Sentences with phrase «child adiposity»

The relations of child adiposity with parent - to - child and parent - to - parent hostility.

Objectives: This study sought to identify factors associated with maternal feeding behaviors and to test the hypothesis that more maternal prompts to eat, more assertive prompts, and more intrusiveness are associated with greater child adiposity.

The association between maternal prompting to eat and child adiposity has been inconsistent.

In contrast, when prompting was conceptualized as assertive (3, 4), studies usually found a positive association with child adiposity.

Although maternal feeding behaviors and child adiposity were concurrently associated at ages 15, 24, and 36 mo, maternal feeding behaviors at age 15 mo were not associated with child adiposity at age 36 mo..

Background: Few studies have examined observed maternal feeding behaviors and their potential association with child adiposity.

The adjusted models predicting child adiposity z scores across ages 15, 24, and 36 mo are shown in Table 3.

Adjusted regression analyses evaluated predictors of prompts, the percentage of assertive prompts, and intrusiveness and the relation of each of these factors with child adiposity (weight - for - length z score at 15 mo and BMI z score at 24 and 36 mo) after control for the child's race - ethnicity and sex, family income - to - needs ratio, and maternal education, weight status, and depressive symptoms.

Julie C Lumeng, Tina N Ozbeki, Danielle P Appugliese, Niko Kaciroti, Robert F Corwyn, Robert H Bradley; Observed assertive and intrusive maternal feeding behaviors increase child adiposity, The American Journal of Clinical Nutrition, Volume 95, Issue 3, 1 March 2012, Pages 640 — 647, https://doi.org/10.3945/ajcn.111.024851

Future work should focus on maternal motivations for assertive and intrusive feeding styles and mechanisms through which these feeding styles might increase child adiposity.

Conclusions: Assertive prompting and an intrusive style had small but significant associations with greater child adiposity.

The findings also indicate that the way in which prompting or «pressure to eat» is defined is an important determinant of the relation with child adiposity.

This suggests that maternal feeding behaviors affect child adiposity contemporaneously, but do not have long - lasting effects on children's adiposity 2 y later — a finding consistent with some studies in the literature (11, 49 — 52) but not others (9, 15, 53 — 57).

The data associating maternal feeding behaviors with child weight status (2), however, have been inconsistent, particularly for feeding behaviors related to prompting or pressuring the child to eat, which have been positively (3, 4), negatively (5 — 15), and not (11, 16 — 21) associated with markers of child adiposity.

Children were videotaped eating a standardized snack with their mothers at 3 time points across toddlerhood to address 2 objectives: 1) to identify maternal and child characteristics that are associated with observed maternal feeding behaviors, and 2) to test the hypothesis that more prompts, a greater percentage of prompts being assertive, and more intrusiveness are associated with greater child adiposity.

Not exact matches

Breakfast consumption and adiposity among children and adolescents: an updated review of the literature.

Moreover, higher levels of physical activity were related to better fitness levels also in children with higher levels of body adiposity.

A recent Finnish study shows that high body adiposity, low physical activity, and particularly their combination are related to poorer physical fitness among 6 - 8 year old children.

Longitudinal Associations Between Ambient Air Pollution With Insulin Sensitivity, b - Cell Function, and Adiposity in Los Angeles Latino Children.

The study showed that the more children spent time doing physical activities, the lower their total body and central body adiposity were.

The study investigated the associations of physical activity and sedentary time with body adiposity and cardiorespiratory fitness in 410 Finnish 6 -8-year-old children.

Methylation varied greatly at particular CpGs: for 31 CpGs with median methylation ≥ 5 % and a 5 — 95 % range ≥ 10 % we related methylation status to maternal pregnancy diet and to child's adiposity at age 9 years.

Our findings suggest that blunted leptin release in relation to increasing levels of adiposity can contribute to the obesity risk among maltreated children.

Prospective studies such as «Fruit juice intake predicts increased adiposity gain in children from low - income families» show a close correlation between intake of juice and childhood obesity over the 48 month follow up period.

Epigenetic gene promoter methylation at birth is associated with child's later adiposity.

In addition, a recent rise in the incidence of abdominal adiposity, the unhealthiest form of excess body fat, has been observed in both adults and children, indicating a direct link to insulin - resistance, the body's natural propensity to convert and store carbohydrate foods as fat.

Furthermore, phthalates may have antiandrogenic effects, decrease sperm motility in men, is associated with increased infant birth weight, and increases adiposity in children.

An activity stimulation programme during a child's first year reduces some indicators of adiposity at the age of two - and - a-half

The final model was repeated predicting adiposity z score and whether or not the child was overweight at 36 mo, with control for adiposity z score at 15 mo..

Child weight status was also examined as «overweight» compared with «not overweight» based on an adiposity z score ≥ 85th percentile for age and sex.

This observation suggests that interventions focused on maternal feeding practices can have a beneficial effect on children's adiposity.

Testing the hypothesis that certain maternal feeding behaviors increase children's adiposity is difficult because the suspect feeding behaviors tend to cluster within low - income and minority populations, which have a higher risk of childhood obesity (22, 23).

Associations between multiple measures of parental feeding and children's adiposity in United Kingdom preschoolers.

Association of exposure to diabetes in utero with adiposity and fat distribution in a multiethnic population of youth: the Exploring Perinatal Outcomes among Children (EPOCH) Study

Regarding the child, the importance of the intrauterine and early postnatal environments for metabolic programming and modifications of the epigenome is increasingly recognised, 12 — 14 particularly for metabolic diseases such as obesity and diabetes.15 Thus, GDM is related to macrosomia at birth (> 4 kg), to excess body fat and (central) obesity and to insulin secretion in infants and children, the obesity being in part mediated by maternal body mass index (BMI) or birth weight.16 — 23 Intrauterine exposure to GDM also doubles the risk for subsequent type 2 diabetes in offspring compared with offspring of mothers with a high genetic predisposition for type 2 diabetes, but with normal glucose tolerance during the index pregnancy.24 Maternal prepregnancy overweight and excessive gestational weight gain also predict high birth weight and adiposity during infancy.12 25 This is highly relevant, as up to 60 % — 70 % of women with GDM are overweight or obese before pregnancy.26 Finally, maternal lifestyle behaviour such as a high fat diet or lack of physical activity during pregnancy can influence offspring adiposity independent of maternal obesity.12 27