors; mode of action of GABAergic drugs including general anesthetics and ethanol Jean - Philippe Pin, France (Past Core Member)- GPCR - mGLuR - GABAB - structure function relationship - pharmacology - biophysics Helgi Schiöth, Sweden David Searls, USA - Bioinformatics Graeme Semple, USA - GPCR Medicinal Chemistry Patrick M. Sexton, Australia - G protein - coupled receptors Roland Staal, USA - Microglia and neuroinflammation in neuropathic pain and neurological disorders Bart Staels, France - Nuclear receptor signaling in metabolic and cardiovascular diseases Katerina Tiligada, Greece - Immunopharmacology, histamine, histamine receptors, hypersensitivity, drug allergy, inflammation Georg Terstappen, Germany - Drug discovery for neurodegenerative diseases with a focus on AD Mary Vore, USA - Activity and regulation of expression and function of the ATP - binding cassette (ABC) tran
Estrogen Deficiency, Endothelium - derived Contracting Factors in the Regulation of Vascular Tone, Adipose Tissue Regulation of Vascular Function in Obesity, Diabetes and Hypertension, Pharmacological Characterization of New Anti-diabetic and Anti-hypertensive Drugs, Hypotensive and antioxidant Actions of Biologically Active Components of Traditional Chinese Herbs and Natural Plants including Polypehnols and Ginsenosides Adriaan P. IJzerman, The Netherlands - G protein - coupled receptors; allosteric modulation; binding kinetics Michael F Jarvis, USA - Purines and Purinergic Receptors and Voltage-gated ion channel (sodium and calcium) pharmacology Pain mechanisms Research Reproducibility Bong - Kiun Kaang, Korea - G protein - coupled receptors; Glutamate receptors; Neuropsychiatric disorders Eamonn Kelly, Prof, UK - Molecular Pharmacology of G protein - coupled receptors, in particular opioid receptors, regulation of GPCRs by kinasis and arrestins Terry Kenakin, USA - Drug receptor pharmacodynamics, receptor theory Janos Kiss, Hungary - Neurodegenerative disorders, Alzheimer's disease Stefan Knapp, Germany - Rational design of highly selective inhibitors (so call
chemical probes) targeting protein kinases as well as protein interaction inhibitors of the bromodomain family Andrew Knight, UK Chris Langmead, Australia - Drug discovery, GPCRs, neuroscience and analytical pharmacology Vincent Laudet, France (Past Core Member)- Evolution of the Nuclear Receptor / Ligand couple Margaret R. MacLean, UK - Serotonin, endothelin,
estrogen, microRNAs and pulmonary hyperten Neil Marrion, UK - Calcium - activated potassium channels, neuronal excitability Fiona Marshall, UK - GPCR molecular pharmacology, structure and drug discovery Alistair Mathie, UK - Ion channel structure, function and regulation, pain and the nervous system Ian McGrath, UK - Adrenoceptors; autonomic transmission; vascular pharmacology Graeme Milligan, UK - Structure, function and regulation of G protein - coupled receptors Richard Neubig, USA (Past Core Member)- G protein signaling; academic drug discovery Stefan Offermanns, Germany - G protein - coupled receptors, vascular / metabolic signaling Richard Olsen, USA - Structure and function of GABA - A receptors; mode of action of GABAergic drugs including general anesthetics and ethanol Jean - Philippe Pin, France (Past Core Member)- GPCR - mGLuR - GABAB - structure function relationship - pharmacology - biophysics Helgi Schiöth, Sweden David Searls, USA - Bioinformatics Graeme Semple, USA - GPCR Medicinal Chemistry Patrick M. Sexton, Australia - G protein - coupled receptors Roland Staal, USA - Microglia and neuroinflammation in neuropathic pain and neurological disorders Bart Staels, France - Nuclear receptor signaling in metabolic and cardiovascular diseases Katerina Tiligada, Greece - Immunopharmacology, histamine, histamine receptors, hypersensitivity, drug allergy, inflammation Georg Terstappen, Germany - Drug discovery for neurodegenerative diseases with a focus on AD Mary Vore, USA - Activity and regulation of expression and function of the ATP - binding cassette (ABC) tran
estrogen, microRNAs and pulmonary hyperten Neil Marrion, UK - Calcium - activated potassium channels, neuronal excitability Fiona Marshall, UK - GPCR molecular pharmacology, structure and drug discovery Alistair Mathie, UK - Ion channel structure, function and regulation, pain and the nervous system Ian McGrath, UK - Adrenoceptors; autonomic transmission; vascular pharmacology Graeme Milligan, UK - Structure, function and regulation of G protein - coupled receptors Richard Neubig, USA (Past Core Member)- G protein signaling; academic drug discovery Stefan Offermanns, Germany - G protein - coupled receptors, vascular / metabolic signaling Richard Olsen, USA - Structure and function of GABA - A receptors; mode of action of GABAergic drugs including general anesthetics and ethanol Jean - Philippe Pin, France (Past Core Member)- GPCR - mGLuR - GABAB - structure function relationship - pharmacology - biophysics Helgi Schiöth, Sweden David Searls, USA - Bioinformatics Graeme Semple, USA - GPCR Medicinal Chemistry Patrick M. Sexton, Australia - G protein - coupled receptors Roland Staal, USA - Microglia and neuroinflammation in neuropathic pain and neurological disorders Bart Staels, France - Nuclear receptor signaling in metabolic and cardiovascular diseases Katerina Tiligada, Greece - Immunopharmacology, histamine, histamine receptors, hypersensitivity, drug allergy, inflammation Georg Terstappen, Germany - Drug discovery for neurodegenerative diseases
with a focus on AD Mary Vore, USA - Activity and regulation of expression and function of the ATP - binding cassette (ABC) transporters
In this report the American Council on Science and Health (ACSH) explores the endocrine disrupter hypothesis, which asserts that certain (primarily man - made)
chemicals act as, or interfere
with, human hormones (specifically
estrogens) in the body and thus cause a range of defects and diseases related to the endocrine system.
A general theme
with these tips is the end result of excreting excess
estrogen and
chemicals out of our bodies.
Harsh
chemicals in anti-wrinkle creams can wreak havoc on your hormonal levels, and research shows phthalates and parabens in many beauty products can mess
with your
estrogen levels.
This is why you need to know that a lot of reasons can contribute to a low testosterone level and these are things as simple as diets that won't provide your body
with enough material to create testosterone, lack of physical ability or as complex as exposure to
chemical estrogens or modern food lacking nutrients.
5MTHF, along
with several other nutrients, is also used to create and process neurotransmitters (messengers in the nervous system like serotonin, epinephrine, norepinephrine, and dopamine); create immune cells and process hormones (such as
estrogen); as well as to produce energy and detoxify
chemicals.
The
chemicals in cigarette smoke have been shown to interfere
with the ability of cells in the ovary to make
estrogen.
This reasoning is part of the two - pronged approach to treating
estrogen overload: support the heatlhy processing and elimination of
estrogen with food and supplements and avoid exposure to toxic
chemical that can mimic
estrogen's action in the body.
They are also packed
with lignans, a
chemical compounds that act like
estrogens in the body.
These are
chemical compounds that are structurally similar to
estrogen, so when they enter the body, they behave
with estrogenic effect on the reproductive system function.
The liver, kidneys and other internal organs can become burdened
with estrogen,
chemicals, and toxic metals.
active ingredient in antiperspirants, all natural deodorant, aluminum based compounds and parabans, baby was nursing, benefits of coconut oil,
chemicals entering her body, coconut oil is great to cook
with, coconut oil to cook, connection between underam antiperspirants or deodorants and breast cancer, cooking
with coconut oil is much healthier than olive oil, deodorant breastfeeding, developement of breast cancer,
estrogen has ability to promote growth of breast cancer cells, homemade deodorant, ingredients in deodorants and antiperspirants, Mommy Footprint fan page, sticking on stainless steel pans, stop using deodorant, tandem nursing twins, traditional deodorant, women and men sweat and react to deodorants differently
They are effective at removing a wide range of different
chemicals including
estrogens, but don't fare so well
with dissolved minerals like fluoride.
Testosterone,
estrogen, dopamine, serotonin, adrenaline, oxytocin and vasopressin... These brain
chemicals and hormones have everything to do
with... Read More
If you've destroyed your gut bacteria
with: SUGAR (the kind in Girl Scout Cookies), Processed Refined Foods (like a Girl Scout Cookie), Antibiotics, Cortisol / Stress,
chemicals in water (fluoride, chlorine, etc), birth control pills, anti-inflammatories, acid suppressors, or Splenda, you've likely impaired your bodies ability to break down and get rid of «old
estrogen»; back into the system the
estrogen goes instead of being pooped or peed out of you.
See: Interaction of estrogenic
chemicals and phytoestrogens
with estrogen receptor β Soy isoflavone genistein has about 4 % of 17β - estradiol's affinity for
estrogen receptor α, responsible for
estrogen's feminizing effects, and 87 % of its affinity for ERβ, which acts as a tumor suppressor.
The unnamed products that were used to conduct the study contained lavender and tea tree oils, but they were diluted
with a
chemical that is a known
estrogen mimicker (dimethyl sulfoxide.)
Estrogen is a hormone, which is a
chemical messengers that your body uses to communicate
with cells.
Whenever your liver and large intestine are congested
with sugar, hormonal waste,
chemicals, and toxins, it can't properly break down and remove
estrogen from your body anymore.
Estrogen is intimately connected
with serotonin, a brain
chemical that is converted to melatonin, a sleep hormone.
Many of these
chemicals are Xenoestrogens, which means they mimic
estrogen in the body and mess
with the proper functioning of our endocrine system.
The
chemical binds
with estrogen receptors on pancreatic cells to boost insulin secretion.
Animal studies have linked the
chemical with changes in testosterone,
estrogen, and thyroid hormones.
For women, the hazardous
chemicals in cigarettes are poisonous to ovaries by decreasing blood flow, interfering
with estrogen production, and causing genetic abnormalities to eggs.
Scientists are still looking into why this is, but one promising line of research has found that some people have high levels of a certain type of intestinal bacteria that interacts
with chemicals in soy to produce an
estrogen - like compound called equol.
In our
chemical filled world
with estrogen mimickers everywhere, this is not good news.
According to Discovery News, lab tests on more than twenty top - brand baby bottles along
with more than four hundred fifty plastic food and beverage packages, found that virtually all leached
chemicals —
chemicals that act like the hormone
estrogen — even though many were free of BPA.
Of those 300
chemical reactions, several of them have to do
with the proper utilization and the proper metabolism of
estrogen.
A study published in Toxicology and Applied Pharmacology showed that the
chemicals have been linked to endocrine disruption; parabens compete
with estrogen for sites to attach throughout the body, thereby potentially affecting hormonal balance.
These
chemicals have endocrine disruption effects
with clear
estrogen - like actions on the developing baby's brain and reproductive organs.
According to the FDA, 17 % of the American diet comes out of cans, and many of those have an epoxy liner made
with Bisphenol A, a
chemical which can mimic human
estrogen and which is linked to breast cancer and early puberty in women.