Not exact matches
Unlike MCFAs, LCFAs are harder for the body to break down, put more strain
on the pancreas and liver, are usually stored as fat in the body, and
deposited in the arteries, often as
cholesterol.
When large quantities of LDL
cholesterol circulate through the bloodstream, it can be
deposited on artery walls, leading to a buildup of plaque and triggering inflammation.
The tiny particles are 1,000 times smaller than the tip of a human hair, and are designed to latch
on to atherosclerotic plaques — hard
deposits made from accumulated fat,
cholesterol and calcium that build up
on the walls of arteries and are prone to rupture, producing dangerous clots.
While working at the Institute in 1959, the biochemist and expert in human nutrition David Kritchevsky, Ph.D., published the influential textbook
Cholesterol, which explored his studies on the mechanisms of atherosclerosis, or fat deposits in blood vessels, and the role of cholesterol deposits in cardiovascul
Cholesterol, which explored his studies
on the mechanisms of atherosclerosis, or fat
deposits in blood vessels, and the role of
cholesterol deposits in cardiovascul
cholesterol deposits in cardiovascular disease.
On top of that, there is the «good» (HDL) and «bad» (LDL) kind of
cholesterol — according to the American Heart Association, the first one contributes to the production of thick, hard
deposits that can clog arteries and make them less flexible, increasing the risk of heart attacks and other cardiovascular diseases.
Cholesterol is a healing substance within the body (among many other important functions), and responds to arterial inflammation by getting
deposited in combination with other substances, forming «plaque» as a healing agent
on the artery lining.
It is clinically shown to lower LDL («bad»)
cholesterol, and raise HDL («good»)
cholesterol, by removing fatty
cholesterol deposits that build up
on artery walls that may cause blockages of blood flow to the heart, brain, and other organs.